Jonathan W. Artates

2.1k total citations · 1 hit paper
5 papers, 754 citations indexed

About

Jonathan W. Artates is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Jonathan W. Artates has authored 5 papers receiving a total of 754 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 2 papers in Neurology. Recurrent topics in Jonathan W. Artates's work include Genetic Neurodegenerative Diseases (4 papers), Muscle Physiology and Disorders (3 papers) and Mitochondrial Function and Pathology (2 papers). Jonathan W. Artates is often cited by papers focused on Genetic Neurodegenerative Diseases (4 papers), Muscle Physiology and Disorders (3 papers) and Mitochondrial Function and Pathology (2 papers). Jonathan W. Artates collaborates with scholars based in United States, United Kingdom and Spain. Jonathan W. Artates's co-authors include Don W. Cleveland, Seng H. Cheng, Holly Kordasiewicz, Curt Mazur, Lisa M. Stanek, Andreas Weiss, Edward V. Wancewicz, Lamya S. Shihabuddin, Gene Hung and C. Frank Bennett and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Neuron and Molecular Neurodegeneration.

In The Last Decade

Jonathan W. Artates

4 papers receiving 746 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Sustained Therapeutic Reversal of Huntington's Disease by Transient Repression of Huntingtin Synthesis

2012 563 citations Holly Kordasiewicz, Lisa M. Stanek et al. Neuron profile →

Peers

Jonathan W. Artates

MB

CMN

NEURO

GENET

Lodewijk J.A. Toonen Netherlands

Edith Lopez United States

Karla P. Figueroa United States

Nayeon Lee South Korea

Mário Gomes‐Pereira France

Frédérique Rau France

Armand Soriano United States

Xiuyin Teng United States

Marián Hruška-Plocháň United States

Pabitra K. Sahoo United States

Lodewijk J.A. Toonen Netherlands

Jonathan W. Artates

539 ×0.8

MB

312 ×0.7

CMN

89 ×0.5

NEURO

95 ×0.9

GENET

66 ×1.1

GENET

Citations per year, relative to Jonathan W. Artates Jonathan W. Artates (= 1×) peers Lodewijk J.A. Toonen

Countries citing papers authored by Jonathan W. Artates

Since Specialization

Citations

This map shows the geographic impact of Jonathan W. Artates's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan W. Artates with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan W. Artates more than expected).

Fields of papers citing papers by Jonathan W. Artates

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan W. Artates. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan W. Artates. The network helps show where Jonathan W. Artates may publish in the future.

Co-authorship network of co-authors of Jonathan W. Artates

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan W. Artates. A scholar is included among the top collaborators of Jonathan W. Artates based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan W. Artates. Jonathan W. Artates is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

5 of 5 papers shown

1.

Beccari, Melinda S., Michael W. Baughn, Jonathan W. Artates, et al.. (2025). Stathmin-2 enhances motor axon regeneration after injury independent of its binding to tubulin. Proceedings of the National Academy of Sciences. 122(21). e2502294122–e2502294122.

2.

Vazquez‐Sanchez, Sonia, F. Gasset-Rosa, Melissa McAlonis‐Downes, et al.. (2024). Frontotemporal dementia-like disease progression elicited by seeded aggregation and spread of FUS. Molecular Neurodegeneration. 19(1). 46–46. 3 indexed citations

3.

Gasset-Rosa, F., Carlos Chillón-Marinas, Alexander Goginashvili, et al.. (2017). Polyglutamine-Expanded Huntingtin Exacerbates Age-Related Disruption of Nuclear Integrity and Nucleocytoplasmic Transport. Neuron. 94(1). 48–57.e4. 171 indexed citations

4.

Hu, Jiaxin, Jing Liu, Dongbo Yu, et al.. (2014). Exploring the Effect of Sequence Length and Composition on Allele-Selective Inhibition of Human Huntingtin Expression by Single-Stranded Silencing RNAs. Nucleic Acid Therapeutics. 24(3). 199–209. 17 indexed citations

5.

Kordasiewicz, Holly, Lisa M. Stanek, Edward V. Wancewicz, et al.. (2012). Sustained Therapeutic Reversal of Huntington's Disease by Transient Repression of Huntingtin Synthesis. Neuron. 74(6). 1031–1044. 563 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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