Kris A. Reedquist
About
Kris A. Reedquist is a scholar working on Molecular Biology, Oncology and Immunology.
According to data from OpenAlex, Kris A. Reedquist has authored 91 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 30 papers in Oncology and 30 papers in Immunology. Recurrent topics in Kris A. Reedquist's work include Cytokine Signaling Pathways and Interactions (19 papers), Monoclonal and Polyclonal Antibodies Research (16 papers) and Rheumatoid Arthritis Research and Therapies (15 papers). Kris A. Reedquist is often cited by papers focused on Cytokine Signaling Pathways and Interactions (19 papers), Monoclonal and Polyclonal Antibodies Research (16 papers) and Rheumatoid Arthritis Research and Therapies (15 papers). Kris A. Reedquist collaborates with scholars based in Netherlands, United States and United Kingdom. Kris A. Reedquist's co-authors include Paul P. Tak, Johannes L. Bos, Johan de Rooij, Aleksander M. Grabiec, Hamid Band, Brian Druker, Toru Fukazawa, Dominique Baeten, Steven E. Shoelson and Sarah Krausz and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and SHILAP Revista de lepidopterología.
In The Last Decade
Kris A. Reedquist
90 papers receiving 5.5k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Kris A. Reedquist Netherlands | 43 | 2.9k | 2.1k | 1.1k | 749 | 731 | 91 | 5.6k | ||
| Katherine A. Siminovitch Canada | 52 | 3.6k 1.3× | 4.2k 2.0× | 1.2k 1.1× | 733 1.0× | 1.2k 1.6× | 136 | 8.8k | ||
| Kenji Oritani Japan | 39 | 2.3k 0.8× | 2.4k 1.2× | 1.4k 1.3× | 281 0.4× | 746 1.0× | 177 | 6.3k | ||
| Paul S. Changelian United States | 27 | 1.7k 0.6× | 1.6k 0.8× | 1.5k 1.4× | 395 0.5× | 339 0.5× | 39 | 4.4k | ||
| Ann Ranger United States | 24 | 2.9k 1.0× | 2.9k 1.4× | 1.0k 0.9× | 425 0.6× | 230 0.3× | 46 | 6.0k | ||
| Rie Watanabe‐Fukunaga Japan | 19 | 4.2k 1.5× | 3.2k 1.6× | 976 0.9× | 448 0.6× | 229 0.3× | 21 | 6.9k | ||
| Paolo Vezzoni Italy | 40 | 3.6k 1.3× | 1.9k 0.9× | 1.6k 1.5× | 328 0.4× | 249 0.3× | 175 | 6.7k | ||
| Ronald Herbst United States | 39 | 2.4k 0.8× | 2.8k 1.3× | 2.0k 1.8× | 445 0.6× | 192 0.3× | 130 | 6.5k | ||
| Martin F. Wolfson United States | 13 | 2.0k 0.7× | 1.7k 0.8× | 1.6k 1.4× | 252 0.3× | 1.1k 1.5× | 19 | 5.1k | ||
| Ana C. Carrera Spain | 42 | 3.3k 1.2× | 1.6k 0.8× | 1.1k 1.0× | 205 0.3× | 451 0.6× | 96 | 5.4k | ||
| Carl J. Kozlosky United States | 18 | 3.1k 1.1× | 2.1k 1.0× | 2.0k 1.8× | 255 0.3× | 1.4k 1.8× | 20 | 6.8k |
Countries citing papers authored by Kris A. Reedquist
Since
Specialization
Citations
This map shows the geographic impact of Kris A. Reedquist's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kris A. Reedquist with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kris A. Reedquist more than expected).
Fields of papers citing papers by Kris A. Reedquist
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Kris A. Reedquist. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kris A. Reedquist. The network helps show where Kris A. Reedquist may publish in the future.
Co-authorship network of co-authors of Kris A. Reedquist
This figure shows the co-authorship network connecting the top 25 collaborators of Kris A. Reedquist. A scholar is included among the top collaborators of Kris A. Reedquist based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kris A. Reedquist. Kris A. Reedquist is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Angiolilli, Chiara, Pawel A. Kabala, Aleksander M. Grabiec, et al.. (2018). Control of cytokine mRNA degradation by the histone deacetylase inhibitor ITF2357 in rheumatoid arthritis fibroblast-like synoviocytes: beyond transcriptional regulation. Arthritis Research & Therapy. 20(1). 148–148.
2.
Kabala, Pawel A., Chiara Angiolilli, Nataliya Yeremenko, et al.. (2017). Endoplasmic reticulum stress cooperates with Toll-like receptor ligation in driving activation of rheumatoid arthritis fibroblast-like synoviocytes. Arthritis Research & Therapy. 19(1). 207–207.
3.
Angiolilli, Chiara, Pawel A. Kabala, Aleksander M. Grabiec, et al.. (2016). Histone deacetylase 3 regulates the inflammatory gene expression programme of rheumatoid arthritis fibroblast-like synoviocytes. Annals of the Rheumatic Diseases. 76(1). 277–285.
4.
Schilderink, Ronald, Matthew Bell, Inmaculada Rioja, et al.. (2016). BET bromodomain inhibition reduces maturation and enhances tolerogenic properties of human and mouse dendritic cells. Molecular Immunology. 79. 66–76.
5.
Klein, Kerstin, Pawel A. Kabala, Aleksander M. Grabiec, et al.. (2014). The bromodomain protein inhibitor I-BET151 suppresses expression of inflammatory genes and matrix degrading enzymes in rheumatoid arthritis synovial fibroblasts. Annals of the Rheumatic Diseases. 75(2). 422–429.
6.
Grabiec, Aleksander M., Chiara Angiolilli, Linda M. Hartkamp, et al.. (2014). JNK-dependent downregulation of FoxO1 is required to promote the survival of fibroblast-like synoviocytes in rheumatoid arthritis. Annals of the Rheumatic Diseases. 74(9). 1763–1771.
7.
García, Samuel, Sarah Krausz, Carmen A. Ambarus, et al.. (2014). Tie2 Signaling Cooperates with TNF to Promote the Pro-Inflammatory Activation of Human Macrophages Independently of Macrophage Functional Phenotype. PLoS ONE. 9(1). e82088–e82088.
8.
Hartkamp, Linda M., Jay S. Fine, Inge E. van Es, et al.. (2014). Btk inhibition suppresses agonist-induced human macrophage activation and inflammatory gene expression in RA synovial tissue explants. Annals of the Rheumatic Diseases. 74(8). 1603–1611.
9.
Salinas, Gabriela, Sarah Krausz, Wendy Dontje, et al.. (2012). Sustained Rap1 activation in autoantigen-specific T lymphocytes attenuates experimental autoimmune encephalomyelitis. Journal of Neuroimmunology. 250(1-2). 35–43.
10.
Grabiec, Aleksander M., Sarah Krausz, Wilco de Jager, et al.. (2010). Histone Deacetylase Inhibitors Suppress Inflammatory Activation of Rheumatoid Arthritis Patient Synovial Macrophages and Tissue. The Journal of Immunology. 184(5). 2718–2728.
11.
Abreu, Joana R. F., Sarah Krausz, Wendy Dontje, et al.. (2010). Sustained T cell Rap1 signaling is protective in the collagen‐induced arthritis model of rheumatoid arthritis. Arthritis & Rheumatism. 62(11). 3289–3299.
12.
Hoek, Robert M., Feng Lin, Kris A. Reedquist, et al.. (2010). Deletion of either CD55 or CD97 ameliorates arthritis in mouse models. Arthritis & Rheumatism. 62(4). 1036–1042.
13.
Abreu, Joana R. F., Aleksander M. Grabiec, Sarah Krausz, et al.. (2009). The Presumed Hyporesponsive Behavior of Rheumatoid Arthritis T Lymphocytes Can Be Attributed to Spontaneous Ex Vivo Apoptosis rather than Defects in T Cell Receptor Signaling. The Journal of Immunology. 183(1). 621–630.
14.
Ulfman, Laurien H., Vera Kamp, Liesbeth P. Verhagen, et al.. (2008). Homeostatic Intracellular-Free Ca2+ Is Permissive for Rap1-Mediated Constitutive Activation of α4 Integrins on Eosinophils. The Journal of Immunology. 180(8). 5512–5519.
15.
Oosterhout, M. van, Tom Smeets, M. Sanders, et al.. (2005). Intracellular free radical production in synovial T lymphocytes from patients with rheumatoid arthritis. Arthritis & Rheumatism. 52(7). 2003–2009.
16.
Gringhuis, Sonja I., Jacob M. van Laar, M. Sanders, et al.. (2004). Rap1 Signaling Is Required for Suppression of Ras-Generated Reactive Oxygen Species and Protection Against Oxidative Stress in T Lymphocytes. The Journal of Immunology. 173(2). 920–931.
17.
Bos, Johannes L., Johan de Rooij, & Kris A. Reedquist. (2001). Rap1 signalling: adhering to new models. Nature Reviews Molecular Cell Biology. 2(5). 369–377.
18.
Rao, Navin, Mark Lupher, Satoshi Ota, et al.. (2000). The Linker Phosphorylation Site Tyr292 Mediates the Negative Regulatory Effect of Cbl on ZAP-70 in T Cells. The Journal of Immunology. 164(9). 4616–4626.
19.
Lupher, Mark, Kris A. Reedquist, Sachiko Miyake, Wallace Y. Langdon, & Hamid Band. (1996). A Novel Phosphotyrosine-binding Domain in the N-terminal Transforming Region of Cbl Interacts Directly and Selectively with ZAP-70 in T Cells. Journal of Biological Chemistry. 271(39). 24063–24068.
20.
Fukazawa, Toru, Lesley A. Stolz, Gillian Payne, et al.. (1994). Physical and Functional Interactions between SH2 and SH3 Domains of the Src Family Protein Tyrosine Kinase p59 fyn . Molecular and Cellular Biology. 14(9). 6372–6385.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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