Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Leptin Inhibits Bone Formation through a Hypothalamic Relay
20001.6k citationsPatricia Ducy, Shu Takeda et al.Cellprofile →
Leptin Regulates Bone Formation via the Sympathetic Nervous System
20021.3k citationsShu Takeda, Florent Elefteriou et al.Cellprofile →
Leptin regulation of bone resorption by the sympathetic nervous system and CART
20051.0k citationsFlorent Elefteriou, Shu Takeda et al.profile →
Mice lacking the vitamin D receptor exhibit impaired bone formation, uterine hypoplasia and growth retardation after weaning
This map shows the geographic impact of Shu Takeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shu Takeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shu Takeda more than expected).
This network shows the impact of papers produced by Shu Takeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shu Takeda. The network helps show where Shu Takeda may publish in the future.
Co-authorship network of co-authors of Shu Takeda
This figure shows the co-authorship network connecting the top 25 collaborators of Shu Takeda.
A scholar is included among the top collaborators of Shu Takeda based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Shu Takeda. Shu Takeda is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Sato, Tsuyoshi, Yuichiro Enoki, Masahiko OKUBO, et al.. (2017). Semaphorin 3A Promotes Dendrite Elongation of Osteocytes in Association with Down-regulation of CDK6.. PubMed. 30(3). 231–6.9 indexed citations
Ochi, Hiroki & Shu Takeda. (2014). [Bone metabolism and cardiovascular function update. Nerve system and mutual interaction between bone and blood vessel].. PubMed. 24(7). 77–83.2 indexed citations
9.
Sato, Shingo & Shu Takeda. (2014). [Regulation of bone metastasis by microRNAs].. PubMed. 24(8). 1209–15.1 indexed citations
10.
Fukuda, Toru & Shu Takeda. (2013). [Regulation of osteoblastic differentiation by Wnt signals].. PubMed. 23(6). 825–30.1 indexed citations
11.
Fukuda, Toru & Shu Takeda. (2013). [Secondary osteoporosis or secondary contributors to bone loss in fracture. Regulation of bone homeostasis by nerve system].. PubMed. 23(9). 1279–83.3 indexed citations
Takeda, Shu. (2005). [Central control of bone metabolism].. PubMed. 15(5). 797–804.2 indexed citations
18.
Takeda, Shu. (2004). [Leptin and beta-blockers in bone metabolism].. PubMed. 14(2). 241–7.2 indexed citations
19.
Takeda, Shu, Florent Elefteriou, Régis Levasseur, et al.. (2002). Leptin Regulates Bone Formation via the Sympathetic Nervous System. Cell. 111(3). 305–317.1330 indexed citations breakdown →
20.
Takeda, Shu & Gérard Karsenty. (2001). Central control of bone formation. Journal of Bone and Mineral Metabolism. 19(3). 195–198.85 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.