John M. Streicher

3.0k total citations
83 papers, 2.3k citations indexed

About

John M. Streicher is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, John M. Streicher has authored 83 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Molecular Biology, 47 papers in Cellular and Molecular Neuroscience and 30 papers in Physiology. Recurrent topics in John M. Streicher's work include Neuropeptides and Animal Physiology (39 papers), Pain Mechanisms and Treatments (27 papers) and Receptor Mechanisms and Signaling (26 papers). John M. Streicher is often cited by papers focused on Neuropeptides and Animal Physiology (39 papers), Pain Mechanisms and Treatments (27 papers) and Receptor Mechanisms and Signaling (26 papers). John M. Streicher collaborates with scholars based in United States, Italy and China. John M. Streicher's co-authors include Cynthia L. Bethea, Nick Z. Lu, Chrisana Gundlah, Yibin Wang, Justin LaVigne, Attila Keresztes, Laura Bohn, Wei Lei, Cullen L. Schmid and Edward J. Bilsky and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Journal of Neuroscience.

In The Last Decade

John M. Streicher

81 papers receiving 2.2k citations

Peers

John M. Streicher

CMN

PHYSI

PHARM

Qi Yang China

Gian Marco Leggio Italy

Reza Rahimian Iran

Francisco J. Gil‐Bea Spain

Mitsutoshi Yuzurihara Japan

John E. Piletz United States

Chantelle E. Terrillion United States

Heinz Bönisch Germany

Shupeng Li China

Yinghe Hu China

Qi Yang China

John M. Streicher

831 ×0.8

533 ×0.6

CMN

413 ×0.8

PHYSI

144 ×0.5

203 ×0.8

PHARM

Citations per year, relative to John M. Streicher John M. Streicher (= 1×) peers Qi Yang

Countries citing papers authored by John M. Streicher

Since Specialization

Citations

This map shows the geographic impact of John M. Streicher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John M. Streicher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John M. Streicher more than expected).

Fields of papers citing papers by John M. Streicher

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John M. Streicher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John M. Streicher. The network helps show where John M. Streicher may publish in the future.

Co-authorship network of co-authors of John M. Streicher

This figure shows the co-authorship network connecting the top 25 collaborators of John M. Streicher. A scholar is included among the top collaborators of John M. Streicher based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John M. Streicher. John M. Streicher is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Streicher, John M., et al.. (2025). Terpene blends from Cannabis sativa are cannabimimetic and antinociceptive in a mouse chronic neuropathic pain model via activation of adenosine A2a receptors. Neuroscience Letters. 854. 138205–138205. 1 indexed citations

Cárdenas, Paco, et al.. (2025). Analgesic Properties of the Natural Product Barettin Are Mediated by the 5HT2A/C Receptors (Abstract ID: 165920). Journal of Pharmacology and Experimental Therapeutics. 392(3). 101346–101346.

Szabó, Lajos, Fahad Al‐Obeidi, Mitchell J. Bartlett, et al.. (2023). Structure-Based Design of Glycosylated Oxytocin Analogues with Improved Selectivity and Antinociceptive Activity. ACS Medicinal Chemistry Letters. 14(2). 163–170. 5 indexed citations

Ranaldi, Robert, et al.. (2023). The D3 receptor antagonist SR 21502 reduces cue-induced reinstatement of methamphetamine-seeking in rats. Neuroscience Letters. 806. 137237–137237. 1 indexed citations

Streicher, John M., et al.. (2023). Heat shock protein 90 inhibition in the spinal cord upregulates Src kinase in microglia to enhance opioid antinociception in acute pain. Journal of Pharmacology and Experimental Therapeutics. 385. 489–489. 1 indexed citations

Liktor‐Busa, Erika, et al.. (2022). Inhibition of HSP90 Preserves Blood–Brain Barrier Integrity after Cortical Spreading Depression. Pharmaceutics. 14(8). 1665–1665. 5 indexed citations

Langlais, Paul R., et al.. (2022). Extracellular Alterations in pH and K+ Modify the Murine Brain Endothelial Cell Total and Phospho-Proteome. Pharmaceutics. 14(7). 1469–1469. 2 indexed citations

Karom, Mary C., et al.. (2022). Age-Induced Changes in µ-Opioid Receptor Signaling in the Midbrain Periaqueductal Gray of Male and Female Rats. Journal of Neuroscience. 42(32). 6232–6242. 4 indexed citations

Molnár, G, Mitchell J. Bartlett, Lajos Szabó, et al.. (2022). Design and Synthesis of Brain Penetrant Glycopeptide Analogues of PACAP With Neuroprotective Potential for Traumatic Brain Injury and Parkinsonism. PubMed. 1. 14 indexed citations

10.

Keresztes, Attila, Natalie K. Barker, Victor J. Hruby, et al.. (2021). Antagonism of the mu-delta opioid receptor heterodimer enhances opioid antinociception by activating Src and calcium/calmodulin-dependent protein kinase II signaling. Pain. 163(1). 146–158. 16 indexed citations

11.

Lei, Wei, Natalie K. Barker, Sanket J. Mishra, et al.. (2020). Inhibition of Hsp90 in the spinal cord enhances the antinociceptive effects of morphine by activating an ERK-RSK pathway. Science Signaling. 13(630). 14 indexed citations

12.

Khanna, Rajesh, Jie Yu, Xiaofang Yang, et al.. (2019). Targeting the CaVα–CaVβ interaction yields an antagonist of the N-type CaV2.2 channel with broad antinociceptive efficacy. Pain. 160(7). 1644–1661. 35 indexed citations

13.

Streicher, John M.. (2019). The role of heat shock protein 90 in regulating pain, opioid signaling, and opioid antinociception. Vitamins and hormones. 111. 91–103. 9 indexed citations

14.

Shah, Nirav, Sanjay Kumar, Christopher C. Pan, et al.. (2018). TAK1 activation of alpha-TAT1 and microtubule hyperacetylation control AKT signaling and cell growth. Nature Communications. 9(1). 1696–1696. 46 indexed citations

15.

Xie, Jennifer Y., Milena De Felice, Caroline Machado Kopruszinski, et al.. (2017). Kappa opioid receptor antagonists: A possible new class of therapeutics for migraine prevention. Cephalalgia. 37(8). 780–794. 76 indexed citations

16.

Frankowski, Kevin J., Kimberly M. Lovell, Angela M. Phillips, et al.. (2014). Potency enhancement of the κ-opioid receptor antagonist probe ML140 through sulfonamide constraint utilizing a tetrahydroisoquinoline motif. Bioorganic & Medicinal Chemistry. 23(14). 3948–3956. 6 indexed citations

17.

Tarselli, Michael A., Kirsten M. Raehal, John M. Streicher, et al.. (2011). Synthesis of conolidine, a potent non-opioid analgesic for tonic and persistent pain. Nature Chemistry. 3(6). 449–453. 52 indexed citations

18.

Béguin, Cécile, Justin S. Potuzak, Wei Xu, et al.. (2011). Differential signaling properties at the kappa opioid receptor of 12-epi-salvinorin A and its analogues. Bioorganic & Medicinal Chemistry Letters. 22(2). 1023–1026. 22 indexed citations

19.

Streicher, John M., Ken‐ichiro Kamei, Tomo‐o Ishikawa, Harvey R. Herschman, & Yibin Wang. (2010). Compensatory hypertrophy induced by ventricular cardiomyocyte-specific COX-2 expression in mice. Journal of Molecular and Cellular Cardiology. 49(1). 88–94. 24 indexed citations

20.

Bethea, Cynthia L., et al.. (2005). Serotonin-related gene expression in female monkeys with individual sensitivity to stress. Neuroscience. 132(1). 151–166. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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