L Shi

2.0k total citations · 1 hit paper
8 papers, 1.7k citations indexed

About

L Shi is a scholar working on Molecular Biology, Oncology and Infectious Diseases. According to data from OpenAlex, L Shi has authored 8 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Oncology and 1 paper in Infectious Diseases. Recurrent topics in L Shi's work include Cell death mechanisms and regulation (3 papers), Ubiquitin and proteasome pathways (3 papers) and Mitochondrial Function and Pathology (2 papers). L Shi is often cited by papers focused on Cell death mechanisms and regulation (3 papers), Ubiquitin and proteasome pathways (3 papers) and Mitochondrial Function and Pathology (2 papers). L Shi collaborates with scholars based in Canada, United States and Italy. L Shi's co-authors include Harry B. Greenberg, Ruedi Aebersold, James C. Powers, Chih Min Kam, L Bergeron, Junying Yuan, G. MacDonald, Susannah Varmuza, Keith E. Latham and Jodi A. Flaws and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and Genes & Development.

In The Last Decade

L Shi

8 papers receiving 1.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Defects in regulation of apoptosis in caspase-2-deficient mice

1998 560 citations L Bergeron, Gloria I. Perez et al. Genes & Development profile →

Peers

Countries citing papers authored by L Shi

Since Specialization

Citations

This map shows the geographic impact of L Shi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by L Shi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites L Shi more than expected).

Fields of papers citing papers by L Shi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by L Shi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by L Shi. The network helps show where L Shi may publish in the future.

Co-authorship network of co-authors of L Shi

This figure shows the co-authorship network connecting the top 25 collaborators of L Shi. A scholar is included among the top collaborators of L Shi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with L Shi. L Shi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

8 of 8 papers shown

#	Work	Indexed citations
1	Wee1 inhibitor optimization through deep-learning-driven decision making 2024 ·European Journal of Medicinal Chemistry ·Yan Yang, (unknown), Yanxing Wang, (unknown), (unknown), (unknown), (unknown), (unknown), Lijun Wang, L Shi, Chengtao Li	1
2	Defects in regulation of apoptosis in caspase-2-deficient mice breakdown → 1998 ·Genes & Development ·L Bergeron, Gloria I. Perez, G. MacDonald, L Shi, Yunfu Sun, Andrea Jurisicova, Susannah Varmuza, Keith E. Latham, Jodi A. Flaws, Janine Salter, Hideaki Hara, Michael A. Moskowitz, (unknown), Harry B. Greenberg, Jonathan L. Tilly, Junying Yuan	560
3	Granzyme B induces apoptosis and cyclin A-associated cyclin-dependent kinase activity in all stages of the cell cycle 1996 ·The Journal of Immunology ·L Shi, (unknown), Dongning He, Denis G. Bosc, David W. Litchfield, Harry B. Greenberg	50
4	The cytotoxic cell protease granzyme B initiates apoptosis in a cell-free system by proteolytic processing and activation of the ICE/CED-3 family protease, CPP32, via a novel two-step mechanism. 1996 ·The EMBO Journal ·Séamus J. Martin, Gustavo P. Amarante‐Mendes, L Shi, Tsung‐Hsien Chuang, Carlos A. Casiano, (unknown), Patrick Fitzgerald, Eng M. Tan, Gary Bokoch, Harry B. Greenberg, Douglas R. Green	250
5	Activation of an interleukin 1 converting enzyme-dependent apoptosis pathway by granzyme B. 1996 ·Proceedings of the National Academy of Sciences ·L Shi, (unknown), G. MacDonald, L Bergeron, (unknown), Masayuki Miura, Rocco J. Rotello, Douglas K. Miller, Peng Li, Tara Seshadri, Junying Yuan, Harry B. Greenberg	74
6	Rescue from granzyme B-induced apoptosis by Wee1 kinase. 1995 ·The Journal of Experimental Medicine ·(unknown), L Shi, David W. Litchfield, Harry B. Greenberg	53
7	A natural killer cell granule protein that induces DNA fragmentation and apoptosis. 1992 ·The Journal of Experimental Medicine ·L Shi, (unknown), Ruedi Aebersold, Harry B. Greenberg	333
8	Purification of three cytotoxic lymphocyte granule serine proteases that induce apoptosis through distinct substrate and target cell interactions. 1992 ·The Journal of Experimental Medicine ·L Shi, Chih Min Kam, James C. Powers, Ruedi Aebersold, Harry B. Greenberg	392

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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