Jens Kockskämper

2.5k total citations
62 papers, 1.8k citations indexed

About

Jens Kockskämper is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Jens Kockskämper has authored 62 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Cardiology and Cardiovascular Medicine, 45 papers in Molecular Biology and 12 papers in Cellular and Molecular Neuroscience. Recurrent topics in Jens Kockskämper's work include Cardiac electrophysiology and arrhythmias (43 papers), Ion channel regulation and function (34 papers) and Cardiomyopathy and Myosin Studies (8 papers). Jens Kockskämper is often cited by papers focused on Cardiac electrophysiology and arrhythmias (43 papers), Ion channel regulation and function (34 papers) and Cardiomyopathy and Myosin Studies (8 papers). Jens Kockskämper collaborates with scholars based in Germany, United States and Austria. Jens Kockskämper's co-authors include Lothar A. Blatter, Aleksey V. Zima, Burkert Pieske, Katherine A. Sheehan, Stephen L. Lipsius, Stefanie Walther, Jörg Hüser, Simon Sedej, Martin D. Bootman and Dirk von Lewinski and has published in prestigious journals such as Circulation, Journal of the American College of Cardiology and The Journal of Physiology.

In The Last Decade

Jens Kockskämper

59 papers receiving 1.8k citations

Peers

Jens Kockskämper

CCM

CMN

PHYSI

Han‐Gang Yu United States

Yasutaka Kurata Japan

Njanoor Narayanan Canada

Óscar Casis Spain

Rosana A. Bassani Brazil

A M Brown United States

Alexey E. Lyashkov United States

André De Léan Canada

Peter Karczewski Germany

R. W. Hadley United States

Han‐Gang Yu United States

Jens Kockskämper

1.3k ×1.0

CCM

1.3k ×1.1

639 ×2.1

CMN

102 ×0.6

PHYSI

75 ×0.8

Citations per year, relative to Jens Kockskämper Jens Kockskämper (= 1×) peers Han‐Gang Yu

Countries citing papers authored by Jens Kockskämper

Since Specialization

Citations

This map shows the geographic impact of Jens Kockskämper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jens Kockskämper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jens Kockskämper more than expected).

Fields of papers citing papers by Jens Kockskämper

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jens Kockskämper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jens Kockskämper. The network helps show where Jens Kockskämper may publish in the future.

Co-authorship network of co-authors of Jens Kockskämper

This figure shows the co-authorship network connecting the top 25 collaborators of Jens Kockskämper. A scholar is included among the top collaborators of Jens Kockskämper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jens Kockskämper. Jens Kockskämper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kisko, Theresa M., et al.. (2024). Altered Protein Kinase A-Dependent Phosphorylation of Cav1.2 in Left Ventricular Myocardium from Cacna1c Haploinsufficient Rat Hearts. International Journal of Molecular Sciences. 25(24). 13713–13713.

Kim, Walter, Aytuğ K. Kiper, Theresa M. Kisko, et al.. (2023). Calcium Handling Remodeling Underlies Impaired Sympathetic Stress Response in Ventricular Myocardium from Cacna1c Haploinsufficient Rats. International Journal of Molecular Sciences. 24(12). 9795–9795. 1 indexed citations

Hermes, Júlia, et al.. (2023). Store-Operated Calcium Entry Increases Nuclear Calcium in Adult Rat Atrial and Ventricular Cardiomyocytes. Cells. 12(23). 2690–2690.

Euler, Gerhild, Jens Kockskämper, Rainer Schulz, & Mariana S. Parahuleva. (2021). JDP2, a Novel Molecular Key in Heart Failure and Atrial Fibrillation?. International Journal of Molecular Sciences. 22(8). 4110–4110. 3 indexed citations

Niemann, Bernd, Ling Li, Andreas Simm, et al.. (2021). Caloric restriction reduces sympathetic activity similar to beta-blockers but conveys additional mitochondrio-protective effects in aged myocardium. Scientific Reports. 11(1). 1931–1931. 10 indexed citations

Parahuleva, Mariana S., Jens Kockskämper, Jacqueline Heger, et al.. (2020). Structural, Pro-Inflammatory and Calcium Handling Remodeling Underlies Spontaneous Onset of Paroxysmal Atrial Fibrillation in JDP2-Overexpressing Mice. International Journal of Molecular Sciences. 21(23). 9095–9095. 9 indexed citations

Thomas, Dierk, Torsten Christ, Larissa Fabritz, et al.. (2018). German Cardiac Society Working Group on Cellular Electrophysiology state-of-the-art paper: impact of molecular mechanisms on clinical arrhythmia management. Clinical Research in Cardiology. 108(6). 577–599. 26 indexed citations

Gess, Bernhard, et al.. (2016). The angiotensin receptor-associated protein Atrap is a stimulator of the cardiac Ca²⁺-ATPase SERCA2a. Cardiovascular Research. 110(3). 359–370. 12 indexed citations

Preißl, Sebastian, et al.. (2016). Enhanced nucleoplasmic Ca2+ signaling in ventricular myocytes from young hypertensive rats. Journal of Molecular and Cellular Cardiology. 101. 58–68. 16 indexed citations

10.

Pluteanu, Florentina, Alicja Bukowska, Ulrich Schotten, et al.. (2015). Early subcellular Ca2+ remodelling and increased propensity for Ca2+ alternans in left atrial myocytes from hypertensive rats. Cardiovascular Research. 106(1). 87–97. 46 indexed citations

11.

Ljubojević-Holzer, Senka, Snježana Radulović, Gerd Leitinger, et al.. (2014). Early Remodeling of Perinuclear Ca ²⁺ Stores and Nucleoplasmic Ca ²⁺ Signaling During the Development of Hypertrophy and Heart Failure. Circulation. 130(3). 244–255. 62 indexed citations

12.

Sedej, Simon, Albrecht Schmidt, Marco Denegri, et al.. (2013). Subclinical Abnormalities in Sarcoplasmic Reticulum Ca2+ Release Promote Eccentric Myocardial Remodeling and Pump Failure Death in Response to Pressure Overload. Journal of the American College of Cardiology. 63(15). 1569–1579. 39 indexed citations

13.

Dybkova, Nataliya, Simon Sedej, Carlo Napolitano, et al.. (2011). Overexpression of CaMKIIδc in RyR2R4496C+/− Knock-In Mice Leads to Altered Intracellular Ca2+ Handling and Increased Mortality. Journal of the American College of Cardiology. 57(4). 469–479. 28 indexed citations

14.

Walther, Stefanie, et al.. (2011). Urocortin II Causes Phosphorylation of eNOS and Stimulation of NO Production in Cardiac Myocytes. Biophysical Journal. 100(3). 257a–258a. 1 indexed citations

15.

Sedej, Simon, Frank R. Heinzel, Stefanie Walther, et al.. (2010). Na+-dependent SR Ca2+ overload induces arrhythmogenic events in mouse cardiomyocytes with a human CPVT mutation. Cardiovascular Research. 87(1). 50–59. 69 indexed citations

16.

Lewinski, Dirk von, Jens Kockskämper, Jan D. Schmitto, et al.. (2008). Direct Pro-Arrhythmogenic Effects of Angiotensin II can be Suppressed by AT1 Receptor Blockade in Human Atrial Myocardium. European Journal of Heart Failure. 10(12). 1172–1176. 35 indexed citations

17.

Lewinski, Dirk von, et al.. (2007). Mechanistic Insight Into the Functional and Toxic Effects of Strophanthidin in the Failing Human Myocardium. European Journal of Heart Failure. 9(11). 1086–1094. 11 indexed citations

18.

Lüers, Claus, et al.. (2005). Stretch-dependent modulation of [Na+]i, [Ca2+]i, and pHi in rabbit myocardium—a mechanism for the slow force response. Cardiovascular Research. 68(3). 454–463. 39 indexed citations

19.

Yang, Lei, Jens Kockskämper, Frank R. Heinzel, et al.. (2005). Urocortin II enhances contractility in rabbit ventricular myocytes via CRF2 receptor-mediated stimulation of protein kinase A. Cardiovascular Research. 69(2). 402–411. 35 indexed citations

20.

Kockskämper, Jens, Katherine A. Sheehan, Dan J. Bare, et al.. (2001). Activation and Propagation of Ca2+ Release during Excitation-Contraction Coupling in Atrial Myocytes. Biophysical Journal. 81(5). 2590–2605. 110 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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