Boris V. Skryabin

5.3k total citations
78 papers, 3.6k citations indexed

About

Boris V. Skryabin is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Genetics. According to data from OpenAlex, Boris V. Skryabin has authored 78 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 63 papers in Molecular Biology, 16 papers in Cardiology and Cardiovascular Medicine and 14 papers in Genetics. Recurrent topics in Boris V. Skryabin's work include RNA Research and Splicing (16 papers), CRISPR and Genetic Engineering (11 papers) and RNA and protein synthesis mechanisms (11 papers). Boris V. Skryabin is often cited by papers focused on RNA Research and Splicing (16 papers), CRISPR and Genetic Engineering (11 papers) and RNA and protein synthesis mechanisms (11 papers). Boris V. Skryabin collaborates with scholars based in Germany, United States and Russia. Boris V. Skryabin's co-authors include Michaela Kühn, Rita Holtwick, Hideo A. Baba, Timofey S. Rozhdestvensky, Martin F. Rath, Mads Bak, Sakari Kauppinen, Niels Tommerup, Mette Christensen and Morten Møller and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Boris V. Skryabin

75 papers receiving 3.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Boris V. Skryabin Germany 28 2.5k 675 664 638 359 78 3.6k
Luca Cartegni United States 27 5.2k 2.1× 412 0.6× 289 0.4× 913 1.4× 242 0.7× 38 6.3k
Hsiao‐Huei Chen Canada 31 1.9k 0.8× 240 0.4× 370 0.6× 981 1.5× 364 1.0× 73 3.2k
Karl Pfeifer United States 30 4.1k 1.6× 691 1.0× 439 0.7× 1.1k 1.7× 440 1.2× 58 4.8k
Marcelo A. Nóbrega United States 35 4.7k 1.9× 616 0.9× 435 0.7× 1.4k 2.2× 140 0.4× 69 5.8k
Douglas S. Annis United States 33 1.6k 0.6× 693 1.0× 273 0.4× 339 0.5× 556 1.5× 72 3.7k
Margaret Fox United Kingdom 30 3.3k 1.3× 575 0.9× 445 0.7× 817 1.3× 693 1.9× 99 4.5k
Karen Wolburg‐Buchholz Germany 25 2.4k 1.0× 527 0.8× 320 0.5× 187 0.3× 601 1.7× 38 4.4k
Philippe Chafey France 28 2.4k 0.9× 197 0.3× 260 0.4× 766 1.2× 540 1.5× 60 3.8k
Jorge Laborda Spain 39 2.9k 1.2× 509 0.8× 166 0.3× 925 1.4× 170 0.5× 86 4.4k
Magali Williamson United Kingdom 22 2.1k 0.8× 287 0.4× 430 0.6× 244 0.4× 837 2.3× 41 3.1k

Countries citing papers authored by Boris V. Skryabin

Since Specialization
Citations

This map shows the geographic impact of Boris V. Skryabin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boris V. Skryabin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boris V. Skryabin more than expected).

Fields of papers citing papers by Boris V. Skryabin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Boris V. Skryabin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boris V. Skryabin. The network helps show where Boris V. Skryabin may publish in the future.

Co-authorship network of co-authors of Boris V. Skryabin

This figure shows the co-authorship network connecting the top 25 collaborators of Boris V. Skryabin. A scholar is included among the top collaborators of Boris V. Skryabin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Boris V. Skryabin. Boris V. Skryabin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Skryabin, Boris V., et al.. (2025). CRISPR-Cas9 HDR optimization: RAD52, denatured, and 5′-modified DNA templates in knock-in mice generation. iScience. 28(11). 113803–113803.
2.
Belozertseva, Irina, et al.. (2024). Advancing 3Rs: The Mouse Estrus Detector (MED) as a Low-Stress, Painless, and Efficient Tool for Estrus Determination in Mice. International Journal of Molecular Sciences. 25(17). 9429–9429. 1 indexed citations
3.
Walter, Carolin, Natalia Moreno, Marc Hotfilder, et al.. (2023). A Carboxy-terminal Smarcb1 Point Mutation Induces Hydrocephalus Formation and Affects AP-1 and Neuronal Signalling Pathways in Mice. Cellular and Molecular Neurobiology. 43(7). 3511–3526. 4 indexed citations
4.
5.
Werner, Franziska, Katharina Völker, Marco Abeßer, et al.. (2023). Ablation of C-type natriuretic peptide/cGMP signaling in fibroblasts exacerbates adverse cardiac remodeling in mice. JCI Insight. 8(13). 7 indexed citations
6.
Skryabin, Boris V., et al.. (2022). Reference Genes across Nine Brain Areas of Wild Type and Prader-Willi Syndrome Mice: Assessing Differences in Igfbp7, Pcsk1, Nhlh2 and Nlgn3 Expression. International Journal of Molecular Sciences. 23(15). 8729–8729. 5 indexed citations
7.
O’Reilly, Molly, Christopher O’Shea, Jasmeet S. Reyat, et al.. (2022). Familial atrial fibrillation mutation M1875T-SCN5A increases early sodium current and dampens the effect of flecainide. EP Europace. 25(3). 1152–1161. 12 indexed citations
8.
Heinick, Alexander, Frank U. Müller, Timofey S. Rozhdestvensky, et al.. (2022). Impaired myocellular Ca2+ cycling in protein phosphatase PP2A-B56α KO mice is normalized by β-adrenergic stimulation. Journal of Biological Chemistry. 298(9). 102362–102362. 4 indexed citations
9.
Raabe, Carsten A., et al.. (2021). A Comprehensive Review of Genetically Engineered Mouse Models for Prader-Willi Syndrome Research. International Journal of Molecular Sciences. 22(7). 3613–3613. 14 indexed citations
10.
Bork, Nadja I., Cristina E. Molina, B. Reiter, et al.. (2021). Rise of cGMP by partial phosphodiesterase-3A degradation enhances cardioprotection during hypoxia. Redox Biology. 48. 102179–102179. 8 indexed citations
11.
Eddiry, Sanaa, Gwénaëlle Diene, Catherine Molinas, et al.. (2021). SNORD116 and growth hormone therapy impact IGFBP7 in Prader–Willi syndrome. Genetics in Medicine. 23(9). 1664–1672. 12 indexed citations
12.
Li, Xinping, et al.. (2020). Circular RNA Encoded Amyloid Beta peptides—A Novel Putative Player in Alzheimer’s Disease. Cells. 9(10). 2196–2196. 37 indexed citations
13.
Gorinski, Nataliya, Daniel Wojciechowski, Daria Guseva, et al.. (2020). DHHC7-mediated palmitoylation of the accessory protein barttin critically regulates the functions of ClC-K chloride channels. Journal of Biological Chemistry. 295(18). 5970–5983. 9 indexed citations
14.
Skryabin, Boris V., Johannes Roth, Sven G. Meuth, et al.. (2020). Pervasive head-to-tail insertions of DNA templates mask desired CRISPR-Cas9–mediated genome editing events. Science Advances. 6(7). eaax2941–eaax2941. 53 indexed citations
15.
Herwig, Melissa, Franziska Werner, Marco Abeßer, et al.. (2020). C-type natriuretic peptide moderates titin-based cardiomyocyte stiffness. JCI Insight. 5(22). 31 indexed citations
16.
Fishman, Veniamin, et al.. (2019). DNA barcoding reveals that injected transgenes are predominantly processed by homologous recombination in mouse zygote. Nucleic Acids Research. 48(2). 719–735. 21 indexed citations
17.
Dankworth, Beatrice, Martin Kruse, Michael Hartmann, et al.. (2011). A cardiac pathway of cyclic GMP-independent signaling of guanylyl cyclase A, the receptor for atrial natriuretic peptide. Proceedings of the National Academy of Sciences. 108(45). 18500–18505. 35 indexed citations
18.
Bäumer, Nicole, Lara Tickenbrock, Petra Tschanter, et al.. (2011). Inhibitor of Cyclin-dependent Kinase (CDK) Interacting with Cyclin A1 (INCA1) Regulates Proliferation and Is Repressed by Oncogenic Signaling. Journal of Biological Chemistry. 286(32). 28210–28222. 19 indexed citations
19.
Holtwick, Rita, Martin van Eickels, Boris V. Skryabin, et al.. (2003). Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A. Journal of Clinical Investigation. 111(9). 1399–1407. 290 indexed citations
20.
Skryabin, Boris V. & Claudia Schmauss. (1997). Enhanced selection for homologous-recombinant embryonic stem cell clones with a neomycin phosphotransferase gene in antisense orientation. Transgenic Research. 6(1). 27–35. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Breakdown of academic impact, for papers by Luca Cartegni Breakdown of academic impact, for papers by Hsiao‐Huei Chen Breakdown of academic impact, for papers by Karl Pfeifer Breakdown of academic impact, for papers by Marcelo A. Nóbrega Breakdown of academic impact, for papers by Douglas S. Annis Breakdown of academic impact, for papers by Margaret Fox Breakdown of academic impact, for papers by Karen Wolburg‐Buchholz Breakdown of academic impact, for papers by Philippe Chafey Breakdown of academic impact, for papers by Jorge Laborda Breakdown of academic impact, for papers by Magali Williamson
Rankless by CCL
2026