Paul G. Matteson

1.3k total citations
26 papers, 1.0k citations indexed

About

Paul G. Matteson is a scholar working on Molecular Biology, Genetics and Cognitive Neuroscience. According to data from OpenAlex, Paul G. Matteson has authored 26 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 16 papers in Genetics and 8 papers in Cognitive Neuroscience. Recurrent topics in Paul G. Matteson's work include Genetics and Neurodevelopmental Disorders (10 papers), Congenital heart defects research (8 papers) and Autism Spectrum Disorder Research (8 papers). Paul G. Matteson is often cited by papers focused on Genetics and Neurodevelopmental Disorders (10 papers), Congenital heart defects research (8 papers) and Autism Spectrum Disorder Research (8 papers). Paul G. Matteson collaborates with scholars based in United States, Netherlands and Czechia. Paul G. Matteson's co-authors include James H. Millonig, Shirley M. Tilghman, Jennifer V. Schmidt, Beverly K. Jones, S M Tilghman, Paul B. Vrana, Tony del Rio, Emanuel DiCicco‐Bloom, John Fossella and Michael J. O’Neill and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Nature Genetics.

In The Last Decade

Paul G. Matteson

26 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Paul G. Matteson United States 15 676 638 204 184 85 26 1.0k
Susan Walker Canada 20 466 0.7× 572 0.9× 210 1.0× 153 0.8× 176 2.1× 48 1.1k
Valérie Matagne United States 19 702 1.0× 751 1.2× 143 0.7× 81 0.4× 148 1.7× 28 1.5k
Brent Bill United States 16 761 1.1× 362 0.6× 244 1.2× 77 0.4× 160 1.9× 20 1.3k
Yong-hui Jiang United States 10 823 1.2× 854 1.3× 406 2.0× 134 0.7× 174 2.0× 11 1.3k
Jyotsna Sudi United States 13 705 1.0× 1.0k 1.6× 451 2.2× 189 1.0× 148 1.7× 14 1.5k
Britt‐Marie Anderlid Sweden 22 607 0.9× 813 1.3× 200 1.0× 209 1.1× 51 0.6× 56 1.3k
Frank J. Probst United States 19 781 1.2× 446 0.7× 172 0.8× 62 0.3× 52 0.6× 27 1.4k
Zohreh Talebizadeh United States 17 973 1.4× 1.0k 1.6× 422 2.1× 123 0.7× 75 0.9× 31 1.7k
Gokul Ramaswami United States 15 1.4k 2.1× 1.2k 1.8× 426 2.1× 142 0.8× 109 1.3× 16 2.0k
Sylvia Dobrzeniecka Canada 16 575 0.9× 573 0.9× 127 0.6× 78 0.4× 151 1.8× 17 969

Countries citing papers authored by Paul G. Matteson

Since Specialization
Citations

This map shows the geographic impact of Paul G. Matteson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paul G. Matteson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paul G. Matteson more than expected).

Fields of papers citing papers by Paul G. Matteson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Paul G. Matteson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paul G. Matteson. The network helps show where Paul G. Matteson may publish in the future.

Co-authorship network of co-authors of Paul G. Matteson

This figure shows the co-authorship network connecting the top 25 collaborators of Paul G. Matteson. A scholar is included among the top collaborators of Paul G. Matteson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Paul G. Matteson. Paul G. Matteson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
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Matteson, Paul G., et al.. (2024). mTORC1 activity oscillates throughout the cell cycle, promoting mitotic entry and differentially influencing autophagy induction. Cell Reports. 43(8). 114543–114543. 7 indexed citations
3.
Salamon, Iva, Yongkyu Park, Paul G. Matteson, et al.. (2023). Celf4 controls mRNA translation underlying synaptic development in the prenatal mammalian neocortex. Nature Communications. 14(1). 6025–6025. 15 indexed citations
4.
Yeung, Percy Luk, Paul G. Matteson, Monal Mehta, et al.. (2022). Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. 17(6). 1380–1394. 13 indexed citations
5.
Mehta, Monal, et al.. (2020). Using iPSC-Based Models to Understand the Signaling and Cellular Phenotypes in Idiopathic Autism and 16p11.2 Derived Neurons. Advances in neurobiology. 25. 79–107. 3 indexed citations
6.
Zhou, Xiaofeng, Paul G. Matteson, Percy Luk Yeung, et al.. (2018). Rapid Detection of Neurodevelopmental Phenotypes in Human Neural Precursor Cells (NPCs). Journal of Visualized Experiments. 11 indexed citations
7.
Wiseman, Jennifer A., Yu Meng, Paul G. Matteson, et al.. (2017). Chronic Enzyme Replacement to the Brain of a Late Infantile Neuronal Ceroid Lipofuscinosis Mouse Has Differential Effects on Phenotypes of Disease. Molecular Therapy — Methods & Clinical Development. 4. 204–212. 13 indexed citations
8.
Matteson, Paul G., et al.. (2017). Congenital Cataract in Gpr161vl/vl Mice Is Modified by Proximal Chromosome 15. PLoS ONE. 12(1). e0170724–e0170724. 3 indexed citations
9.
10.
Matteson, Paul G., Alejandro Q. Nato, Yong Lin, et al.. (2015). The orphan GPCR, Gpr161, regulates the retinoic acid and canonical Wnt pathways during neurulation. Developmental Biology. 402(1). 17–31. 22 indexed citations
11.
Genestine, Matthieu, Lulu Lin, Yan Yan, et al.. (2015). Engrailed-2(En2) deletion produces multiple neurodevelopmental defects in monoamine systems, forebrain structures and neurogenesis and behavior. Human Molecular Genetics. 24(20). 5805–5827. 41 indexed citations
12.
Choi, Jiyeon, et al.. (2014). Autism Associated Gene, ENGRAILED2, and Flanking Gene Levels Are Altered in Post-Mortem Cerebellum. PLoS ONE. 9(2). e87208–e87208. 34 indexed citations
13.
Brielmaier, Jennifer, Paul G. Matteson, Jill L. Silverman, et al.. (2012). Autism-Relevant Social Abnormalities and Cognitive Deficits in Engrailed-2 Knockout Mice. PLoS ONE. 7(7). e40914–e40914. 131 indexed citations
14.
Choi, Jiyeon, et al.. (2011). Cut-like homeobox 1 and nuclear factor I/B mediate ENGRAILED2 autism spectrum disorder-associated haplotype function. Human Molecular Genetics. 21(7). 1566–1580. 20 indexed citations
15.
Choi, Jiyeon, et al.. (2009). Autism-Associated Haplotype Affects the Regulation of the Homeobox Gene, ENGRAILED 2. Biological Psychiatry. 66(10). 911–917. 56 indexed citations
16.
Huang, Yungui, Paul G. Matteson, Marco A. Azaro, et al.. (2009). Identification of a Schizophrenia-Associated Functional Noncoding Variant in NOS1AP. American Journal of Psychiatry. 166(4). 434–441. 57 indexed citations
17.
Matteson, Paul G., Jigar Desai, Ron Korstanje, et al.. (2008). The orphan G protein-coupled receptor, Gpr161 , encodes the vacuolated lens locus and controls neurulation and lens development. Proceedings of the National Academy of Sciences. 105(6). 2088–2093. 57 indexed citations
18.
Vrana, Paul B., John Fossella, Paul G. Matteson, et al.. (2000). Genetic and epigenetic incompatibilities underlie hybrid dysgenesis in Peromyscus. Nature Genetics. 25(1). 120–124. 146 indexed citations
19.
20.
Pavan, William J., et al.. (1995). A high-resolution linkage map of the lethal spotting locus: a mouse model for Hirschsprung disease. Mammalian Genome. 6(1). 1–7. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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