Hong‐Yu Hu
- Neurology top 2%
- Parkinson's Disease Mechanisms and Treatments 13
- Molecular Biology top 5%
- Ubiquitin and proteasome pathways 21
- Mitochondrial Function and Pathology 11
- Glycosylation and Glycoproteins Research 8
- Protein Structure and Dynamics 8
- Cell Biology top 5%
- Endoplasmic Reticulum Stress and Disease 7
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- Genetic Neurodegenerative Diseases 15
- Physiology top 10%
- Alzheimer's disease research and treatments 11
- Co-authors
- Hai‐Ning DuAi‐Xin SongHui YangZi‐Ren ZhouJun HuLeilei JiangLin TangYuhang Zhang
- Journals
- PLoS ONE (6 papers)Journal of Biological Chemistry (5 papers)Chinese Journal of Chemistry (4 papers)
- Partner nations
- ChinaUnited StatesUnited Kingdom
In The Last Decade
Hong‐Yu Hu
80 papers receiving 1.9k citations
Peers
Comparison fields: 5 of 113
- Neurology 470
- Molecular Biology 1.3k
- Cell Biology 298
- Cellular and Molecular Neuroscience 278
- Physiology 354
Countries citing papers authored by Hong‐Yu Hu
This map shows the geographic impact of Hong‐Yu Hu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hong‐Yu Hu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hong‐Yu Hu more than expected).
Fields of papers citing papers by Hong‐Yu Hu
This network shows the impact of papers produced by Hong‐Yu Hu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hong‐Yu Hu. The network helps show where Hong‐Yu Hu may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Hong‐Yu Hu, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2023 | 3 | |
| 3 | 2016 | 71 | |
| 4 | 2016 | 5 | |
| 5 | 2013 | 22 | |
| 6 | 2013 | 5 | |
| 7 | 2013 | 38 | |
| 8 | 2012 | 11 | |
| 9 | 2011 | 45 | |
| 10 | 2009 | 2 | |
| 11 | 2007 | 1 | |
| 12 | 2006 | 77 | |
| 13 | 2006 | 18 | |
| 14 | 2006 | 25 | |
| 15 | 2002 | 64 | |
| 16 | 2001 | 11 | |
| 17 | 2000 | 54 | |
| 18 | 1999 | 3 | |
| 19 | 1999 | 7 | |
| 20 | 1992 | 12 |
About Hong‐Yu Hu
Hong‐Yu Hu is a scholar working on Neurology, Cell Biology and Molecular Biology, having authored 83 papers that have together received 1.9k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (21 papers), Genetic Neurodegenerative Diseases (15 papers), Parkinson's Disease Mechanisms and Treatments (13 papers), Mitochondrial Function and Pathology (11 papers), Alzheimer's disease research and treatments (11 papers), Glycosylation and Glycoproteins Research (8 papers), Protein Structure and Dynamics (8 papers) and Endoplasmic Reticulum Stress and Disease (7 papers). The work is most often cited by research in Neurology (470 citations), Molecular Biology (1.3k citations) and Cell Biology (298 citations). Hong‐Yu Hu has collaborated with scholars based in China, United States and United Kingdom. Frequent co-authors include Hai‐Ning Du, Ai‐Xin Song, Hui Yang, Zi‐Ren Zhou, Jun Hu, Leilei Jiang, Lin Tang, Yuhang Zhang, Xuechao Gao and Shuai Liu. Their work appears in journals such as PLoS ONE, Journal of Biological Chemistry, Chinese Journal of Chemistry, Protein Science and Acta Biochimica et Biophysica Sinica.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.