Jasmina S. Redzic

2.4k citations
37 papers · 1.9k indexed · h-index 22
Topics
Extracellular vesicles in disease (6 papers)Signaling Pathways in Disease (6 papers)Erythrocyte Function and Pathophysiology (5 papers)

In The Last Decade

Jasmina S. Redzic

36 papers receiving 1.9k citations

Peers

Jasmina S. Redzic
Comparison fields: 5 of 100
  • Molecular Biology 1.3k
  • Cancer Research 549
  • Immunology 374
  • Immunology and Allergy 193
  • Dermatology 189
Replace Yoko Endo with:
Yoko Endo Japan
Anne M. Fourie United States
Huiyun Gao United States
Yabo Zhou China
Yuya Terashima Japan
Sakari Joenväärä Finland
Noriaki Inamura Japan
Justin Lim Australia
Adam Mor United States
Ernst Peter Rieber Germany
Jasmina S. Redzic relative to Yoko Endo Japan Yoko Endo's profile →
Citations per field
00.5×4.8×
Yoko Endo · 1×
Citations per year

Countries citing papers authored by Jasmina S. Redzic

Since Specialization
Citations

This map shows the geographic impact of Jasmina S. Redzic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jasmina S. Redzic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jasmina S. Redzic more than expected).

Fields of papers citing papers by Jasmina S. Redzic

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jasmina S. Redzic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jasmina S. Redzic. The network helps show where Jasmina S. Redzic may publish in the future.

Co-authorship network of co-authors of Jasmina S. Redzic

This figure shows the co-authorship network connecting the top 25 collaborators of Jasmina S. Redzic. A scholar is included among the top collaborators of Jasmina S. Redzic based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jasmina S. Redzic. Jasmina S. Redzic is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 2
2 4
3 1
4 6
5 2
6 2
7 30
8 58
9 22
10 19
11 26
12 80
13 168
14 67
15 42
16 36
17 30
18 213
19 184
20 45

About Jasmina S. Redzic

Jasmina S. Redzic is a scholar working on Microbiology, Immunology and Cell Biology, having authored 37 papers that have together received 1.9k indexed citations. Recurring topics across this work include Extracellular vesicles in disease (6 papers), Signaling Pathways in Disease (6 papers) and Erythrocyte Function and Pathophysiology (5 papers). The work is most often cited by research in Cancer Research (549 citations), Immunology and Allergy (193 citations) and Dermatology (189 citations). Jasmina S. Redzic has collaborated with scholars based in United States, Canada and Netherlands. Frequent co-authors include Michael W. Graner, Thomas J. Anchordoquy, Tyson Smyth, Kirk C. Hansen, Peter Smith‐Jones, Indushekhar Persaud, Nicole M. Payton, Elan Eisenmesser, J. Thorburn and Andrew Thorburn. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Communications and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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