Hugh J. Cornell

1.4k total citations
37 papers, 1.1k citations indexed

About

Hugh J. Cornell is a scholar working on Nutrition and Dietetics, Gastroenterology and Molecular Biology. According to data from OpenAlex, Hugh J. Cornell has authored 37 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Nutrition and Dietetics, 18 papers in Gastroenterology and 6 papers in Molecular Biology. Recurrent topics in Hugh J. Cornell's work include Celiac Disease Research and Management (18 papers), Microbial Metabolites in Food Biotechnology (12 papers) and Food composition and properties (10 papers). Hugh J. Cornell is often cited by papers focused on Celiac Disease Research and Management (18 papers), Microbial Metabolites in Food Biotechnology (12 papers) and Food composition and properties (10 papers). Hugh J. Cornell collaborates with scholars based in Australia, Germany and Italy. Hugh J. Cornell's co-authors include Colin Rix, Donald E. Rivett, Andrew P. Negri, Scott J. McGrane, Darryl M. Small, David E. Mainwaring, Thomas Mothes, Oliver A.H. Jones, Adrian C. Herington and T Stelmasiak and has published in prestigious journals such as SHILAP Revista de lepidopterología, Food Chemistry and Clinica Chimica Acta.

In The Last Decade

Hugh J. Cornell

37 papers receiving 1.1k citations

Peers

Hugh J. Cornell

GASTR

Olivier Tranquet France

Shujuan Jiang China

Maoyi Li China

Anshu Yang China

Masahiko Nonaka Japan

A Misiewicz Poland

Sinéad M. Ryan Ireland

He Huang China

Shuji Kawai Japan

Makarov Va Russia

Olivier Tranquet France

Hugh J. Cornell

192 ×0.4

217 ×0.7

126 ×0.5

GASTR

429 ×2.1

2 ×0.0

Citations per year, relative to Hugh J. Cornell Hugh J. Cornell (= 1×) peers Olivier Tranquet

Countries citing papers authored by Hugh J. Cornell

Since Specialization

Citations

This map shows the geographic impact of Hugh J. Cornell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hugh J. Cornell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hugh J. Cornell more than expected).

Fields of papers citing papers by Hugh J. Cornell

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hugh J. Cornell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hugh J. Cornell. The network helps show where Hugh J. Cornell may publish in the future.

Co-authorship network of co-authors of Hugh J. Cornell

This figure shows the co-authorship network connecting the top 25 collaborators of Hugh J. Cornell. A scholar is included among the top collaborators of Hugh J. Cornell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hugh J. Cornell. Hugh J. Cornell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Cornell, Hugh J., T Stelmasiak, & Aaron Lerner. (2021). Enzyme Therapy for Patients with Celiac Disease - An Update. 9(1). 28–34. 1 indexed citations

Cornell, Hugh J., et al.. (2021). Antibacterial Profile of a Microbicidal Agent Targeting Tyrosine Phosphatases and Redox Thiols, Novel Drug Targets. Antibiotics. 10(11). 1310–1310. 3 indexed citations

Cornell, Hugh J., et al.. (2016). The effect of enzyme supplementation on symptoms and duodenal histology in celiac patients. RMIT Research Repository (RMIT University Library). 4(2). 40–47. 7 indexed citations

Cornell, Hugh J. & T Stelmasiak. (2016). The Significance of Key Amino Acid Sequences in the Digestibility and Toxicity of Gliadin Peptides in Celiac Disease. RMIT Research Repository (RMIT University Library). 4(4). 113–120. 7 indexed citations

Cornell, Hugh J., et al.. (2015). Application of the rat liver lysosome assay to determining the reduction of toxic gliadin content during breadmaking. Food Chemistry. 192. 924–927. 1 indexed citations

Cornell, Hugh J., et al.. (2014). Reduction of toxic gliadin content of wholegrain bread by the enzyme caricain. Food Chemistry. 170. 343–347. 25 indexed citations

Cornell, Hugh J., et al.. (2014). Comparisons of Halogenated β-Nitrostyrenes as Antimicrobial Agents. Applied Sciences. 4(3). 380–389. 15 indexed citations

Cornell, Hugh J. & T Stelmasiak. (2011). Caricain : a basis for enzyme therapy for coeliac disease : research article. South African Journal of Science. 107. 1–5. 1 indexed citations

Silano, Marco, Rita Di Benedetto, Francesca Maialetti, et al.. (2007). Avenins from different cultivars of oats elicit response by coeliac peripheral lymphocytes. Scandinavian Journal of Gastroenterology. 42(11). 1302–1305. 44 indexed citations

10.

Silano, Marco, Mariarita Dessı̀, Massimo De Vincenzi, & Hugh J. Cornell. (2006). In vitro tests indicate that certain varieties of oats may be harmful to patients with coeliac disease. Journal of Gastroenterology and Hepatology. 22(4). 528–531. 26 indexed citations

11.

Cornell, Hugh J., et al.. (2005). Enzyme therapy for management of coeliac disease. Scandinavian Journal of Gastroenterology. 40(11). 1304–1312. 33 indexed citations

12.

Cornell, Hugh J.. (1998). Partial In Vitro Digestion of Active Gliadin-Related Peptides in Celiac Disease. Journal of Protein Chemistry. 17(8). 739–744. 9 indexed citations

13.

Cornell, Hugh J. & Thomas Mothes. (1995). Further studies of the in vitro activity of synthetic gliadin peptides in coeliac disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1270(2-3). 168–172. 14 indexed citations

14.

Cornell, Hugh J., et al.. (1994). Particle Size Distribution in Wheat Starch and Its Importance in Processing. Starch - Stärke. 46(6). 203–207. 21 indexed citations

15.

Cornell, Hugh J. & Thomas Mothes. (1993). The activity of wheat gliadin peptides in in vitro assays for coeliac disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1181(2). 169–173. 26 indexed citations

16.

Tesch, Greg H., Christopher J. Handley, Hugh J. Cornell, & Adrian C. Herington. (1992). Effects of free and bound insulin‐like growth factors on proteoglycan metabolism in articular cartilage explants. Journal of Orthopaedic Research®. 10(1). 14–22. 50 indexed citations

17.

Cornell, Hugh J., Herbert Wieser, & Hans‐Dieter Belitz. (1992). Characterization of the gliadin-derived peptides which are biologically active in coeliac disease. Clinica Chimica Acta. 213(1-3). 37–50. 22 indexed citations

18.

Negri, Andrew P., Hugh J. Cornell, & Donald E. Rivett. (1992). Effects of Processing on the Bound and Free Fatty Acid Levels in Wool. Textile Research Journal. 62(7). 381–387. 29 indexed citations

19.

Cornell, Hugh J., et al.. (1987). Application of preparative reversed-phase high-performance liquid chromatography to the isolation of insulin-like growth factor II from human serum. Journal of Chromatography B Biomedical Sciences and Applications. 421(1). 61–69. 2 indexed citations

20.

Cornell, Hugh J. & Adrian C. Herington. (1983). Evidence for a biologically active, carrier-bound form of non-suppressible insulin-like activity (nsila) in human serum. International Journal of Biochemistry. 15(4). 553–558. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact