William E. Louch

6.5k total citations
150 papers, 4.3k citations indexed

About

William E. Louch is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, William E. Louch has authored 150 papers receiving a total of 4.3k indexed citations (citations by other indexed papers that have themselves been cited), including 115 papers in Cardiology and Cardiovascular Medicine, 90 papers in Molecular Biology and 36 papers in Cellular and Molecular Neuroscience. Recurrent topics in William E. Louch's work include Cardiac electrophysiology and arrhythmias (93 papers), Ion channel regulation and function (67 papers) and Neuroscience and Neural Engineering (29 papers). William E. Louch is often cited by papers focused on Cardiac electrophysiology and arrhythmias (93 papers), Ion channel regulation and function (67 papers) and Neuroscience and Neural Engineering (29 papers). William E. Louch collaborates with scholars based in Norway, United States and United Kingdom. William E. Louch's co-authors include Ole M. Sejersted, Ivar Sjaastad, Beata M. Wolska, Katherine A. Sheehan, Michael Frisk, Geir Christensen, Jan Magnus Aronsen, Halvor K. Mørk, Åsmund T. Røe and Fredrik Swift and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

William E. Louch

144 papers receiving 4.3k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
William E. Louch	2.9k	2.6k	826	413	285	150	4.3k
Itsuo Kodama	4.2k 1.5×	2.9k 1.1×	913 1.1×	471 1.1×	224 0.8×	209	5.9k
Joshua I. Goldhaber	3.1k 1.1×	3.6k 1.4×	1.1k 1.3×	466 1.1×	189 0.7×	113	5.3k
Karin R. Sipido	4.7k 1.6×	4.1k 1.6×	1.3k 1.6×	402 1.0×	169 0.6×	169	6.4k
Thomas J. Hund	3.3k 1.1×	3.5k 1.4×	753 0.9×	396 1.0×	245 0.9×	126	5.0k
Ivar Sjaastad	3.5k 1.2×	3.0k 1.2×	582 0.7×	544 1.3×	452 1.6×	231	5.9k
Patrick Most	2.0k 0.7×	3.0k 1.2×	264 0.3×	357 0.9×	137 0.5×	104	4.2k
Gonzalo Pizarro	1.9k 0.6×	1.9k 0.7×	1.2k 1.4×	151 0.4×	108 0.4×	100	3.3k
Anthony O. Gramolini	2.0k 0.7×	4.2k 1.6×	650 0.8×	628 1.5×	530 1.9×	120	6.0k
Dayue Darrel Duan	1.5k 0.5×	2.7k 1.0×	860 1.0×	201 0.5×	130 0.5×	92	3.6k
Anne‐Marie Lompré	3.5k 1.2×	3.8k 1.5×	458 0.6×	483 1.2×	420 1.5×	92	6.1k

Countries citing papers authored by William E. Louch

Since Specialization

Citations

This map shows the geographic impact of William E. Louch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William E. Louch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William E. Louch more than expected).

Fields of papers citing papers by William E. Louch

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William E. Louch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William E. Louch. The network helps show where William E. Louch may publish in the future.

Co-authorship network of co-authors of William E. Louch

This figure shows the co-authorship network connecting the top 25 collaborators of William E. Louch. A scholar is included among the top collaborators of William E. Louch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William E. Louch. William E. Louch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Jæger, Karoline Horgmo, William E. Louch, & Aslak Tveito. (2025). Reduced gap junction coupling amplifies the effects of cardiomyocyte variability and destabilizes the heartbeat. Physiological Reports. 13(13). e70461–e70461. 2 indexed citations

Lauritzen, Knut H., Kuan Yang, Michael Frisk, et al.. (2025). Apigenin inhibits NLRP3 inflammasome activation in monocytes and macrophages independently of CD38. Frontiers in Immunology. 15. 1497984–1497984. 2 indexed citations

Louch, William E., et al.. (2025). Bioimpedance and electrophysiology measurements of engineered heart tissues outside the culture medium. Biosensors and Bioelectronics. 287. 117634–117634.

Louch, William E., et al.. (2025). Interplay Between Ryanodine Receptor Arrangement and Function: Implications for (Patho)physiological Control of Calcium Release. Circulation Research. 137(6). 902–923. 1 indexed citations

Lunde, Marianne, et al.. (2024). Exploring Syndecan-4 and MLP and Their Interaction in Primary Cardiomyocytes and H9c2 Cells. Cells. 13(11). 947–947. 1 indexed citations

Zhang, Xianwei, Yixuan Wu, Charlotte Smith, et al.. (2024). Enhanced Ca2+-Driven Arrhythmogenic Events in Female Patients With Atrial Fibrillation. JACC. Clinical electrophysiology. 10(11). 2371–2391. 4 indexed citations

Frisk, Michael, Cathrine R. Carlson, Andreas Brech, et al.. (2023). BIN1, Myotubularin, and Dynamin-2 Coordinate T-Tubule Growth in Cardiomyocytes. Circulation Research. 132(11). e188–e205. 15 indexed citations

Veletić, Mladen, et al.. (2023). Human Heart Organoid-derived Extracellular Vesicles for Cardiac Intercellular Communication. 122–128.

Li, Jia, Joakim Sundnes, Yufeng Hou, et al.. (2023). Stretch Harmonizes Sarcomere Strain Across the Cardiomyocyte. Circulation Research. 133(3). 255–270. 11 indexed citations

10.

Zhang, Xianwei, Charlotte Smith, Stefano Morotti, et al.. (2022). Mechanisms of spontaneous Ca ²⁺ release‐mediated arrhythmia in a novel 3D human atrial myocyte model: II. Ca ²⁺ ‐handling protein variation. The Journal of Physiology. 601(13). 2685–2710. 10 indexed citations

11.

Zhang, Xianwei, Haibo Ni, Stefano Morotti, et al.. (2022). Mechanisms of spontaneous Ca ²⁺ release‐mediated arrhythmia in a novel 3D human atrial myocyte model: I. Transverse‐axial tubule variation. The Journal of Physiology. 601(13). 2655–2683. 12 indexed citations

12.

Melleby, Arne Olav, Emma Robinson, Jia Li, et al.. (2022). ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overload. Communications Biology. 5(1). 1392–1392. 9 indexed citations

13.

Shen, Xin, et al.. (2022). Discordant Ca2+ release in cardiac myocytes: characterization and susceptibility to pharmacological RyR2 modulation. Pflügers Archiv - European Journal of Physiology. 474(6). 625–636. 7 indexed citations

14.

Kobirumaki-Shimozawa, Fuyu, Tomohiro Nakanishi, Togo Shimozawa, et al.. (2020). Real-Time In Vivo Imaging of Mouse Left Ventricle Reveals Fluctuating Movements of the Intercalated Discs. Nanomaterials. 10(3). 532–532. 5 indexed citations

15.

Shen, Xin, Yufeng Hou, Terje R. Kolstad, et al.. (2018). 3D dSTORM imaging reveals novel detail of ryanodine receptor localization in rat cardiac myocytes. The Journal of Physiology. 597(2). 399–418. 40 indexed citations

16.

Kolstad, Terje R., Niall Macquaide, Per Lunde, et al.. (2018). Ryanodine receptor dispersion disrupts Ca2+ release in failing cardiac myocytes. eLife. 7. 71 indexed citations

17.

Louch, William E., Peter Vangheluwe, Virginie Bito, et al.. (2012). Phospholamban ablation in hearts expressing the high affinity SERCA2b isoform normalizes global Ca ²⁺ homeostasis but not Ca ²⁺ -dependent hypertrophic signaling. American Journal of Physiology-Heart and Circulatory Physiology. 302(12). H2574–H2582. 10 indexed citations

18.

Wæhre, Anne, Bente Halvorsen, Arne Yndestad, et al.. (2011). Lack of Chemokine Signaling through CXCR5 Causes Increased Mortality, Ventricular Dilatation and Deranged Matrix during Cardiac Pressure Overload. PLoS ONE. 6(4). e18668–e18668. 28 indexed citations

19.

Stokke, Mathis K., Karina Hougen, Ivar Sjaastad, et al.. (2009). Reduced SERCA2 abundance decreases the propensity for Ca2+ wave development in ventricular myocytes. Cardiovascular Research. 86(1). 63–71. 45 indexed citations

20.

Vangheluwe, Peter, Marc Tjwa, An Van den Bergh, et al.. (2006). A SERCA2 pump with an increased Ca2+ affinity can lead to severe cardiac hypertrophy, stress intolerance and reduced life span. Journal of Molecular and Cellular Cardiology. 41(2). 308–317. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact