Belma Korkmaz

490 total citations
27 papers, 437 citations indexed

About

Belma Korkmaz is a scholar working on Physiology, Biochemistry and Immunology. According to data from OpenAlex, Belma Korkmaz has authored 27 papers receiving a total of 437 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Physiology, 21 papers in Biochemistry and 7 papers in Immunology. Recurrent topics in Belma Korkmaz's work include Eicosanoids and Hypertension Pharmacology (21 papers), Nitric Oxide and Endothelin Effects (20 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (5 papers). Belma Korkmaz is often cited by papers focused on Eicosanoids and Hypertension Pharmacology (21 papers), Nitric Oxide and Endothelin Effects (20 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (5 papers). Belma Korkmaz collaborates with scholars based in Türkiye, United States and Germany. Belma Korkmaz's co-authors include Bahar Tunçtan, C. Kemal Buharalıoğlu, Kafait U. Malik, Seyhan Şahan-Fırat, John R. Falck, Ayse Nihal Sari, Lülüfer Tamer, Uğur Atik, Mehmet Sami Serin and Wolf-Hagen Schunck and has published in prestigious journals such as The FASEB Journal, European Journal of Pharmacology and Pharmacological Research.

In The Last Decade

Belma Korkmaz

27 papers receiving 429 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Belma Korkmaz Türkiye 14 211 153 125 94 72 27 437
Ayse Nihal Sari Türkiye 12 136 0.6× 89 0.6× 181 1.4× 59 0.6× 102 1.4× 24 426
Seyhan Şahan-Fırat Türkiye 17 250 1.2× 157 1.0× 268 2.1× 139 1.5× 106 1.5× 40 692
Ludmila Kazdová Czechia 13 83 0.4× 201 1.3× 184 1.5× 112 1.2× 37 0.5× 24 584
Yeshao Wen United States 11 144 0.7× 176 1.2× 296 2.4× 134 1.4× 150 2.1× 12 748
Shinsuke Kiritoshi Japan 6 56 0.3× 124 0.8× 205 1.6× 97 1.0× 70 1.0× 6 575
Frank Fan Zhang United States 7 183 0.9× 67 0.4× 145 1.2× 154 1.6× 19 0.3× 7 414
Tricia M. Miller United States 11 212 1.0× 57 0.4× 181 1.4× 110 1.2× 18 0.3× 14 529
Alexandra K. Leamy United States 6 121 0.6× 154 1.0× 270 2.2× 176 1.9× 26 0.4× 8 742
Larry N Agbor United States 16 136 0.6× 108 0.7× 258 2.1× 104 1.1× 47 0.7× 22 575
Marc‐André Laplante Canada 10 105 0.5× 177 1.2× 137 1.1× 89 0.9× 49 0.7× 14 507

Countries citing papers authored by Belma Korkmaz

Since Specialization
Citations

This map shows the geographic impact of Belma Korkmaz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Belma Korkmaz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Belma Korkmaz more than expected).

Fields of papers citing papers by Belma Korkmaz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Belma Korkmaz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Belma Korkmaz. The network helps show where Belma Korkmaz may publish in the future.

Co-authorship network of co-authors of Belma Korkmaz

This figure shows the co-authorship network connecting the top 25 collaborators of Belma Korkmaz. A scholar is included among the top collaborators of Belma Korkmaz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Belma Korkmaz. Belma Korkmaz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sari, Ayse Nihal, et al.. (2018). NF‐κB activation mediates LPS‐or zymosan‐induced hypotension and inflammation reversed by BAY61‐3606, a selective Syk inhibitor, in rat models of septic and non‐septic shock. Clinical and Experimental Pharmacology and Physiology. 46(2). 173–182. 11 indexed citations
3.
Sari, Ayse Nihal, C. Kemal Buharalıoğlu, Belma Korkmaz, et al.. (2013). Contribution of RhoA/Rho-kinase/MEK1/ERK1/2/iNOS pathway to ischemia/reperfusion-induced oxidative/nitrosative stress and inflammation leading to distant and target organ injury in rats. European Journal of Pharmacology. 723. 234–245. 20 indexed citations
4.
Şahan-Fırat, Seyhan, Belma Korkmaz, Hatice Kalkan Yı̇ldırım, et al.. (2012). Increased Production of Nitric Oxide Mediates Selective Organ-Specific Effects of Endotoxin on Oxidative Stress. Anti-Inflammatory & Anti-Allergy Agents in Medicinal Chemistry. 11(2). 161–172. 5 indexed citations
5.
Tunçtan, Bahar, Ayse Nihal Sari, C. Kemal Buharalıoğlu, et al.. (2012). NS-398 reverses hypotension in endotoxemic rats: Contribution of eicosanoids, NO, and peroxynitrite. Prostaglandins & Other Lipid Mediators. 104-105. 93–108. 17 indexed citations
6.
Korkmaz, Belma, C. Kemal Buharalıoğlu, Abdullah Tuncay Demiryürek, et al.. (2012). Contribution of MEK1/ERK1/2/iNOS Pathway to Oxidative Stress and Decreased Caspase-3 Activity in Endotoxemic Rats. Anti-Inflammatory & Anti-Allergy Agents in Medicinal Chemistry. 11(3). 243–252. 4 indexed citations
7.
Tunçtan, Bahar, Belma Korkmaz, Ayse Nihal Sari, et al.. (2012). A Novel Treatment Strategy for Sepsis and Septic Shock Based on the Interactions between Prostanoids, Nitric Oxide, and 20-Hydroxyeicosatetraenoic Acid. Anti-Inflammatory & Anti-Allergy Agents in Medicinal Chemistry. 11(2). 121–150. 28 indexed citations
9.
Buharalıoğlu, C. Kemal, Belma Korkmaz, Seyhan Şahan-Fırat, et al.. (2011). Piroxicam Reverses Endotoxin‐Induced Hypotension in Rats: Contribution of Vasoactive Eicosanoids and Nitric Oxide. Basic & Clinical Pharmacology & Toxicology. 109(3). 186–194. 9 indexed citations
10.
Korkmaz, Belma, C. Kemal Buharalıoğlu, Seyhan Şahan-Fırat, et al.. (2010). A Synthetic Analogue of 20‐HETE, 5,14‐HEDGE, Reverses Endotoxin‐Induced Hypotension via Increased 20‐HETE Levels Associated with Decreased iNOS Protein Expression and Vasodilator Prostanoid Production in Rats. Basic & Clinical Pharmacology & Toxicology. 106(5). 378–388. 23 indexed citations
11.
Tunçtan, Bahar, Belma Korkmaz, C. Kemal Buharalıoğlu, et al.. (2010). Contribution of Vasoactive Eicosanoids and Nitric Oxide Production to the Effect of Selective Cyclooxygenase‐2 Inhibitor, NS‐398, on Endotoxin‐Induced Hypotension in Rats. Basic & Clinical Pharmacology & Toxicology. 107(5). 877–882. 21 indexed citations
14.
Tunçtan, Bahar, Belma Korkmaz, C. Kemal Buharalıoğlu, et al.. (2008). A 20-HYDROXYEICOSATETRAENOIC ACID AGONIST, N-[20-HYDROXYEICOSA-5(Z),14(Z)-DIENOYL]GLYCINE, OPPOSES THE FALL IN BLOOD PRESSURE AND VASCULAR REACTIVITY IN ENDOTOXIN-TREATED RATS. Shock. 30(3). 329–335. 29 indexed citations
15.
Korkmaz, Belma, et al.. (2008). Endotoksemik Sıçanların Karaciğerinde Artan Oksidatif Stres Üzerinde İndüklenebilir Nitrik Oksit Sentaz İnhibisyonunun Etkisi. DergiPark (Istanbul University). 1 indexed citations
17.
Tunçtan, Bahar, Belma Korkmaz, Hatice Kalkan Yı̇ldırım, et al.. (2006). Reversal of endotoxin-induced hypotension by inhibition of inducible nitric oxide synthase activity is associated with improved oxidative status in rat heart, aorta and mesenteric artery. TURKISH JOURNAL OF MEDICAL SCIENCES. 36(2). 71–80. 9 indexed citations
18.
Korkmaz, Belma, et al.. (2006). Involvement of calcium/calmodulin-dependent protein kinase II to endotoxin-induced vascular hyporeactivity in rat superior mesenteric artery. Pharmacological Research. 54(3). 208–218. 7 indexed citations
19.
Tunçtan, Bahar, et al.. (2005). Nitric oxide reverses endotoxin-induced inflammatory hyperalgesia via inhibition of prostacyclin production in mice. Pharmacological Research. 53(2). 177–192. 24 indexed citations
20.
Ark, Mustafa, et al.. (2004). Effects of HA-1077 and Y-27632, Two Rho-Kinase Inhibitors, in the Human Umbilical Artery. Cell Biochemistry and Biophysics. 41(3). 331–342. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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