Kim Milarski
- Molecular Biology top 5%
- Immunology top 5%
- Cell Biology top 5%
- Oncology top 10%
- Endocrinology, Diabetes and Metabolism top 10%
- Co-authors
- Richard I. MorimotoAlan R. SaltielKosei YamauchiJeffrey E. PessinJerrold M. OlefskyDonna ReichartB L SeelyWilliam J. Welch
- Topics
- Protein Kinase Regulation and GTPase Signaling (7 papers)PI3K/AKT/mTOR signaling in cancer (6 papers)Protein Tyrosine Phosphatases (5 papers)
- Cited by
- ImmunologyAgingMolecular Biology
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryThe Journal of Cell Biology
- Partner nations
- United StatesCanadaGermany
In The Last Decade
Kim Milarski
18 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 1.7k
- Immunology 709
- Cell Biology 260
- Oncology 257
- Endocrinology, Diabetes and Metabolism 153
Countries citing papers authored by Kim Milarski
This map shows the geographic impact of Kim Milarski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kim Milarski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kim Milarski more than expected).
Fields of papers citing papers by Kim Milarski
This network shows the impact of papers produced by Kim Milarski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kim Milarski. The network helps show where Kim Milarski may publish in the future.
Co-authorship network of co-authors of Kim Milarski
This figure shows the co-authorship network connecting the top 25 collaborators of Kim Milarski. A scholar is included among the top collaborators of Kim Milarski based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kim Milarski. Kim Milarski is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 21 | |
| 2 | 135 | |
| 3 | 2 | |
| 4 | 156 | |
| 5 | 180 | |
| 6 | 263 | |
| 7 | 59 | |
| 8 | 22 | |
| 9 | 18 | |
| 10 | 235 | |
| 11 | 80 | |
| 12 | 29 | |
| 13 | 274 | |
| 14 | 89 | |
| 15 | 21 | |
| 16 | 154 | |
| 17 | 131 | |
| 18 | 283 |
About Kim Milarski
Kim Milarski is a scholar working on Immunology and Allergy, Molecular Biology and Clinical Biochemistry, having authored 18 papers that have together received 2.2k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (7 papers), PI3K/AKT/mTOR signaling in cancer (6 papers) and Protein Tyrosine Phosphatases (5 papers). The work is most often cited by research in Immunology (709 citations), Aging (50 citations) and Molecular Biology (1.7k citations). Kim Milarski has collaborated with scholars based in United States, Canada and Germany. Frequent co-authors include Richard I. Morimoto, Alan R. Saltiel, Kosei Yamauchi, Jeffrey E. Pessin, Jerrold M. Olefsky, Donna Reichart, B L Seely, William J. Welch, Paulos Berhanu and Jyotirmoy Kusari. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.