Edward S. Miller
- Molecular Biology top 5%
- Oncology top 5%
- Cell Biology top 10%
- Cancer Research
- Genetics
- Co-authors
- Grant S. StewartKelly TownsendTatjana StankovićPhilip J. ByrdA. Malcolm R. TaylorJarkko YlankoMegan MendezNadine Taubenheim
- Topics
- DNA Repair Mechanisms (7 papers)Microtubule and mitosis dynamics (3 papers)Microbial Metabolic Engineering and Bioproduction (3 papers)
- Partner nations
- United KingdomUnited StatesNetherlands
In The Last Decade
Edward S. Miller
18 papers receiving 1.4k citations
Hit Papers
Peers
Comparison fields: 5 of 83
- Molecular Biology 1.3k
- Oncology 488
- Cell Biology 155
- Cancer Research 144
- Genetics 128
Countries citing papers authored by Edward S. Miller
This map shows the geographic impact of Edward S. Miller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward S. Miller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward S. Miller more than expected).
Fields of papers citing papers by Edward S. Miller
This network shows the impact of papers produced by Edward S. Miller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward S. Miller. The network helps show where Edward S. Miller may publish in the future.
Co-authorship network of co-authors of Edward S. Miller
This figure shows the co-authorship network connecting the top 25 collaborators of Edward S. Miller. A scholar is included among the top collaborators of Edward S. Miller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edward S. Miller. Edward S. Miller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 4 | |
| 3 | 51 | |
| 4 | 148 | |
| 5 | 60 | |
| 6 | 42 | |
| 7 | 12 | |
| 8 | The RIDDLE Syndrome Protein Mediates a Ubiquitin-Dependent Signaling Cascade at Sites of DNA Damagebreakdown → | 599 |
| 9 | 39 | |
| 10 | 186 | |
| 11 | 61 | |
| 12 | 135 | |
| 13 | 20 | |
| 14 | 14 | |
| 15 | 8 | |
| 16 | Sound Military Decision | 1 |
| 17 | 2 | |
| 18 | 5 | |
| 19 | 1 |
About Edward S. Miller
Edward S. Miller is a scholar working on Biochemistry, Computer Science Applications and Cell Biology, having authored 19 papers that have together received 1.4k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (7 papers), Microtubule and mitosis dynamics (3 papers) and Microbial Metabolic Engineering and Bioproduction (3 papers). The work is most often cited by research in Molecular Biology (1.3k citations), Oncology (488 citations) and Cancer Research (144 citations). Edward S. Miller has collaborated with scholars based in United Kingdom, United States and Netherlands. Frequent co-authors include Grant S. Stewart, Kelly Townsend, Tatjana Stanković, Philip J. Byrd, A. Malcolm R. Taylor, Jarkko Ylanko, Megan Mendez, Nadine Taubenheim, Abdallah Al-Hakim and Laurence Pelletier. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.