Keizo Nishikawa

2.6k total citations
28 papers, 2.0k citations indexed

About

Keizo Nishikawa is a scholar working on Molecular Biology, Cancer Research and Immunology. According to data from OpenAlex, Keizo Nishikawa has authored 28 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 8 papers in Cancer Research and 7 papers in Immunology. Recurrent topics in Keizo Nishikawa's work include Bone Metabolism and Diseases (10 papers), Genomics, phytochemicals, and oxidative stress (6 papers) and NF-κB Signaling Pathways (4 papers). Keizo Nishikawa is often cited by papers focused on Bone Metabolism and Diseases (10 papers), Genomics, phytochemicals, and oxidative stress (6 papers) and NF-κB Signaling Pathways (4 papers). Keizo Nishikawa collaborates with scholars based in Japan, United States and Cameroon. Keizo Nishikawa's co-authors include Masayuki Yamamoto, Makoto Kobayashi, Takafumi Suzuki, Ken Itoh, Shigeaki Kato, Hiroshi Takayanagi, Atsushi Maruyama, Yasutake Katoh, Satoru Takahashi and Tomonori Hosoya and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Keizo Nishikawa

26 papers receiving 2.0k citations

Peers

Keizo Nishikawa

IMMUN

ONCOL

RHEUM

Anling Liu China

Simonetta Astigiano Italy

Sudarshana M. Sharma United States

Hyunsoo Kim South Korea

Gerold Untergasser Austria

Kotaro Sugimoto Japan

Kim E. Boulukos France

Danilo Cucchi Italy

Xiaojun Zhang China

Daewon Jeong South Korea

Anling Liu China

Keizo Nishikawa

1.1k ×0.8

214 ×0.7

IMMUN

268 ×0.9

258 ×1.0

ONCOL

196 ×1.4

RHEUM

Citations per year, relative to Keizo Nishikawa Keizo Nishikawa (= 1×) peers Anling Liu

Countries citing papers authored by Keizo Nishikawa

Since Specialization

Citations

This map shows the geographic impact of Keizo Nishikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keizo Nishikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keizo Nishikawa more than expected).

Fields of papers citing papers by Keizo Nishikawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keizo Nishikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keizo Nishikawa. The network helps show where Keizo Nishikawa may publish in the future.

Co-authorship network of co-authors of Keizo Nishikawa

This figure shows the co-authorship network connecting the top 25 collaborators of Keizo Nishikawa. A scholar is included among the top collaborators of Keizo Nishikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keizo Nishikawa. Keizo Nishikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kawabata, Hiroshi, et al.. (2025). Clustered peptide regulating the multivalent interaction between RANK and TRAF6 inhibits osteoclastogenesis by fine-tuning signals. Communications Biology. 8(1). 643–643. 1 indexed citations

Yamashita, Erika, Junichi Kikuta, Tomoka Ao, et al.. (2022). Osteoblast-derived vesicles induce a switch from bone-formation to bone-resorption in vivo. Nature Communications. 13(1). 1066–1066. 67 indexed citations

Yoshihara, Toshitada, Junichi Kikuta, Reiko Sakaguchi, et al.. (2022). Determination of the physiological range of oxygen tension in bone marrow monocytes using two-photon phosphorescence lifetime imaging microscopy. Scientific Reports. 12(1). 3497–3497. 15 indexed citations

Kikuta, Junichi, Keizo Nishikawa, Takao Sudo, et al.. (2021). SLPI is a critical mediator that controls PTH-induced bone formation. Nature Communications. 12(1). 2136–2136. 31 indexed citations

Nishikawa, Keizo & Masaru Ishii. (2020). Novel method for gain-of-function analyses in primary osteoclasts using a non-viral gene delivery system. Journal of Bone and Mineral Metabolism. 39(3). 353–359. 2 indexed citations

Nishikawa, Keizo, et al.. (2018). Roles of Enhancer RNAs in RANKL-induced Osteoclast Differentiation Identified by Genome-wide Cap-analysis of Gene Expression using CRISPR/Cas9. Scientific Reports. 8(1). 7504–7504. 15 indexed citations

Nishikawa, Keizo. (2016). [Elucidation of the role of metabolic reprogramming in osteoclast differentiation].. PubMed. 26(5). 713–9.

Nishikawa, Keizo, Yoriko Iwamoto, Yasuhiro Kobayashi, et al.. (2015). DNA methyltransferase 3a regulates osteoclast differentiation by coupling to an S-adenosylmethionine–producing metabolic pathway. Nature Medicine. 21(3). 281–287. 179 indexed citations

Naito, Atsushi, Hirofumi Yamamoto, Yoshinori Kagawa, et al.. (2015). RFPL4A Increases the G1 Population and Decreases Sensitivity to Chemotherapy in Human Colorectal Cancer Cells. Journal of Biological Chemistry. 290(10). 6326–6337. 4 indexed citations

10.

Nishikawa, Keizo, Yoriko Iwamoto, & Masaru Ishii. (2013). Development of an in vitro culture method for stepwise differentiation of mouse embryonic stem cells and induced pluripotent stem cells into mature osteoclasts. Journal of Bone and Mineral Metabolism. 32(3). 331–336. 8 indexed citations

11.

Nishikawa, Keizo, Tomoki Nakashima, Mikihito Hayashi, et al.. (2012). Active repression by Blimp1 play an important role in osteoclast differentiation. Arthritis Research & Therapy. 14(S1).

12.

Takeuchi, Miki, Hiroshi Kaneko, Keizo Nishikawa, et al.. (2010). Efficient transient rescue of hematopoietic mutant phenotypes in zebrafish usingTol2‐mediated transgenesis. Development Growth & Differentiation. 52(2). 245–250. 10 indexed citations

13.

Nishikawa, Keizo, Tomoki Nakashima, Shu Takeda, et al.. (2010). Maf promotes osteoblast differentiation in mice by mediating the age-related switch in mesenchymal cell differentiation. Journal of Clinical Investigation. 120(10). 3455–3465. 149 indexed citations

14.

Asagiri, Masataka, Toshitake Hirai, Toshihiro Kunigami, et al.. (2008). Cathepsin K-Dependent Toll-Like Receptor 9 Signaling Revealed in Experimental Arthritis. Science. 319(5863). 624–627. 290 indexed citations

15.

Maruyama, Atsushi, Keizo Nishikawa, Aruto Yoshida, et al.. (2008). Nrf2 regulates the alternative first exons of CD36 in macrophages through specific antioxidant response elements. Archives of Biochemistry and Biophysics. 477(1). 139–145. 87 indexed citations

16.

Shimizu, Ritsuko, Cecelia D. Trainor, Keizo Nishikawa, et al.. (2007). GATA-1 Self-association Controls Erythroid Development in Vivo. Journal of Biological Chemistry. 282(21). 15862–15871. 22 indexed citations

17.

Kobayashi, Makoto, et al.. (2004). MafT, a new member of the small Maf protein family in zebrafish. Biochemical and Biophysical Research Communications. 320(1). 62–69. 48 indexed citations

18.

Kobayashi, Makoto, Ken Itoh, Takafumi Suzuki, et al.. (2002). Identification of the interactive interface and phylogenic conservation of the Nrf2‐Keap1 system. Genes to Cells. 7(8). 807–820. 296 indexed citations

19.

Kobayashi, Makoto, Keizo Nishikawa, & Masayuki Yamamoto. (2001). Hematopoietic regulatory domain ofgata1gene is positively regulated by GATA1 protein in zebrafish embryos. Development. 128(12). 2341–2350. 66 indexed citations

20.

Kobayashi, Makoto, Keizo Nishikawa, Takafumi Suzuki, & Masayuki Yamamoto. (2001). The Homeobox Protein Six3 Interacts with the Groucho Corepressor and Acts as a Transcriptional Repressor in Eye and Forebrain Formation. Developmental Biology. 232(2). 315–326. 152 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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