Jonathan J. Hulce
- Organic Chemistry top 5%
- Click Chemistry and Applications 4
- Pharmaceutical Science top 5%
- Molecular Biology top 10%
- Sphingolipid Metabolism and Signaling 2
- Biochemistry top 10%
- Cell Biology top 10%
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- Peptidase Inhibition and Analysis 3
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- Monoclonal and Polyclonal Antibodies Research 2
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- Pancreatic function and diabetes 2
- Cardiovascular, Neuropeptides, and Oxidative Stress Research 1
- Cholesterol and Lipid Metabolism 1
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- Adenosine and Purinergic Signaling 1
- Co-authors
- Benjamin F. CravattArmand B. CognettaMicah J. NiphakisSarah E. TullyEnrique SáezYu LiuSuhua LiAndrea Galmozzi
- Partner nations
- United StatesGermany
In The Last Decade
Jonathan J. Hulce
11 papers receiving 1.1k citations
Hit Papers
Peers
Comparison fields: 5 of 80
- Organic Chemistry 504
- Pharmaceutical Science 89
- Molecular Biology 797
- Biochemistry 62
- Cell Biology 132
Countries citing papers authored by Jonathan J. Hulce
This map shows the geographic impact of Jonathan J. Hulce's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan J. Hulce with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan J. Hulce more than expected).
Fields of papers citing papers by Jonathan J. Hulce
This network shows the impact of papers produced by Jonathan J. Hulce. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan J. Hulce. The network helps show where Jonathan J. Hulce may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Jonathan J. Hulce, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 31 | |
| 2 | 2019 | 30 | |
| 3 | 2018 | 54 | |
| 4 | 2016 | 63 | |
| 5 | Arylfluorosulfates Inactivate Intracellular Lipid Binding Protein(s) through Chemoselective SuFEx Reaction with a Binding Site Tyr Residuebreakdown → | 2016 | 248 |
| 6 | 2015 | 49 | |
| 7 | 2014 | 265 | |
| 8 | An in Vivo Active Carbamate-based Dual Inhibitor of Lysophospholipase 1 (LYPLA1) and Lysophospholipase 2 (LYPLA2) | 2014 | 2 |
| 9 | 2013 | 345 | |
| 10 | 2009 | 7 | |
| 11 | 2009 | 49 |
About Jonathan J. Hulce
Jonathan J. Hulce is a scholar working on Physiology, Pharmaceutical Science and Organic Chemistry, having authored 11 papers that have together received 1.1k indexed citations. Recurring topics across this work include Click Chemistry and Applications (4 papers), Peptidase Inhibition and Analysis (3 papers), Sphingolipid Metabolism and Signaling (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), Pancreatic function and diabetes (2 papers), Adenosine and Purinergic Signaling (1 paper), Cardiovascular, Neuropeptides, and Oxidative Stress Research (1 paper) and Cholesterol and Lipid Metabolism (1 paper). The work is most often cited by research in Organic Chemistry (504 citations), Pharmaceutical Science (89 citations) and Molecular Biology (797 citations). Jonathan J. Hulce has collaborated with scholars based in United States and Germany. Frequent co-authors include Benjamin F. Cravatt, Armand B. Cognetta, Micah J. Niphakis, Sarah E. Tully, Enrique Sáez, Yu Liu, Suhua Li, Andrea Galmozzi, Wentao Chen and Ian A. Wilson.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.