Howard Feit

3.4k total citations
40 papers, 1.4k citations indexed

About

Howard Feit is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Howard Feit has authored 40 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 10 papers in Cell Biology and 7 papers in Cellular and Molecular Neuroscience. Recurrent topics in Howard Feit's work include Muscle Physiology and Disorders (12 papers), Cardiomyopathy and Myosin Studies (6 papers) and Microtubule and mitosis dynamics (5 papers). Howard Feit is often cited by papers focused on Muscle Physiology and Disorders (12 papers), Cardiomyopathy and Myosin Studies (6 papers) and Microtubule and mitosis dynamics (5 papers). Howard Feit collaborates with scholars based in United States, Canada and France. Howard Feit's co-authors include Michael L. Shelanski, S H Barondes, Ning Jiang, Donald G. Stein, Michael Chopp, Erwin R. Thal, Gary R. Dutton, Samuel H. Barondes, William I. Manton and Steven R. Levine and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Cell Biology and Annals of Internal Medicine.

In The Last Decade

Howard Feit

38 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Howard Feit United States	18	618	350	292	181	178	40	1.4k
Masataka Fujita Japan	18	765 1.2×	290 0.8×	183 0.6×	77 0.4×	267 1.5×	41	1.4k
Hiromichi Kawai Japan	19	1.0k 1.7×	303 0.9×	303 1.0×	212 1.2×	438 2.5×	40	1.9k
Kerstin Boström Sweden	14	451 0.7×	130 0.4×	144 0.5×	294 1.6×	233 1.3×	28	1.1k
J. Douglas Balentine United States	20	461 0.7×	148 0.4×	527 1.8×	276 1.5×	254 1.4×	44	1.7k
Irina V. Smirnova United States	27	538 0.9×	112 0.3×	229 0.8×	236 1.3×	263 1.5×	61	1.8k
Robert Surtees United Kingdom	23	848 1.4×	141 0.4×	384 1.3×	380 2.1×	246 1.4×	45	2.2k
T Forrester United States	14	491 0.8×	192 0.5×	157 0.5×	97 0.5×	534 3.0×	30	1.7k
D K Anderson United States	14	378 0.6×	122 0.3×	478 1.6×	311 1.7×	156 0.9×	21	1.7k
Gen Wen United States	19	584 0.9×	179 0.5×	253 0.9×	199 1.1×	165 0.9×	34	1.3k
D. A. S. G. Mary United Kingdom	27	804 1.3×	131 0.4×	402 1.4×	47 0.3×	335 1.9×	106	2.5k

Countries citing papers authored by Howard Feit

Since Specialization

Citations

This map shows the geographic impact of Howard Feit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Howard Feit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Howard Feit more than expected).

Fields of papers citing papers by Howard Feit

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Howard Feit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Howard Feit. The network helps show where Howard Feit may publish in the future.

Co-authorship network of co-authors of Howard Feit

This figure shows the co-authorship network connecting the top 25 collaborators of Howard Feit. A scholar is included among the top collaborators of Howard Feit based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Howard Feit. Howard Feit is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Koffman, Boyd M., Larry Junck, Stanton B. Elias, Howard Feit, & Steven R. Levine. (1999). Polyradiculopathy in sarcoidosis. Muscle & Nerve. 22(5). 608–613. 39 indexed citations

Koffman, Boyd M., Larry Junck, Stanton B. Elias, Howard Feit, & Steven R. Levine. (1999). Polyradiculopathy in sarcoidosis. Muscle & Nerve. 22(5). 608–613. 5 indexed citations

Feit, Howard, Alice K. Silbergleit, J. Antonio Gutierrez, et al.. (1998). Vocal Cord and Pharyngeal Weakness with Autosomal Dominant Distal Myopathy: Clinical Description and Gene Localization to 5q31. The American Journal of Human Genetics. 63(6). 1732–1742. 97 indexed citations

Jiang, Ning, Michael Chopp, Donald G. Stein, & Howard Feit. (1996). Progesterone is neuroprotective after transient middle cerebral artery occlusion in male rats. Brain Research. 735(1). 101–107. 198 indexed citations

Feit, Howard, Masataka Kawai, & Asma Mostafapour. (1989). Increased resistance of the collagen in avian dystrophic muscle to collagenolytic attack: Evidence for increased crosslinking. Muscle & Nerve. 12(6). 476–485. 13 indexed citations

Feit, Howard, Masataka Kawai, & Asma Mostafapour. (1989). The role of collagen crosslinking in the increased stiffness of avian dystrophic muscle. Muscle & Nerve. 12(6). 486–492. 21 indexed citations

Feit, Howard, et al.. (1985). Stiffness and contractile properties of avian normal and dystrophic muscle bundles as measured by sinusoidal length perturbations. Muscle & Nerve. 8(6). 503–510. 12 indexed citations

Feit, Howard. (1985). Acute subdural hematomas may appear isodense in anemia. Neurology. 35(1). 141–141. 1 indexed citations

Feit, Howard & Cathy A. Farrell. (1984). The interaction of unregulated actin and myosin in avian muscular dystrophy. Muscle & Nerve. 7(8). 668–675. 1 indexed citations

10.

Feit, Howard. (1983). Possible biochemical consequences of different subsets of contractile proteins in single dystrophic muscle fibers.. PubMed. 6(2). 171–2. 2 indexed citations

11.

Feit, Howard, John W. Fuseler, & Jay D. Cook. (1983). Myofibrillar creatine kinase in Duchenne and avian muscular dystrophy. Biochemical Medicine. 29(3). 355–359. 4 indexed citations

12.

Feit, Howard, et al.. (1982). Persistence of the expression of β‐tropomyosin in dystrophic avian pectoral muscle. Cell Motility. 2(3). 309–315. 9 indexed citations

13.

Shay, Jerry W. & Howard Feit. (1980). Avian muscular dystrophy: Microtubules, righting ability, CPK and muscle histology following isaxonine treatment. European Journal of Cell Biology. 22(1).

14.

Feit, Howard & Jerry W. Shay. (1980). The assembly of tubulin into membranes. Biochemical and Biophysical Research Communications. 94(1). 324–331. 19 indexed citations

15.

Feit, Howard. (1979). A form of chorea that responds to dantrolene sodium. Neurology. 29(12). 1631–1632. 4 indexed citations

16.

Feit, Howard, et al.. (1977). Anomalous electrophoretic properties of brain filament protein subunits. Brain Research. 133(2). 341–349. 7 indexed citations

17.

Feit, Howard, et al.. (1977). COMPARISON OF THE ISOELECTRIC AND MOLECULAR WEIGHT PROPERTIES OF TUBULIN SUBUNITS. Journal of Neurochemistry. 28(4). 697–706. 48 indexed citations

18.

Feit, Howard, Gary R. Dutton, Samuel H. Barondes, & Michael L. Shelanski. (1971). MICROTUBULE PROTEIN. The Journal of Cell Biology. 51(1). 138–147. 124 indexed citations

19.

Feit, Howard, et al.. (1971). Heterogeneity of Tubulin Subunits. Proceedings of the National Academy of Sciences. 68(9). 2028–2031. 108 indexed citations

20.

Feit, Howard & S H Barondes. (1970). COLCHICINE‐BINDING ACTIVITY IN PARTICULATE FRACTIONS OF MOUSE BRAIN¹. Journal of Neurochemistry. 17(9). 1355–1364. 160 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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