John H. Viles

6.9k citations

66 papers · 5.5k indexed · 1 hit paper · h-index 36

Molecular Biology top 2%
Physiology top 0.5%
Nutrition and Dietetics top 0.2%
Neurology top 0.5%
Oncology top 5%

Co-authors: Christopher D. Syme Stephen E. J. Rigby David R. Brown Nadine D. Younan Peter E. Wright H. Jane Dyson Fred E. Cohen Stanley B. Prusiner
Topics: Alzheimer's disease research and treatments (33 papers)Prion Diseases and Protein Misfolding (25 papers)Trace Elements in Health (23 papers)
Cited by: Nutrition and Dietetics Neurology Physiology
Journals: Proceedings of the National Academy of Sciences Journal of the American Chemical Society Journal of Biological Chemistry
Partner nations: United Kingdom United States Australia

In The Last Decade

John H. Viles

66 papers receiving 5.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Structure of the recombinant full-length hamster prion protein PrP(29–231): The N terminus is highly flexible

1997 568 citations John H. Viles et al. profile →

Peers

Countries citing papers authored by John H. Viles

Since Specialization

Citations

This map shows the geographic impact of John H. Viles's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John H. Viles with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John H. Viles more than expected).

Fields of papers citing papers by John H. Viles

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John H. Viles. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John H. Viles. The network helps show where John H. Viles may publish in the future.

Co-authorship network of co-authors of John H. Viles

This figure shows the co-authorship network connecting the top 25 collaborators of John H. Viles. A scholar is included among the top collaborators of John H. Viles based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John H. Viles. John H. Viles is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	The p3 peptides (Aβ17-40/42) rapidly form amyloid fibrils that cross-seed with full-length Aβ 2025 ·Nature Communications ·Yao Tian,(unknown),Qi Shang,Hui Zhang,Anum Khursheed,Islam Bogachan Tahirbegi,Patrick N. Pallier,John H. Viles	3
2	Copper(II) Can Kinetically Trap Arctic and Italian Amyloid-β₄₀ as Toxic Oligomers, Mimicking Cu(II) Binding to Wild-Type Amyloid-β₄₂: Implications for Familial Alzheimer’s Disease 2024 ·SHILAP Revista de lepidopterología ·Yao Tian,Qi Shang,(unknown),John H. Viles	7
3	Impact of Membrane Phospholipids and Exosomes on the Kinetics of Amyloid-β Fibril Assembly 2024 ·Journal of Molecular Biology ·Anum Khursheed,John H. Viles	7
4	Cross-seeding of WT amyloid-β with Arctic but not Italian familial mutants accelerates fibril formation in Alzheimer's disease 2022 ·Journal of Biological Chemistry ·(unknown),Yao Tian,John H. Viles	12
5	Prion protein stabilizes amyloid-β (Aβ) oligomers and enhances Aβ neurotoxicity in a Drosophila model of Alzheimer's disease 2018 ·Journal of Biological Chemistry ·Nadine D. Younan,Ko‐Fan Chen,Ruth‐Sarah Rose,Damian C. Crowther,John H. Viles	29
6	Methionine oxidation reduces lag-times for amyloid-β(1–40) fiber formation but generates highly fragmented fibers 2016 ·Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics ·(unknown),John H. Viles	27
7	Endocytosis of the tachykinin neuropeptide, neurokinin B, in astrocytes and its role in cellular copper uptake 2016 ·Journal of Inorganic Biochemistry ·(unknown),Mark R. Jones,John H. Viles,Christopher E. Jones	16
8	Truncated Amyloid-β(11–40/42) from Alzheimer Disease Binds Cu2+ with a Femtomolar Affinity and Influences Fiber Assembly 2015 ·Journal of Biological Chemistry ·Joseph D. Barritt,John H. Viles	53
9	Bioactivity and structural properties of chimeric analogs of the starfish SALMFamide neuropeptides S1 and S2 2014 ·Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics ·Christopher E. Jones,(unknown),Nadine D. Younan,John H. Viles,Maurice R. Elphick	4
10	Structural analysis of the starfish SALMFamide neuropeptides S1 and S2: The N-terminal region of S2 facilitates self-association 2013 ·Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics ·(unknown),Christopher E. Jones,Nadine D. Younan,John H. Viles,Maurice R. Elphick	6
11	Methionine Oxidation Perturbs the Structural Core of the Prion Protein and Suggests a Generic Misfolding Pathway 2012 ·Journal of Biological Chemistry ·Nadine D. Younan,(unknown),Paul Davies,David R. Brown,John H. Viles	40
12	Substoichiometric Levels of Cu2+ Ions Accelerate the Kinetics of Fiber Formation and Promote Cell Toxicity of Amyloid-β from Alzheimer Disease 2010 ·Journal of Biological Chemistry ·Claire J. Sarell,Shane R. Wilkinson,John H. Viles	172
13	Manganese Binding to the Prion Protein 2008 ·Journal of Biological Chemistry ·Marcus W. Brazier,Paul Davies,(unknown),Frank Marken,John H. Viles,David R. Brown	81
14	Prion protein does not redox-silence Cu2+, but is a sacrificial quencher of hydroxyl radicals 2006 ·Free Radical Biology and Medicine ·(unknown),Salama R. Abdelraheim,Marcus W. Brazier,Stephen E. J. Rigby,David R. Brown,John H. Viles	64
15	Solution 1H NMR investigation of Zn2+ and Cd2+ binding to amyloid-beta peptide (Aβ) of Alzheimer's disease 2005 ·Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics ·Christopher D. Syme,John H. Viles	157
16	A survey of diamagnetic probes for copper²⁺binding to the prion protein.¹H NMR solution structure of the palladium²⁺bound single octarepeat 2005 ·Dalton Transactions ·(unknown),Christopher E. Jones,John H. Viles	24
17	Probing Copper2+ Binding to the Prion Protein Using Diamagnetic Nickel2+ and 1H NMR: The Unstructured N terminus Facilitates the Coordination of Six Copper2+ Ions at Physiological Concentrations 2005 ·Journal of Molecular Biology ·Christopher E. Jones,(unknown),Salama R. Abdelraheim,David R. Brown,John H. Viles	137
18	Copper Binding to the Amyloid-β (Aβ) Peptide Associated with Alzheimer's Disease 2004 ·Journal of Biological Chemistry ·Christopher D. Syme,(unknown),Stephen E. J. Rigby,John H. Viles	401
19	Observation of albumin resonances in proton nuclear magnetic resonance spectra of human blood plasma: N-terminal assignments aided by use of modified recombinant albumin 1996 ·The Analyst ·R. Harris,(unknown),Peter J. Sadler,John H. Viles	4
20	Towards meeting the paracelsus challenge: The design, synthesis, and characterization of paracelsin-43, an α-helical protein with over 50% sequence identity to an all-β protein 1996 ·Proteins Structure Function and Bioinformatics ·David T. Jones,Claire M. Moody,Julia Uppenbrink,John H. Viles,(unknown),C.J. Harris,Laurence H. Pearl,Peter J. Sadler,Janet M. Thornton	27

About John H. Viles

John H. Viles is a scholar working on Neurology, Nutrition and Dietetics and Biomaterials, having authored 66 papers that have together received 5.5k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (33 papers), Prion Diseases and Protein Misfolding (25 papers) and Trace Elements in Health (23 papers). The work is most often cited by research in Nutrition and Dietetics (2.2k citations), Neurology (1.1k citations) and Physiology (2.3k citations). John H. Viles has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include Christopher D. Syme, Stephen E. J. Rigby, David R. Brown, Nadine D. Younan, Peter E. Wright, H. Jane Dyson, Fred E. Cohen, Stanley B. Prusiner, Peter J. Sadler and Claire J. Sarell. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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