James H. Millonig

2.3k total citations
40 papers, 1.7k citations indexed

About

James H. Millonig is a scholar working on Molecular Biology, Genetics and Cognitive Neuroscience. According to data from OpenAlex, James H. Millonig has authored 40 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 20 papers in Genetics and 14 papers in Cognitive Neuroscience. Recurrent topics in James H. Millonig's work include Genetics and Neurodevelopmental Disorders (16 papers), Autism Spectrum Disorder Research (14 papers) and Congenital heart defects research (13 papers). James H. Millonig is often cited by papers focused on Genetics and Neurodevelopmental Disorders (16 papers), Autism Spectrum Disorder Research (14 papers) and Congenital heart defects research (13 papers). James H. Millonig collaborates with scholars based in United States, Netherlands and United Kingdom. James H. Millonig's co-authors include Kathleen J. Millen, Mary E. Hatten, Linda M. Brzustowicz, Neda Gharani, Paul G. Matteson, Emanuel DiCicco‐Bloom, Henry A. Lester, Magdalena Hofer, Paulo Kofuji and Norman Davidson and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

James H. Millonig

40 papers receiving 1.7k citations

Peers

James H. Millonig
Sonia Cohen United States
Dawna D. Armstrong United States
Luis de la Torre-Ubieta United States
Ilse Gantois Belgium
Kerstin Ure United States
Nikolaos Mellios United States
Geeske M. van Woerden Netherlands
Shen‐Ju Chou Taiwan
Francesco Bedogni Italy
Sonia Cohen United States
James H. Millonig
Citations per year, relative to James H. Millonig James H. Millonig (= 1×) peers Sonia Cohen

Countries citing papers authored by James H. Millonig

Since Specialization
Citations

This map shows the geographic impact of James H. Millonig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James H. Millonig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James H. Millonig more than expected).

Fields of papers citing papers by James H. Millonig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James H. Millonig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James H. Millonig. The network helps show where James H. Millonig may publish in the future.

Co-authorship network of co-authors of James H. Millonig

This figure shows the co-authorship network connecting the top 25 collaborators of James H. Millonig. A scholar is included among the top collaborators of James H. Millonig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James H. Millonig. James H. Millonig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Salamon, Iva, Yongkyu Park, Paul G. Matteson, et al.. (2023). Celf4 controls mRNA translation underlying synaptic development in the prenatal mammalian neocortex. Nature Communications. 14(1). 6025–6025. 15 indexed citations
3.
Yeung, Percy Luk, Paul G. Matteson, Monal Mehta, et al.. (2022). Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. 17(6). 1380–1394. 13 indexed citations
4.
Mehta, Monal, et al.. (2020). Using iPSC-Based Models to Understand the Signaling and Cellular Phenotypes in Idiopathic Autism and 16p11.2 Derived Neurons. Advances in neurobiology. 25. 79–107. 3 indexed citations
5.
Millonig, James H., et al.. (2020). Dysregulation of Neurite Outgrowth and Cell Migration in Autism and Other Neurodevelopmental Disorders. Advances in neurobiology. 25. 109–153. 39 indexed citations
6.
Zhou, Xiaofeng, Paul G. Matteson, Percy Luk Yeung, et al.. (2018). Rapid Detection of Neurodevelopmental Phenotypes in Human Neural Precursor Cells (NPCs). Journal of Visualized Experiments. 11 indexed citations
7.
Wiseman, Jennifer A., Yu Meng, Paul G. Matteson, et al.. (2017). Chronic Enzyme Replacement to the Brain of a Late Infantile Neuronal Ceroid Lipofuscinosis Mouse Has Differential Effects on Phenotypes of Disease. Molecular Therapy — Methods & Clinical Development. 4. 204–212. 13 indexed citations
8.
Matteson, Paul G., et al.. (2017). Congenital Cataract in Gpr161vl/vl Mice Is Modified by Proximal Chromosome 15. PLoS ONE. 12(1). e0170724–e0170724. 3 indexed citations
9.
10.
Matteson, Paul G., Alejandro Q. Nato, Yong Lin, et al.. (2015). The orphan GPCR, Gpr161, regulates the retinoic acid and canonical Wnt pathways during neurulation. Developmental Biology. 402(1). 17–31. 22 indexed citations
11.
Choi, Jiyeon, et al.. (2014). Autism Associated Gene, ENGRAILED2, and Flanking Gene Levels Are Altered in Post-Mortem Cerebellum. PLoS ONE. 9(2). e87208–e87208. 34 indexed citations
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Anderson, Sharon, Bo Li, James H. Millonig, et al.. (2012). Medium chain acyl‐CoA dehydrogenase deficiency detected among Hispanics by New Jersey newborn screening. American Journal of Medical Genetics Part A. 158A(9). 2100–2105. 5 indexed citations
14.
Choi, Jiyeon, et al.. (2011). Cut-like homeobox 1 and nuclear factor I/B mediate ENGRAILED2 autism spectrum disorder-associated haplotype function. Human Molecular Genetics. 21(7). 1566–1580. 20 indexed citations
15.
Matteson, Paul G., Jigar Desai, Ron Korstanje, et al.. (2008). The orphan G protein-coupled receptor, Gpr161 , encodes the vacuolated lens locus and controls neurulation and lens development. Proceedings of the National Academy of Sciences. 105(6). 2088–2093. 57 indexed citations
16.
Cheh, Michelle A., et al.. (2006). En2 knockout mice display neurobehavioral and neurochemical alterations relevant to autism spectrum disorder. Brain Research. 1116(1). 166–176. 151 indexed citations
17.
Gharani, Neda, et al.. (2004). Association of the homeobox transcription factor, ENGRAILED 2, 3, with autism spectrum disorder. Molecular Psychiatry. 9(5). 474–484. 168 indexed citations
18.
Kofuji, Paulo, Magdalena Hofer, Kathleen J. Millen, et al.. (1996). Functional Analysis of the weaver Mutant GIRK2 K+ Channel and Rescue of weaver Granule Cells. Neuron. 16(5). 941–952. 170 indexed citations
19.
Millonig, James H., Kathleen J. Millen, & Mary E. Hatten. (1996). A high-density molecular genetic map around the weaver locus. Mammalian Genome. 7(8). 616–618. 3 indexed citations
20.
Millonig, James H., et al.. (1995). Molecular Analysis of the Distal Enhancer of the Mouse α-Fetoprotein Gene. Molecular and Cellular Biology. 15(7). 3848–3856. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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