Thomas Philips

3.8k total citations · 2 hit papers
15 papers, 2.5k citations indexed

About

Thomas Philips is a scholar working on Neurology, Neurology and Molecular Biology. According to data from OpenAlex, Thomas Philips has authored 15 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Neurology, 9 papers in Neurology and 4 papers in Molecular Biology. Recurrent topics in Thomas Philips's work include Amyotrophic Lateral Sclerosis Research (10 papers), Parkinson's Disease Mechanisms and Treatments (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (5 papers). Thomas Philips is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (10 papers), Parkinson's Disease Mechanisms and Treatments (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (5 papers). Thomas Philips collaborates with scholars based in United States, Belgium and Netherlands. Thomas Philips's co-authors include Wim Robberecht, Jeffrey D. Rothstein, Ludo Van Den Bosch, Philip Van Damme, Kim A. Staats, William D. Richardson, Benno Küsters, André Bento‐Abreu, Wanda Haeck and Annelies Nonneman and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Journal of Neuroscience.

In The Last Decade

Thomas Philips

15 papers receiving 2.5k citations

Hit Papers

The changing scene of amyotrophic lateral sclerosis 2011 2026 2016 2021 2013 2011 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. The changing scene of amyotrophic lateral sclerosis
    2013 756 citations Wim Robberecht, Thomas Philips Nature reviews. Neuroscience profile →
  2. Neuroinflammation in amyotrophic lateral sclerosis: role of glial activation in motor neuron disease
    2011 497 citations Thomas Philips, Wim Robberecht The Lancet Neurology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Philips United States 11 1.6k 870 858 693 465 15 2.5k
Christian S. Lobsiger France 21 1.7k 1.1× 977 1.1× 893 1.0× 955 1.4× 764 1.6× 32 3.0k
Carlos J. Miranda United States 19 1.2k 0.7× 1.4k 1.6× 693 0.8× 675 1.0× 754 1.6× 35 2.8k
Makiko Nagai Japan 27 2.1k 1.3× 1.2k 1.4× 1.0k 1.2× 788 1.1× 803 1.7× 61 3.4k
Lyndsey Braun United States 14 1.1k 0.7× 1.3k 1.5× 1.0k 1.2× 591 0.9× 440 0.9× 20 2.7k
Michael L. Garcia United States 20 1.1k 0.7× 850 1.0× 749 0.9× 423 0.6× 500 1.1× 37 2.2k
Cédric Raoul France 28 1.2k 0.8× 1.3k 1.5× 664 0.8× 359 0.5× 688 1.5× 70 2.7k
László Siklós Hungary 27 1.7k 1.1× 905 1.0× 759 0.9× 1.0k 1.5× 603 1.3× 48 3.1k
Frédérique René France 28 1.7k 1.1× 1.2k 1.4× 1.0k 1.2× 360 0.5× 711 1.5× 59 2.9k
Hristelina Ilieva United States 14 1.3k 0.8× 1.0k 1.2× 731 0.9× 373 0.5× 430 0.9× 24 2.2k
Meraida Polak United States 18 1.6k 1.0× 1.4k 1.6× 1.2k 1.4× 313 0.5× 611 1.3× 24 2.8k

Countries citing papers authored by Thomas Philips

Since Specialization
Citations

This map shows the geographic impact of Thomas Philips's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Philips with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Philips more than expected).

Fields of papers citing papers by Thomas Philips

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Philips. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Philips. The network helps show where Thomas Philips may publish in the future.

Co-authorship network of co-authors of Thomas Philips

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Philips. A scholar is included among the top collaborators of Thomas Philips based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Philips. Thomas Philips is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Poffenberger, Maya C., Benedict Wu, Jonathan Li, et al.. (2025). ZW1528, A Bispecific Antibody Targeting IL-4Rα and IL-33, Potently Inhibits Key Mediators of Airway Inflammation. American Journal of Respiratory and Critical Care Medicine. 211(Supplement_1). A3446–A3446. 2 indexed citations
2.
Thompson, Emily G., Sigvard Åkerman, Benjamin L. Zaepfel, et al.. (2024). A robust evaluation of TDP-43, poly GP, cellular pathology and behavior in an AAV-C9ORF72 (G4C2)66 mouse model. Acta Neuropathologica Communications. 12(1). 203–203. 2 indexed citations
3.
Philips, Thomas, Yevgeniya A. Mironova, Yan Jouroukhin, et al.. (2021). MCT1 Deletion in Oligodendrocyte Lineage Cells Causes Late-Onset Hypomyelination and Axonal Degeneration. Cell Reports. 34(2). 108610–108610. 79 indexed citations
4.
Philips, Thomas & Jeffrey D. Rothstein. (2017). Oligodendroglia: metabolic supporters of neurons. Journal of Clinical Investigation. 127(9). 3271–3280. 237 indexed citations
5.
Chang, Lydia, Polymnia Georgiou, Nathaniel Safren, et al.. (2016). Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS–FTD-linked UBQLN2 mutations. Proceedings of the National Academy of Sciences. 113(47). E7580–E7589. 71 indexed citations
6.
Philips, Thomas & Jeffrey D. Rothstein. (2015). Rodent Models of Amyotrophic Lateral Sclerosis. Current Protocols in Pharmacology. 69(1). 5.67.1–5.67.21. 217 indexed citations
7.
Philips, Thomas & Jeffrey D. Rothstein. (2014). Glial cells in amyotrophic lateral sclerosis. Experimental Neurology. 262. 111–120. 193 indexed citations
8.
Philips, Thomas, Jeffrey D. Rothstein, & Mahmoud A. Pouladi. (2014). Preclinical models: Needed in translation? A Pro/Con debate. Movement Disorders. 29(11). 1391–1396. 8 indexed citations
9.
Robberecht, Wim & Thomas Philips. (2013). The changing scene of amyotrophic lateral sclerosis. Nature reviews. Neuroscience. 14(4). 248–264. 756 indexed citations breakdown →
10.
Philips, Thomas, André Bento‐Abreu, Annelies Nonneman, et al.. (2013). Oligodendrocyte dysfunction in the pathogenesis of amyotrophic lateral sclerosis. Brain. 136(2). 471–482. 205 indexed citations
11.
Philips, Thomas & Wim Robberecht. (2011). Neuroinflammation in amyotrophic lateral sclerosis: role of glial activation in motor neuron disease. The Lancet Neurology. 10(3). 253–263. 497 indexed citations breakdown →
12.
Brozzi, Flora, Frédérique Diraison, Shavanthi Rajatileka, et al.. (2011). Molecular Mechanism of Myosin Va Recruitment to Dense Core Secretory Granules. Traffic. 13(1). 54–69. 44 indexed citations
13.
Philips, Thomas. (2011). Non-neuronal cells in the pathogenesis of Amyotrophic Lateral Sclerosis. 1 indexed citations
14.
Philips, Thomas, Louis De Muynck, Peter Vanacker, et al.. (2010). Microglial Upregulation of Progranulin as a Marker of Motor Neuron Degeneration. Journal of Neuropathology & Experimental Neurology. 69(12). 1191–1200. 59 indexed citations
15.
Gowing, Genevíève, Thomas Philips, Bart Van Wijmeersch, et al.. (2008). Ablation of Proliferating Microglia Does Not Affect Motor Neuron Degeneration in Amyotrophic Lateral Sclerosis Caused by Mutant Superoxide Dismutase. Journal of Neuroscience. 28(41). 10234–10244. 114 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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