John Collinge

34.2k total citations · 7 hit papers
332 papers, 22.0k citations indexed

About

John Collinge is a scholar working on Molecular Biology, Neurology and Nutrition and Dietetics. According to data from OpenAlex, John Collinge has authored 332 papers receiving a total of 22.0k indexed citations (citations by other indexed papers that have themselves been cited), including 312 papers in Molecular Biology, 179 papers in Neurology and 115 papers in Nutrition and Dietetics. Recurrent topics in John Collinge's work include Prion Diseases and Protein Misfolding (305 papers), Neurological diseases and metabolism (178 papers) and Trace Elements in Health (114 papers). John Collinge is often cited by papers focused on Prion Diseases and Protein Misfolding (305 papers), Neurological diseases and metabolism (178 papers) and Trace Elements in Health (114 papers). John Collinge collaborates with scholars based in United Kingdom, United States and Australia. John Collinge's co-authors include Andrew F. Hill, Anthony R. Clarke, Jonathan D. F. Wadsworth, Graham S. Jackson, Sebastian Brandner, James W. Ironside, Mark S. Palmer, Simon Mead, Katie Sidle and Jacqueline M. Linehan and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

John Collinge

327 papers receiving 21.4k citations

Hit Papers

Molecular analysis of prion strain variation and the aeti... 1991 2026 2002 2014 1996 2001 2007 1991 2003 400 800 1.2k

Peers

John Collinge
Comparison fields: 5 of 168
  • Molecular Biology 19.8k
  • Neurology 10.6k
  • Nutrition and Dietetics 7.0k
  • Physiology 3.6k
  • Neurology 2.0k
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Citations per field, relative to John Collinge
John Collinge · 1×
Citations per year, relative to John Collinge
John Collinge · 1×

Countries citing papers authored by John Collinge

Since Specialization
Citations

This map shows the geographic impact of John Collinge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John Collinge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John Collinge more than expected).

Fields of papers citing papers by John Collinge

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John Collinge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John Collinge. The network helps show where John Collinge may publish in the future.

Co-authorship network of co-authors of John Collinge

This figure shows the co-authorship network connecting the top 25 collaborators of John Collinge. A scholar is included among the top collaborators of John Collinge based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John Collinge. John Collinge is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 30
2 7
3 90
4 60
5 33
6 2
7 4
8
A General Model of Prion Strains and Their Pathogenicity breakdown →
819
9
Molecular basis of prion diseases
1
10 197
11 276
12
Depleting Neuronal PrP in Prion Infection Prevents Disease and Reverses Spongiosis breakdown →
566
13
Early onset familial Alzheimer's disease - Mutation frequency in 31 families
77
14
Rescue of a physiological phenotype in mice with ablated PrP gene but expressing a transgene cassette for PrP
2
15 16
16
A new Austrian family with fatal familial insomnia: Brain pathology without detectable PrPres
1
17
Unaltered susceptibility to BSE in transgenic mice expressing human prion protein (vol 378, pg 779, 1995)
5
18
Tissue handling in suspected Creutzfeldt-Jakob disease (CJD) and other human spongiform encephalopathies (prion diseases)
1
19
APOLIPOPROTEIN-E GENOTYPE AND ALZHEIMERS-DISEASE
246
20
CONFIRMATION THAT THE APOLIPOPROTEIN E4 ALLELE IS ASSOCIATED WITH LATE-ONSET, FAMILIAL ALZHEIMERS-DISEASE
6

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026