I. Silos-Santiago
- Cellular and Molecular Neuroscience top 5%
- Molecular Biology
- Sensory Systems top 5%
- Physiology
- Developmental Neuroscience top 5%
- Co-authors
- Mariano BarbacidBernd FritzschPatrik ErnforsGeorge D. YancopoulosFrank L. RiceHeidi PhillipsBengt T. FundínHåkan Aldskogius
- Topics
- Nerve injury and regeneration (8 papers)Pain Mechanisms and Treatments (3 papers)Axon Guidance and Neuronal Signaling (3 papers)
- Journals
- Annals of the New York Academy of SciencesDevelopmental BiologyCurrent Opinion in Neurobiology
- Partner nations
- United StatesSpainItaly
In The Last Decade
I. Silos-Santiago
13 papers receiving 509 citations
Peers
Comparison fields: 5 of 67
- Cellular and Molecular Neuroscience 293
- Molecular Biology 174
- Sensory Systems 116
- Physiology 101
- Developmental Neuroscience 79
Countries citing papers authored by I. Silos-Santiago
This map shows the geographic impact of I. Silos-Santiago's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I. Silos-Santiago with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I. Silos-Santiago more than expected).
Fields of papers citing papers by I. Silos-Santiago
This network shows the impact of papers produced by I. Silos-Santiago. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I. Silos-Santiago. The network helps show where I. Silos-Santiago may publish in the future.
Co-authorship network of co-authors of I. Silos-Santiago
This figure shows the co-authorship network connecting the top 25 collaborators of I. Silos-Santiago. A scholar is included among the top collaborators of I. Silos-Santiago based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with I. Silos-Santiago. I. Silos-Santiago is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 32 | |
| 2 | 18 | |
| 3 | 39 | |
| 4 | 16 | |
| 5 | 12 | |
| 6 | 7 | |
| 7 | 49 | |
| 8 | 27 | |
| 9 | 50 | |
| 10 | 18 | |
| 11 | 51 | |
| 12 | 161 | |
| 13 | 39 |
About I. Silos-Santiago
I. Silos-Santiago is a scholar working on Cellular and Molecular Neuroscience, Developmental Neuroscience and Sensory Systems, having authored 13 papers that have together received 519 indexed citations. Recurring topics across this work include Nerve injury and regeneration (8 papers), Pain Mechanisms and Treatments (3 papers) and Axon Guidance and Neuronal Signaling (3 papers). The work is most often cited by research in Sensory Systems (116 citations), Developmental Neuroscience (79 citations) and Cellular and Molecular Neuroscience (293 citations). I. Silos-Santiago has collaborated with scholars based in United States, Spain and Italy. Frequent co-authors include Mariano Barbacid, Bernd Fritzsch, Patrik Ernfors, George D. Yancopoulos, Frank L. Rice, Heidi Phillips, Bengt T. Fundín, Håkan Aldskogius, Thomas M. DeChiara and José A. Vega. Their work appears in journals such as Annals of the New York Academy of Sciences, Developmental Biology and Current Opinion in Neurobiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.