Inge E. van Es

508 total citations
12 papers, 412 citations indexed

About

Inge E. van Es is a scholar working on Molecular Biology, Infectious Diseases and Neurology. According to data from OpenAlex, Inge E. van Es has authored 12 papers receiving a total of 412 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Infectious Diseases and 3 papers in Neurology. Recurrent topics in Inge E. van Es's work include Myasthenia Gravis and Thymoma (3 papers), Peripheral Neuropathies and Disorders (3 papers) and Antifungal resistance and susceptibility (3 papers). Inge E. van Es is often cited by papers focused on Myasthenia Gravis and Thymoma (3 papers), Peripheral Neuropathies and Disorders (3 papers) and Antifungal resistance and susceptibility (3 papers). Inge E. van Es collaborates with scholars based in Netherlands, United Kingdom and United States. Inge E. van Es's co-authors include Kris A. Reedquist, Paul P. Tak, Linda M. Hartkamp, Jan J.G.M. Verschuuren, Maartje G. Huijbers, Samuel García, Silvère M. van der Maarel, Beatriz Fernández, Jaap J. Plomp and Dominique Baeten and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Annals of the Rheumatic Diseases.

In The Last Decade

Inge E. van Es

10 papers receiving 410 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Inge E. van Es Netherlands 8 125 119 94 71 63 12 412
Anna R. Tröscher Austria 10 221 1.8× 160 1.3× 148 1.6× 40 0.6× 69 1.1× 14 667
Mark Marriott Australia 13 207 1.7× 130 1.1× 133 1.4× 66 0.9× 127 2.0× 28 758
Damiano Marastoni Italy 12 95 0.8× 67 0.6× 124 1.3× 59 0.8× 49 0.8× 35 433
Tobias Sejbæk Denmark 12 84 0.7× 98 0.8× 143 1.5× 56 0.8× 77 1.2× 40 500
Lucienne Costa‐Frossard Spain 17 178 1.4× 153 1.3× 231 2.5× 120 1.7× 105 1.7× 70 812
Adam Santaniello United States 14 220 1.8× 198 1.7× 116 1.2× 116 1.6× 81 1.3× 22 738
Jamie van Langelaar Netherlands 9 325 2.6× 90 0.8× 72 0.8× 48 0.7× 105 1.7× 14 571
Vanessa Daccach Marques Brazil 14 58 0.5× 96 0.8× 263 2.8× 87 1.2× 44 0.7× 32 522
Manuela Paunovic Netherlands 7 193 1.5× 81 0.7× 161 1.7× 39 0.5× 52 0.8× 10 486
Mikkel Carstensen Denmark 8 148 1.2× 135 1.1× 64 0.7× 45 0.6× 46 0.7× 12 366

Countries citing papers authored by Inge E. van Es

Since Specialization
Citations

This map shows the geographic impact of Inge E. van Es's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Inge E. van Es with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Inge E. van Es more than expected).

Fields of papers citing papers by Inge E. van Es

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Inge E. van Es. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Inge E. van Es. The network helps show where Inge E. van Es may publish in the future.

Co-authorship network of co-authors of Inge E. van Es

This figure shows the co-authorship network connecting the top 25 collaborators of Inge E. van Es. A scholar is included among the top collaborators of Inge E. van Es based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Inge E. van Es. Inge E. van Es is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Huijbers, Maartje G., Dana L.E. Vergoossen, Inge E. van Es, et al.. (2019). MuSK myasthenia gravis monoclonal antibodies. Neurology Neuroimmunology & Neuroinflammation. 6(3). e547–e547. 68 indexed citations
2.
Huijbers, Maartje G., Jaap J. Plomp, Inge E. van Es, et al.. (2019). Efgartigimod improves muscle weakness in a mouse model for muscle-specific kinase myasthenia gravis. Experimental Neurology. 317. 133–143. 31 indexed citations
3.
Huijbers, Maartje G., Inge E. van Es, Monique M. van Ostaijen-ten Dam, et al.. (2017). A prospective, placebo controlled study on the humoral immune response to and safety of tetanus revaccination in myasthenia gravis. Vaccine. 35(46). 6290–6296. 13 indexed citations
4.
García, Samuel, Linda M. Hartkamp, Inge E. van Es, et al.. (2016). Colony-stimulating factor (CSF) 1 receptor blockade reduces inflammation in human and murine models of rheumatoid arthritis. Arthritis Research & Therapy. 18(1). 75–75. 91 indexed citations
5.
García, Samuel, Sarah Krausz, Carmen A. Ambarus, et al.. (2014). Tie2 Signaling Cooperates with TNF to Promote the Pro-Inflammatory Activation of Human Macrophages Independently of Macrophage Functional Phenotype. PLoS ONE. 9(1). e82088–e82088. 41 indexed citations
6.
Hartkamp, Linda M., Jay S. Fine, Inge E. van Es, et al.. (2014). Btk inhibition suppresses agonist-induced human macrophage activation and inflammatory gene expression in RA synovial tissue explants. Annals of the Rheumatic Diseases. 74(8). 1603–1611. 38 indexed citations
7.
Angiolilli, Chiara, Aleksander M. Grabiec, Bradley S. Ferguson, et al.. (2014). Inflammatory cytokines epigenetically regulate rheumatoid arthritis fibroblast-like synoviocyte activation by suppressing HDAC5 expression. Annals of the Rheumatic Diseases. 75(2). 430–438. 68 indexed citations
8.
9.
10.
Es, Inge E. van, Jay S. Fine, Mark A. Smith, et al.. (2013). OP0304 BTK Inhibition Suppresses Inflammatory Cytokine Production and Affects Gene Expression in Human Macrophages and RA Synovial Tissue Explants. Annals of the Rheumatic Diseases. 72. A157–A157.
11.
Wiskerke, Joost, Dustin Schetters, Inge E. van Es, et al.. (2011). μ-Opioid Receptors in the Nucleus Accumbens Shell Region Mediate the Effects of Amphetamine on Inhibitory Control But Not Impulsive Choice. Journal of Neuroscience. 31(1). 262–272. 58 indexed citations
12.
Grabiec, Aleksander M., et al.. (2011). Functional interactions between PI3-kinase and NF-κB signaling pathways promote joint destruction in rheumatoid arthritis. Annals of the Rheumatic Diseases. 70. A27–A28. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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