Marcus W. Brazier

847 total citations
18 papers, 698 citations indexed

About

Marcus W. Brazier is a scholar working on Molecular Biology, Nutrition and Dietetics and Neurology. According to data from OpenAlex, Marcus W. Brazier has authored 18 papers receiving a total of 698 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 13 papers in Nutrition and Dietetics and 7 papers in Neurology. Recurrent topics in Marcus W. Brazier's work include Prion Diseases and Protein Misfolding (15 papers), Trace Elements in Health (13 papers) and Neurological diseases and metabolism (7 papers). Marcus W. Brazier is often cited by papers focused on Prion Diseases and Protein Misfolding (15 papers), Trace Elements in Health (13 papers) and Neurological diseases and metabolism (7 papers). Marcus W. Brazier collaborates with scholars based in Australia, United Kingdom and Germany. Marcus W. Brazier's co-authors include Steven Collins, Colin L. Masters, David R. Brown, Andrew F. Hill, John H. Viles, Victoria Lewis, Ashley Fletcher, Salama R. Abdelraheim, Anthony R. White and Roberto Cappai and has published in prestigious journals such as Journal of Biological Chemistry, Annals of Neurology and Biochemical Journal.

In The Last Decade

Marcus W. Brazier

18 papers receiving 691 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Marcus W. Brazier Australia 15 558 307 242 184 49 18 698
Salama R. Abdelraheim United Kingdom 13 663 1.2× 418 1.4× 262 1.1× 131 0.7× 22 0.4× 20 821
Robert Strome Canada 7 996 1.8× 665 2.2× 530 2.2× 231 1.3× 76 1.6× 7 1.2k
Claire J. Sarell United Kingdom 9 325 0.6× 206 0.7× 59 0.2× 513 2.8× 54 1.1× 12 665
Baian Chen China 15 260 0.5× 52 0.2× 96 0.4× 166 0.9× 21 0.4× 27 493
Se Hui Sohn United States 5 188 0.3× 96 0.3× 99 0.4× 166 0.9× 376 7.7× 8 567
Timothy Johanssen Australia 7 235 0.4× 118 0.4× 33 0.1× 452 2.5× 21 0.4× 8 585
Lisa J. Whitson United States 10 275 0.5× 106 0.3× 119 0.5× 169 0.9× 483 9.9× 10 749
Daniela Kaden Germany 11 433 0.8× 127 0.4× 38 0.2× 571 3.1× 17 0.3× 12 742
Rachel L. Redler United States 10 291 0.5× 28 0.1× 67 0.3× 139 0.8× 343 7.0× 15 612
Anjli Venkateswaran United States 7 305 0.5× 20 0.1× 50 0.2× 162 0.9× 65 1.3× 7 609

Countries citing papers authored by Marcus W. Brazier

Since Specialization
Citations

This map shows the geographic impact of Marcus W. Brazier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marcus W. Brazier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marcus W. Brazier more than expected).

Fields of papers citing papers by Marcus W. Brazier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marcus W. Brazier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marcus W. Brazier. The network helps show where Marcus W. Brazier may publish in the future.

Co-authorship network of co-authors of Marcus W. Brazier

This figure shows the co-authorship network connecting the top 25 collaborators of Marcus W. Brazier. A scholar is included among the top collaborators of Marcus W. Brazier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marcus W. Brazier. Marcus W. Brazier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Brazier, Marcus W., Anthony G. Wedd, & Steven Collins. (2014). Antioxidant and Metal Chelation-Based Therapies in the Treatment of Prion Disease. Antioxidants. 3(2). 288–308. 12 indexed citations
2.
Brazier, Marcus W., Alexandra I. Mot, Anthony R. White, & Steven Collins. (2013). Immunotherapeutic Approaches in Prion Disease: progress, Challenges and Potential Directions. Therapeutic Delivery. 4(5). 615–628. 4 indexed citations
3.
Mot, Alexandra I., et al.. (2011). Metal attenuating therapies in neurodegenerative disease. Expert Review of Neurotherapeutics. 11(12). 1717–1745. 26 indexed citations
4.
Brazier, Marcus W., Irene Volitakis, Anthony R. White, et al.. (2010). Manganese chelation therapy extends survival in a mouse model of M1000 prion disease. Journal of Neurochemistry. 114(2). 440–451. 27 indexed citations
5.
Jones, Christopher E., Salama R. Abdelraheim, Marcus W. Brazier, et al.. (2009). Dynamics of a truncated prion protein, PrP(113–231), from 15N NMR relaxation: Order parameters calculated and slow conformational fluctuations localized to a distinct region. Protein Science. 18(2). 410–423. 29 indexed citations
6.
Brazier, Marcus W., et al.. (2009). Therapeutic interventions ameliorating prion disease. Expert Review of Anti-infective Therapy. 7(1). 83–105. 14 indexed citations
7.
Brazier, Marcus W., et al.. (2008). Manganese Binding to the Prion Protein. Journal of Biological Chemistry. 283(19). 12831–12839. 81 indexed citations
8.
Brazier, Marcus W., Susan R. Doctrow, Colin L. Masters, & Steven Collins. (2008). A manganese-superoxide dismutase/catalase mimetic extends survival in a mouse model of human prion disease. Free Radical Biology and Medicine. 45(2). 184–192. 48 indexed citations
9.
Abdelraheim, Salama R., et al.. (2006). Prion protein does not redox-silence Cu2+, but is a sacrificial quencher of hydroxyl radicals. Free Radical Biology and Medicine. 42(1). 79–89. 64 indexed citations
10.
Brazier, Marcus W., et al.. (2006). Mouse galectin‐1 inhibits the toxicity of glutamate by modifying NR1 NMDA receptor expression. European Journal of Neuroscience. 24(11). 3017–3025. 27 indexed citations
11.
Jones, Christopher E., Salama R. Abdelraheim, Andrew R. Thompsett, et al.. (2006). NMR characterization of the pH 4 β-intermediate of the prion protein: the N-terminal half of the protein remains unstructured and retains a high degree of flexibility. Biochemical Journal. 401(2). 533–540. 35 indexed citations
12.
Žerovnik, Eva, et al.. (2006). High affinity copper binding by stefin B (cystatin B) and its role in the inhibition of amyloid fibrillation. FEBS Journal. 273(18). 4250–4263. 25 indexed citations
13.
Collins, Steven, Victoria Lewis, Marcus W. Brazier, et al.. (2005). Extended period of asymptomatic prion disease after low dose inoculation: Assessment of detection methods and implications for infection control. Neurobiology of Disease. 20(2). 336–346. 18 indexed citations
14.
Brazier, Marcus W., Victoria Lewis, Giuseppe D. Ciccotosto, et al.. (2005). Correlative studies support lipid peroxidation is linked to PrPres propagation as an early primary pathogenic event in prion disease. Brain Research Bulletin. 68(5). 346–354. 69 indexed citations
15.
Yuan, Fang, Marcus W. Brazier, Roberto Cappai, et al.. (2005). Detection of prion epitopes on PrPc and PrPsc of transmissible spongiform encephalopathies using specific monoclonal antibodies to PrP. Immunology and Cell Biology. 83(6). 632–637. 16 indexed citations
16.
White, Anthony R., Fran Maher, Marcus W. Brazier, et al.. (2003). Diverse fibrillar peptides directly bind the Alzheimer’s amyloid precursor protein and amyloid precursor-like protein 2 resulting in cellular accumulation. Brain Research. 966(2). 231–244. 24 indexed citations
17.
Collins, Steven, Victoria Lewis, Marcus W. Brazier, et al.. (2002). Quinacrine does not prolong survival in a murine Creutzfeldt‐Jakob disease model. Annals of Neurology. 52(4). 503–506. 119 indexed citations
18.
White, Anthony R., Marcus W. Brazier, Michael F. Jobling, et al.. (2001). Sublethal Concentrations of Prion Peptide PrP106–126 or the Amyloid Beta Peptide of Alzheimer's Disease Activates Expression of Proapoptotic Markers in Primary Cortical Neurons. Neurobiology of Disease. 8(2). 299–316. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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