Kafait U. Malik

7.7k total citations
226 papers, 6.4k citations indexed

About

Kafait U. Malik is a scholar working on Biochemistry, Molecular Biology and Physiology. According to data from OpenAlex, Kafait U. Malik has authored 226 papers receiving a total of 6.4k indexed citations (citations by other indexed papers that have themselves been cited), including 88 papers in Biochemistry, 84 papers in Molecular Biology and 76 papers in Physiology. Recurrent topics in Kafait U. Malik's work include Eicosanoids and Hypertension Pharmacology (88 papers), Nitric Oxide and Endothelin Effects (67 papers) and Hormonal Regulation and Hypertension (48 papers). Kafait U. Malik is often cited by papers focused on Eicosanoids and Hypertension Pharmacology (88 papers), Nitric Oxide and Endothelin Effects (67 papers) and Hormonal Regulation and Hypertension (48 papers). Kafait U. Malik collaborates with scholars based in United States, Türkiye and Canada. Kafait U. Malik's co-authors include Alberto Nasjletti, J C McGiff, Mubarack M. Muthalif, Anne M. Estes, Norberto A. Terragno, Ibrahim F. Benter, Andrew J. Lonigro, John C. McGiff, Jean‐Hugues Parmentier and Dean D. Schwartz and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation Research.

In The Last Decade

Kafait U. Malik

225 papers receiving 6.1k citations

Peers

Kafait U. Malik
Alberto Nasjletti United States
Richard J. Roman United States
Tianxin Yang United States
J C McGiff United States
Richard J. Roman United States
Alberto Nasjletti United States
Kafait U. Malik
Citations per year, relative to Kafait U. Malik Kafait U. Malik (= 1×) peers Alberto Nasjletti

Countries citing papers authored by Kafait U. Malik

Since Specialization
Citations

This map shows the geographic impact of Kafait U. Malik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kafait U. Malik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kafait U. Malik more than expected).

Fields of papers citing papers by Kafait U. Malik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kafait U. Malik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kafait U. Malik. The network helps show where Kafait U. Malik may publish in the future.

Co-authorship network of co-authors of Kafait U. Malik

This figure shows the co-authorship network connecting the top 25 collaborators of Kafait U. Malik. A scholar is included among the top collaborators of Kafait U. Malik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kafait U. Malik. Kafait U. Malik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Dutta, Shubha Ranjan, Purnima Singh, & Kafait U. Malik. (2023). Ovariectomy Via 12/15-lipoxygenase Augments Angiotensin II-Induced Hypertension and Its Pathogenesis in Female Mice. Hypertension. 80(6). 1245–1257. 9 indexed citations
3.
Chen, Xingyong, Ling Chen, Zhengjun Wang, et al.. (2022). White matter damage as a consequence of vascular dysfunction in a spontaneous mouse model of chronic mild chronic hypoperfusion with eNOS deficiency. Molecular Psychiatry. 27(11). 4754–4769. 32 indexed citations
4.
Pingili, Ajeeth K., et al.. (2021). Testosterone Metabolite 6β‐Hydroxytestosterone Contributes to Angiotensin II‐Induced Abdominal Aortic Aneurysms in Apoe –/– Male Mice. Journal of the American Heart Association. 10(7). e018536–e018536. 9 indexed citations
5.
Song, Chi Young, Purnima Singh, Mustafa Motiwala, et al.. (2021). 2-Methoxyestradiol Ameliorates Angiotensin II–Induced Hypertension by Inhibiting Cytosolic Phospholipase A 2 α Activity in Female Mice. Hypertension. 78(5). 1368–1381. 7 indexed citations
6.
Sari, Ayse Nihal, Bahar Tunçtan, Ayşegül Görür, et al.. (2017). Activation of mTOR/IκB-α/NF-κB pathway contributes to LPS-induced hypotension and inflammation in rats. European Journal of Pharmacology. 802. 7–19. 62 indexed citations
7.
Xue, Yue-Qiang, Tao Ma, Xiaofang Wang, et al.. (2015). Partial eNOS deficiency causes spontaneous thrombotic cerebral infarction, amyloid angiopathy and cognitive impairment. Molecular Neurodegeneration. 10(1). 24–24. 94 indexed citations
9.
Jennings, Brett L., Michael May, Anne M. Estes, et al.. (2014). Cytochrome P450 1B1 contributes to increased blood pressure and cardiovascular and renal dysfunction in spontaneously hypertensive rats. Cardiovascular Drugs and Therapy. 28(2). 145–161. 28 indexed citations
10.
Tunçtan, Bahar, Ayse Nihal Sari, C. Kemal Buharalıoğlu, et al.. (2012). NS-398 reverses hypotension in endotoxemic rats: Contribution of eicosanoids, NO, and peroxynitrite. Prostaglandins & Other Lipid Mediators. 104-105. 93–108. 17 indexed citations
11.
Kalyankrishna, Shailaja, Jean‐Hugues Parmentier, & Kafait U. Malik. (2002). Arachidonic acid-derived oxidation products initiate apoptosis in vascular smooth muscle cells. Prostaglandins & Other Lipid Mediators. 70(1-2). 13–29. 23 indexed citations
12.
Muthalif, Mubarack M., et al.. (2001). Small GTP binding protein Ras contributes to norepinephrine-induced mitogenesis of vascular smooth muscle cells. Prostaglandins & Other Lipid Mediators. 65(1). 33–43. 26 indexed citations
13.
14.
Sehic, E., Yongsen Ruan, & Kafait U. Malik. (1997). Mechanism of Resistance to α-Adrenergic Receptor Antagonists of Renal Nerve Stimulation-Induced Vasoconstriction at Low Frequencies. Journal of Cardiovascular Pharmacology. 29(1). 97–108. 2 indexed citations
15.
Sehic, E., Yongsen Ruan, & Kafait U. Malik. (1994). Attenuation by alpha,beta-methylenadenosine-5'-triphosphate of periarterial nerve stimulation-induced renal vasoconstriction is not due to desensitization of purinergic receptors.. Journal of Pharmacology and Experimental Therapeutics. 271(2). 983–992. 6 indexed citations
16.
Malik, Kafait U., et al.. (1993). Neuropeptide Y modulates the vascular response to periarterial nerve stimulation primarily by a postjunctional action in the isolated perfused rat kidney.. Journal of Pharmacology and Experimental Therapeutics. 266(3). 1321–1329. 22 indexed citations
17.
Adeagbo, Ayotunde S.O. & Kafait U. Malik. (1990). Endothelium‐dependent and BRL 34915‐induced vasodilatation in rat isolated perfused mesenteric arteries: role of G‐proteins, K+ and calcium channels. British Journal of Pharmacology. 100(3). 427–434. 33 indexed citations
18.
Jaiswal, Neelam & Kafait U. Malik. (1990). Prostacyclin synthesis elicited by cholinergic agonists is linked to activation of M2α and M2β muscarinic receptors in the rabbit aorta. Prostaglandins. 39(3). 267–280. 10 indexed citations
19.
Malik, Kafait U.. (1978). Prostaglandins--modulation of adrenergic nervous system.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 37(2). 203–7. 49 indexed citations
20.
McGiff, J C, Kafait U. Malik, & Norberto A. Terragno. (1976). Prostaglandins as determinants of vascular reactivity.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 35(12). 2382–7. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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