Kengo Homma

940 total citations
13 papers, 532 citations indexed

About

Kengo Homma is a scholar working on Neurology, Molecular Biology and Genetics. According to data from OpenAlex, Kengo Homma has authored 13 papers receiving a total of 532 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Neurology, 5 papers in Molecular Biology and 4 papers in Genetics. Recurrent topics in Kengo Homma's work include Amyotrophic Lateral Sclerosis Research (8 papers), Neurogenetic and Muscular Disorders Research (4 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Kengo Homma is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (8 papers), Neurogenetic and Muscular Disorders Research (4 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Kengo Homma collaborates with scholars based in Japan, United States and Germany. Kengo Homma's co-authors include Hidenori Ichijo, Yuki Hayashi, Atsushi Matsuzawa, Kohsuke Takeda, Hideki Nishitoh, Isao Naguro, Takao Fujisawa, Takuya Noguchi, Hiroaki Nagai and Hisae Kadowaki and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and SHILAP Revista de lepidopterología.

In The Last Decade

Kengo Homma

13 papers receiving 526 citations

Peers

Kengo Homma
Kengo Homma
Citations per year, relative to Kengo Homma Kengo Homma (= 1×) peers Elisa Lupino

Countries citing papers authored by Kengo Homma

Since Specialization
Citations

This map shows the geographic impact of Kengo Homma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kengo Homma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kengo Homma more than expected).

Fields of papers citing papers by Kengo Homma

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kengo Homma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kengo Homma. The network helps show where Kengo Homma may publish in the future.

Co-authorship network of co-authors of Kengo Homma

This figure shows the co-authorship network connecting the top 25 collaborators of Kengo Homma. A scholar is included among the top collaborators of Kengo Homma based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kengo Homma. Kengo Homma is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Kondo, Takayuki, Mika Suga, Ryu Yamanaka, et al.. (2024). Induced pluripotent stem cell‐based assays recapture multiple properties of human astrocytes. Journal of Cellular and Molecular Medicine. 28(7). e18214–e18214. 3 indexed citations
2.
Tanaka, Masaharu, et al.. (2021). Histopathological changes of the spinal cord and motor neuron dynamics in SOD1 Tg mice. Journal of Toxicologic Pathology. 35(1). 129–133. 1 indexed citations
3.
Homma, Kengo, et al.. (2020). Genome-wide siRNA screening reveals that DCAF4-mediated ubiquitination of optineurin stimulates autophagic degradation of Cu,Zn-superoxide dismutase. Journal of Biological Chemistry. 295(10). 3148–3158. 4 indexed citations
4.
Iriyama, Takayuki, Kensuke Suzuki, Seisuke Sayama, et al.. (2020). ASK1 promotes uterine inflammation leading to pathological preterm birth. Scientific Reports. 10(1). 8 indexed citations
5.
Ichijo, Hidenori, et al.. (2016). Mislocalization, aggregation formation and defect in proteolysis in ALS. SHILAP Revista de lepidopterología. 3(2). 246–268. 2 indexed citations
6.
Zhai, Jinbin, Lixin Zhang, Jelena Mojsilovic-Petrovic, et al.. (2015). Inhibition of Cytohesins Protects against Genetic Models of Motor Neuron Disease. Journal of Neuroscience. 35(24). 9088–9105. 18 indexed citations
7.
Fujisawa, Takao, Hisae Kadowaki, H. Takahashi, et al.. (2015). A systematic immunoprecipitation approach reinforces the concept of common conformational alterations in amyotrophic lateral sclerosis-linked SOD1 mutants. Neurobiology of Disease. 82. 478–486. 7 indexed citations
8.
Fujisawa, Takao, M Takahashi, Hiroshi Kodaira, et al.. (2015). The ASK1-specific inhibitors K811 and K812 prolong survival in a mouse model of amyotrophic lateral sclerosis. Human Molecular Genetics. 25(2). 245–253. 38 indexed citations
9.
Hayashi, Yuki, Kengo Homma, & Hidenori Ichijo. (2015). SOD1 in neurotoxicity and its controversial roles in SOD1 mutation-negative ALS. Advances in Biological Regulation. 60. 95–104. 114 indexed citations
10.
Homma, Kengo, Takao Fujisawa, Hisae Kadowaki, et al.. (2013). SOD1 as a Molecular Switch for Initiating the Homeostatic ER Stress Response under Zinc Deficiency. Molecular Cell. 52(1). 75–86. 110 indexed citations
11.
Fujisawa, Takao, Kengo Homma, Hisae Kadowaki, et al.. (2012). A novel monoclonal antibody reveals a conformational alteration shared by amyotrophic lateral sclerosis‐linked SOD1 mutants. Annals of Neurology. 72(5). 739–749. 62 indexed citations
12.
Nagai, Hiroaki, Takuya Noguchi, Kengo Homma, et al.. (2009). Ubiquitin-like Sequence in ASK1 Plays Critical Roles in the Recognition and Stabilization by USP9X and Oxidative Stress-Induced Cell Death. Molecular Cell. 36(5). 805–818. 122 indexed citations
13.
Homma, Kengo, et al.. (2009). Targeting ASK1 in ER stress-related neurodegenerative diseases. Expert Opinion on Therapeutic Targets. 13(6). 653–664. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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