I. Wyatt

1.8k total citations
54 papers, 1.5k citations indexed

About

I. Wyatt is a scholar working on Molecular Biology, Surgery and Cellular and Molecular Neuroscience. According to data from OpenAlex, I. Wyatt has authored 54 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 18 papers in Surgery and 9 papers in Cellular and Molecular Neuroscience. Recurrent topics in I. Wyatt's work include Paraquat toxicity studies and treatments (17 papers), Neuroscience and Neuropharmacology Research (9 papers) and Nitric Oxide and Endothelin Effects (7 papers). I. Wyatt is often cited by papers focused on Paraquat toxicity studies and treatments (17 papers), Neuroscience and Neuropharmacology Research (9 papers) and Nitric Oxide and Endothelin Effects (7 papers). I. Wyatt collaborates with scholars based in United Kingdom and United States. I. Wyatt's co-authors include Lewis L. Smith, Michael S. Rose, Edward A. Lock, K. A. Fletcher, Gerald M. Cohen, Clifford R. Elcombe, Andrew Martin Wright, Peter Widdowson, Anthony R. Soames and Michael Clay and has published in prestigious journals such as Nature, Biochemical Journal and Brain Research.

In The Last Decade

I. Wyatt

54 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
I. Wyatt United Kingdom 20 674 670 216 166 150 54 1.5k
Ingrid Wiswedel Germany 25 658 1.0× 186 0.3× 107 0.5× 60 0.4× 53 0.4× 53 1.8k
R L Chase United States 8 374 0.6× 122 0.2× 92 0.4× 82 0.5× 67 0.4× 8 1.4k
Véronique Maupoil France 24 427 0.6× 108 0.2× 108 0.5× 104 0.6× 47 0.3× 65 1.6k
Jeanine Fontaine Belgium 25 752 1.1× 208 0.3× 208 1.0× 37 0.2× 84 0.6× 121 1.8k
Lucia Cavallini Italy 23 743 1.1× 105 0.2× 73 0.3× 106 0.6× 99 0.7× 71 1.5k
Miklós Csala Hungary 28 1.1k 1.6× 238 0.4× 105 0.5× 78 0.5× 144 1.0× 93 2.5k
Teresa Tejerina Spain 25 587 0.9× 257 0.4× 96 0.4× 61 0.4× 48 0.3× 85 1.6k
Werner G. Siems Germany 25 842 1.2× 119 0.2× 77 0.4× 72 0.4× 42 0.3× 54 2.2k
Aida Abate Germany 21 575 0.9× 145 0.2× 126 0.6× 222 1.3× 56 0.4× 23 1.3k
Damiano Cottalasso Italy 25 730 1.1× 118 0.2× 65 0.3× 38 0.2× 137 0.9× 82 1.8k

Countries citing papers authored by I. Wyatt

Since Specialization
Citations

This map shows the geographic impact of I. Wyatt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I. Wyatt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I. Wyatt more than expected).

Fields of papers citing papers by I. Wyatt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by I. Wyatt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I. Wyatt. The network helps show where I. Wyatt may publish in the future.

Co-authorship network of co-authors of I. Wyatt

This figure shows the co-authorship network connecting the top 25 collaborators of I. Wyatt. A scholar is included among the top collaborators of I. Wyatt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with I. Wyatt. I. Wyatt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lock, Edward A., et al.. (2000). The absence of cerebellar granule cell necrosis in the mouse following L- 2-chloropropionic acid administration. Archives of Toxicology. 74(9). 547–554. 1 indexed citations
2.
Widdowson, P.S., et al.. (1997). l -2-Chloropropionic acid-induced cerebellar granule cell necrosis is potentiated by L-type calcium channel antagonists. Archives of Toxicology. 71(12). 751–755. 2 indexed citations
3.
Widdowson, Peter, et al.. (1997). L-2-chloropropionic acid-induced neurotoxicity in the rat: A valuable model for studying selective neuronal cell death In Vivo. General Pharmacology The Vascular System. 29(2). 113–119. 3 indexed citations
4.
Lock, Edward A., et al.. (1997). Neuroprotection afforded by MK-801 against l-2-chloropropionic acid-induced cerebellar granule cell necrosis in the rat. Toxicology. 123(1-2). 41–51. 13 indexed citations
5.
Widdowson, P.S., et al.. (1996). L-2-Chloropropionic Acid-Induced Neurotoxicity Is Prevented by MK-801: Possible Role of NMDA Receptors in the Neuropathology. Toxicology and Applied Pharmacology. 136(1). 138–145. 12 indexed citations
6.
Widdowson, Peter, et al.. (1996). Evidence for mediation of L‐2‐chloropropionic acid‐induced delayed neuronal cell death by activation of a constitutive nitric oxide synthase. British Journal of Pharmacology. 119(2). 374–378. 3 indexed citations
7.
Widdowson, Peter, et al.. (1996). Possible role of nitric oxide in the development of L‐2‐chloropropionic acid‐induced cerebellar granule cell necrosis. British Journal of Pharmacology. 117(8). 1761–1767. 9 indexed citations
8.
Wyatt, I., et al.. (1996). Glutathione depletion in the liver and brain produced by 2-chloropropionic acid: relevance to cerebellar granule cell necrosis. Archives of Toxicology. 70(6). 380–389. 18 indexed citations
9.
Widdowson, Peter, Michael G. Simpson, I. Wyatt, & Edward A. Lock. (1995). [125I]Endothelin binding in rat cerebellum is increased following l-2-chloropropionic-acid-induced granule cell necrosis. Peptides. 16(5). 897–902. 6 indexed citations
10.
Widdowson, Peter, et al.. (1995). Chloropropionic acid-induced alterations in glucose metabolic status: possible relevance to cerebellar granule cell necrosis. Archives of Toxicology. 69(9). 640–643. 5 indexed citations
11.
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13.
Wyatt, I., et al.. (1993). The effect of chlorinated paraffins on hepatic enzymes and thyroid hormones. Toxicology. 77(1-2). 81–90. 71 indexed citations
14.
O’Sullivan, Mary C., Bernard T. Golding, Lewis L. Smith, & I. Wyatt. (1991). Molecular features necessary for the uptake of diamines and related compounds by the polyamine receptor of rat lung slices. Biochemical Pharmacology. 41(12). 1839–1848. 26 indexed citations
15.
Styles, J.A., et al.. (1991). Trichloroacetic acid: studies on uptake and effects on hepatic DNA and liver growth in mouse. Carcinogenesis. 12(9). 1715–1719. 25 indexed citations
16.
Smith, Lewis L., et al.. (1990). The importance of epithelial uptake systems in lung toxicity.. Environmental Health Perspectives. 85. 25–30. 24 indexed citations
17.
Wyatt, I., Richard B. Moore, & Lewis L. Smith. (1989). Competition for Polyamine Uptake into Rat Lung Slices by WR2721 and Analogues. International Journal of Radiation Biology. 55(3). 463–472. 19 indexed citations
18.
Wyatt, I., Anthony R. Soames, Michael Clay, & Lewis L. Smith. (1988). The accumulation and localisation of putrescine, spermidine, spermine and paraquat in the rat lung. Biochemical Pharmacology. 37(10). 1909–1918. 51 indexed citations
19.
Rose, Michael S., Lewis L. Smith, & I. Wyatt. (1975). The accumulation of paraquat by the lung and the relevance of this to the treatment of paraquat poisoning. Toxicology and Applied Pharmacology. 33(1). 136. 3 indexed citations
20.
Rose, Michael S., Lewis L. Smith, & I. Wyatt. (1974). Evidence for energy-dependent accumulation of paraquat into rat lung. Nature. 252(5481). 314–315. 249 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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