Martin Hruska

901 total citations
16 papers, 669 citations indexed

About

Martin Hruska is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Martin Hruska has authored 16 papers receiving a total of 669 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cellular and Molecular Neuroscience, 5 papers in Molecular Biology and 2 papers in Cognitive Neuroscience. Recurrent topics in Martin Hruska's work include Neuroscience and Neuropharmacology Research (11 papers), Axon Guidance and Neuronal Signaling (7 papers) and Photoreceptor and optogenetics research (5 papers). Martin Hruska is often cited by papers focused on Neuroscience and Neuropharmacology Research (11 papers), Axon Guidance and Neuronal Signaling (7 papers) and Photoreceptor and optogenetics research (5 papers). Martin Hruska collaborates with scholars based in United States, Germany and Austria. Martin Hruska's co-authors include Matthew B. Dalva, Nathan T. Henderson, Sylvain J. Le Marchand, Haani Jafri, Rae Nishi, Matthew S. Kayser, Mark J. Nolt, R. Suzanne Zukin, Jessica A. Murphy and Ying Lin and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Nature Communications.

In The Last Decade

Martin Hruska

15 papers receiving 668 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Martin Hruska United States 10 471 287 96 95 72 16 669
Tal Laviv Israel 9 414 0.9× 329 1.1× 54 0.6× 111 1.2× 79 1.1× 16 661
Hitomi Matsuno Japan 12 538 1.1× 430 1.5× 162 1.7× 131 1.4× 93 1.3× 19 927
Kalen Berry United States 13 376 0.8× 321 1.1× 77 0.8× 186 2.0× 97 1.3× 20 793
Karen Perez de Arce United States 9 266 0.6× 287 1.0× 116 1.2× 40 0.4× 51 0.7× 10 561
Cezar M. Tigaret United Kingdom 12 595 1.3× 396 1.4× 88 0.9× 179 1.9× 55 0.8× 14 765
Lesley A. Colgan United States 13 449 1.0× 494 1.7× 81 0.8× 126 1.3× 32 0.4× 18 819
Lenka Mikasová France 9 445 0.9× 318 1.1× 67 0.7× 72 0.8× 49 0.7× 9 809
Alberto Pérez‐Alvarez Spain 13 400 0.8× 302 1.1× 61 0.6× 118 1.2× 75 1.0× 22 733
А. P. Bolshakov Russia 14 243 0.5× 368 1.3× 52 0.5× 44 0.5× 63 0.9× 50 641
Ming-Chia Lee United States 7 419 0.9× 382 1.3× 105 1.1× 127 1.3× 28 0.4× 9 728

Countries citing papers authored by Martin Hruska

Since Specialization
Citations

This map shows the geographic impact of Martin Hruska's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Hruska with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Hruska more than expected).

Fields of papers citing papers by Martin Hruska

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin Hruska. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Hruska. The network helps show where Martin Hruska may publish in the future.

Co-authorship network of co-authors of Martin Hruska

This figure shows the co-authorship network connecting the top 25 collaborators of Martin Hruska. A scholar is included among the top collaborators of Martin Hruska based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Hruska. Martin Hruska is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Vargas, Karina J., et al.. (2025). Combining nanobody labeling with STED microscopy reveals input-specific and layer-specific organization of neocortical synapses. PLoS Biology. 23(4). e3002649–e3002649. 1 indexed citations
2.
Schulte, Clemens, Katherina Hemmen, Thomas-Otavio Peulen, et al.. (2025). eSylites: Synthetic Probes for Visualization and Topographic Mapping of Single Excitatory Synapses. Journal of the American Chemical Society. 147(18). 15261–15280.
3.
Hruska, Martin, et al.. (2024). Trans-synaptic Association of Vesicular Zinc Transporter 3 and Shank3 Supports Synapse-Specific Dendritic Spine Structure and Function in the Mouse Auditory Cortex. Journal of Neuroscience. 44(28). e0619242024–e0619242024. 6 indexed citations
4.
Hruska, Martin, et al.. (2022). Nanoscale rules governing the organization of glutamate receptors in spine synapses are subunit specific. Nature Communications. 13(1). 920–920. 31 indexed citations
5.
Henderson, Nathan T., Sylvain J. Le Marchand, Martin Hruska, et al.. (2019). Ephrin-B3 controls excitatory synapse density through cell-cell competition for EphBs. eLife. 8. 11 indexed citations
6.
Hruska, Martin, Nathan T. Henderson, Sylvain J. Le Marchand, Haani Jafri, & Matthew B. Dalva. (2018). Synaptic nanomodules underlie the organization and plasticity of spine synapses. Nature Neuroscience. 21(5). 671–682. 171 indexed citations
7.
Hruska, Martin, et al.. (2018). Levels of Par-1 kinase determine the localization of Bruchpilot at the Drosophila neuromuscular junction synapses. Scientific Reports. 8(1). 16099–16099. 5 indexed citations
8.
Hruska, Martin, Nathan T. Henderson, Nan Xia, Sylvain J. Le Marchand, & Matthew B. Dalva. (2015). Anchoring and synaptic stability of PSD-95 is driven by ephrin-B3. Nature Neuroscience. 18(11). 1594–1605. 49 indexed citations
9.
Dai, Jinxia, Mona Buhusi, Galina P. Demyanenko, et al.. (2013). Neuron Glia-Related Cell Adhesion Molecule (NrCAM) Promotes Topographic Retinocollicular Mapping. PLoS ONE. 8(9). e73000–e73000. 16 indexed citations
10.
Hruska, Martin & Matthew B. Dalva. (2012). Ephrin regulation of synapse formation, function and plasticity. Molecular and Cellular Neuroscience. 50(1). 35–44. 121 indexed citations
11.
Nolt, Mark J., Ying Lin, Martin Hruska, et al.. (2011). EphB Controls NMDA Receptor Function and Synaptic Targeting in a Subunit-Specific Manner. Journal of Neuroscience. 31(14). 5353–5364. 122 indexed citations
12.
Kayser, Matthew S., et al.. (2011). Preferential Control of Basal Dendritic Protrusions by EphB2. PLoS ONE. 6(2). e17417–e17417. 7 indexed citations
13.
McClelland, Andrew C., et al.. (2010). Trans-synaptic EphB2–ephrin–B3 interaction regulates excitatory synapse density by inhibition of postsynaptic MAPK signaling. Proceedings of the National Academy of Sciences. 107(19). 8830–8835. 51 indexed citations
14.
Hruska, Martin, J. R. Keefe, Ayşe B. Tekinay, et al.. (2009). Prostate Stem Cell Antigen Is an Endogenous lynx1-Like Prototoxin That Antagonizes α7-Containing Nicotinic Receptors and Prevents Programmed Cell Death of Parasympathetic Neurons. Journal of Neuroscience. 29(47). 14847–14854. 49 indexed citations
15.
Nishi, Rae, Jutta Stubbusch, Jonathan J. Hulce, et al.. (2009). The cortistatin gene PSS2 rather than the somatostatin gene PSS1 is strongly expressed in developing avian autonomic neurons. The Journal of Comparative Neurology. 518(6). 839–850. 7 indexed citations
16.
Hruska, Martin & Rae Nishi. (2007). Cell-Autonomous Inhibition of α7-Containing Nicotinic Acetylcholine Receptors Prevents Death of Parasympathetic Neurons during Development. Journal of Neuroscience. 27(43). 11501–11509. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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