Jouni Vesa

3.2k total citations · 1 hit paper
35 papers, 2.5k citations indexed

About

Jouni Vesa is a scholar working on Molecular Biology, Physiology and Cell Biology. According to data from OpenAlex, Jouni Vesa has authored 35 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 16 papers in Physiology and 15 papers in Cell Biology. Recurrent topics in Jouni Vesa's work include Lysosomal Storage Disorders Research (16 papers), Cellular transport and secretion (9 papers) and Autophagy in Disease and Therapy (6 papers). Jouni Vesa is often cited by papers focused on Lysosomal Storage Disorders Research (16 papers), Cellular transport and secretion (9 papers) and Autophagy in Disease and Therapy (6 papers). Jouni Vesa collaborates with scholars based in United States, Finland and United Kingdom. Jouni Vesa's co-authors include Virginia Kimonis, Leena Peltonen, Elina Hellsten, Juhani Rapola, Pirkko Santavuori, Sandra L. Hofmann, Linda A. Verkruyse, Giles Watts, Emilie Tresse and Nico P. Dantuma and has published in prestigious journals such as Nature, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Jouni Vesa

34 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mutations in the palmitoyl protein thioesterase gene causing infantile neuronal ceroid lipofuscinosis

1995 606 citations Jouni Vesa, Elina Hellsten et al. profile →

Peers

Jouni Vesa

PHYSI

EPIDE

NEURO

Christine R. Kaneski United States

Özlem Göker-Alpan United States

Frans W. Verheijen Netherlands

Susan L. Cotman United States

Lluı̈sa Vilageliu Spain

Thomas M. Durcan Canada

Anat Bar‐Shira Israel

Livia Poënaru France

Adeline Vanderver United States

Christine R. Kaneski United States

Jouni Vesa

1.1k ×0.8

1.3k ×1.3

PHYSI

833 ×0.9

425 ×0.9

EPIDE

173 ×0.5

NEURO

Citations per year, relative to Jouni Vesa Jouni Vesa (= 1×) peers Christine R. Kaneski

Countries citing papers authored by Jouni Vesa

Since Specialization

Citations

This map shows the geographic impact of Jouni Vesa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jouni Vesa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jouni Vesa more than expected).

Fields of papers citing papers by Jouni Vesa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jouni Vesa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jouni Vesa. The network helps show where Jouni Vesa may publish in the future.

Co-authorship network of co-authors of Jouni Vesa

This figure shows the co-authorship network connecting the top 25 collaborators of Jouni Vesa. A scholar is included among the top collaborators of Jouni Vesa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jouni Vesa. Jouni Vesa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Vesa, Jouni, Antoine Chaillon, Gabriel A. Wagner, et al.. (2017). Increased HIV-1 superinfection risk in carriers of specific human leukocyte antigen alleles. AIDS. 31(8). 1149–1158. 7 indexed citations

Fabritius, Anna‐Liisa, et al.. (2016). Data on characterizing the gene expression patterns of neuronal ceroid lipofuscinosis genes: CLN1, CLN2, CLN3, CLN5 and their association to interneuron and neurotransmission markers: Parvalbumin and Somatostatin. Data in Brief. 8. 741–749. 6 indexed citations

Nalbandian, Angèle, Katrina J. Llewellyn, Hailing Su, et al.. (2015). In vitro studies in VCP-associated multisystem proteinopathy suggest altered mitochondrial bioenergetics. Mitochondrion. 22. 1–8. 24 indexed citations

Nalbandian, Angèle, Svetlana Ghimbovschi, Zuyi Wang, et al.. (2014). Global Gene Expression Profiling in R155H Knock-In Murine Model of VCP Disease. Clinical and Translational Science. 8(1). 8–16. 3 indexed citations

Uusi-Rauva, Kristiina, Aija Kyttälä, Rik van der Kant, et al.. (2012). Neuronal ceroid lipofuscinosis protein CLN3 interacts with motor proteins and modifies location of late endosomal compartments. Cellular and Molecular Life Sciences. 69(12). 2075–2089. 63 indexed citations

Nalbandian, Angèle, Katrina J. Llewellyn, Mallikarjun Badadani, et al.. (2012). A progressive translational mouse model of human valosin‐containing protein disease: The VCP^R155H/+ mouse. Muscle & Nerve. 47(2). 260–270. 51 indexed citations

Nalbandian, Angèle, Sandra Donkervoort, Eric Dec, et al.. (2011). The Multiple Faces of Valosin-Containing Protein-Associated Diseases: Inclusion Body Myopathy with Paget’s Disease of Bone, Frontotemporal Dementia, and Amyotrophic Lateral Sclerosis. Journal of Molecular Neuroscience. 45(3). 522–531. 106 indexed citations

Vesa, Jouni, Hailing Su, Giles D. Watts, et al.. (2009). Valosin containing protein associated inclusion body myopathy: abnormal vacuolization, autophagy and cell fusion in myoblasts. Neuromuscular Disorders. 19(11). 766–772. 54 indexed citations

Kimonis, Virginia, Erin Fulchiero, Jouni Vesa, & Giles Watts. (2008). VCP disease associated with myopathy, Paget disease of bone and frontotemporal dementia: Review of a unique disorder. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1782(12). 744–748. 169 indexed citations

10.

Vesa, Jouni, et al.. (2005). Molecular and cellular characterization of the Down syndrome critical region protein 2. Biochemical and Biophysical Research Communications. 328(1). 235–242. 9 indexed citations

11.

Kopra, Outi, Jouni Vesa, Carina von Schantz, et al.. (2004). A mouse model for Finnish variant late infantile neuronal ceroid lipofuscinosis, CLN5, reveals neuropathology associated with early aging. Human Molecular Genetics. 13(23). 2893–2906. 72 indexed citations

12.

Jalanko, Anu, Jouni Vesa, Tuula Manninen, et al.. (2004). Mice with Ppt1Δex4 mutation replicate the INCL phenotype and show an inflammation-associated loss of interneurons. Neurobiology of Disease. 18(1). 226–241. 68 indexed citations

13.

Visapää, Ilona, Vineta Fellman, Jouni Vesa, et al.. (2002). GRACILE Syndrome, a Lethal Metabolic Disorder with Iron Overload, Is Caused by a Point Mutation in BCS1L. The American Journal of Human Genetics. 71(4). 863–876. 199 indexed citations

14.

Vesa, Jouni, et al.. (2000). Palmitoylation of the p75 neurotrophin receptor has no effect on its interaction with TrkA or on TrkA-mediated down-regulation of cell adhesion molecules. Journal of Neuroscience Research. 62(2). 225–233. 7 indexed citations

15.

Vesa, Jouni, Alex Krüttgen, & Eric M. Shooter. (2000). p75 Reduces TrkB Tyrosine Autophosphorylation in Response to Brain-derived Neurotrophic Factor and Neurotrophin 4/5. Journal of Biological Chemistry. 275(32). 24414–24420. 44 indexed citations

16.

Hellsten, Elina, Jouni Vesa, Anu Jalanko, & Leena Peltonen. (1997). From Locus to Cellular Disturbances: Positional Cloning of the Infantile Neuronal Ceroid Lipofuscinosis Gene. Neuropediatrics. 28(1). 9–11. 7 indexed citations

17.

Vesa, Jouni, Elina Hellsten, Beverly S. Emanuel, et al.. (1994). Assignment of sterol carrier protein X/sterol carrier protein 2 to 1p32 and its exclusion as the causative gene for infantile neuronal ceroid lipofuscinosis. Human Molecular Genetics. 3(2). 341–346. 10 indexed citations

18.

Hellsten, Elina, Jouni Vesa, Marcy C. Speer, et al.. (1993). Refined Assignment of the Infantile Neuronal Ceroid Lipofuscinosis (INCL, CLN1) Locus at 1p32: Incorporation of Linkage Disequilibrium in Multipoint Analysis. Genomics. 16(3). 720–725. 58 indexed citations

19.

Järvelä, Irma, Jouni Vesa, Pirkko Santavuori, Elina Hellsten, & Leena Peltonen. (1992). Molecular Genetics of Neuronal Ceroid Lipofuscinoses. Pediatric Research. 32(6). 645–648. 18 indexed citations

20.

Järvelä, Irma, Juhani Rapola, Leena Peltonen, et al.. (1991). DNA‐based prenatal diagnosis of the infantile form of neuronal ceroid lipofuscinosis (INCL, CLN1). Prenatal Diagnosis. 11(5). 323–328. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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