Holger Richly
Impact in
- Aging top 5%
- Cell Biology top 2%
- Endoplasmic Reticulum Stress and Disease
Papers in
-
- Genomics and Chromatin Dynamics 12
- Epigenetics and DNA Methylation 9
- Ubiquitin and proteasome pathways 8
- DNA Repair Mechanisms 7
- RNA modifications and cancer 5
- CRISPR and Genetic Engineering 3
-
- Autophagy in Disease and Therapy 4
- Co-authors
- Stefan Jentsch (2 shared papers)Michael Rapé (2 shared papers)Sebastian Rumpf (2 shared papers)Luciano Di Croce (5 shared papers)Carsten Hoege (1 shared paper)Sigurd Braun (1 shared paper)Luigi Aloia (1 shared paper)Thorsten Hoppe (1 shared paper)
In The Last Decade
Holger Richly
25 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 81
- Aging 82
- Cell Biology 596
- Molecular Biology 1.4k
- Epidemiology 358
- Cancer Research 112
Countries citing papers authored by Holger Richly
This map shows the geographic impact of Holger Richly's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Holger Richly with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Holger Richly more than expected).
Fields of papers citing papers by Holger Richly
This network shows the impact of papers produced by Holger Richly. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Holger Richly. The network helps show where Holger Richly may publish in the future.
Co-authors
The 25 scholars most cited alongside Holger Richly, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 25 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2005 | 425 | |
| 2 | 2001 | 374 | |
| 3 | 2011 | 195 | |
| 4 | 2004 | 136 | |
| 5 | 2010 | 106 | |
| 6 | 2017 | 75 | |
| 7 | 2017 | 62 | |
| 8 | 2003 | 40 | |
| 9 | 2016 | 33 | |
| 10 | 2017 | 32 | |
| 11 | 2012 | 29 | |
| 12 | 2019 | 23 | |
| 13 | 2017 | 21 | |
| 14 | 2016 | 21 | |
| 15 | 2018 | 19 | |
| 16 | 2010 | 16 | |
| 17 | 2016 | 12 | |
| 18 | 2011 | 11 | |
| 19 | 2018 | 9 | |
| 20 | 2016 | 8 |
About Holger Richly
Holger Richly is a scholar working on Molecular Biology, Epidemiology, Cell Biology, Aging and Neurology, having authored 25 papers that have together received 1.7k indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (12 papers), Epigenetics and DNA Methylation (9 papers), Ubiquitin and proteasome pathways (8 papers), DNA Repair Mechanisms (7 papers), RNA modifications and cancer (5 papers), Autophagy in Disease and Therapy (4 papers), Endoplasmic Reticulum Stress and Disease (3 papers) and CRISPR and Genetic Engineering (3 papers). The work is most often cited by research in Aging (82 citations), Cell Biology (596 citations), Molecular Biology (1.4k citations), Epidemiology (358 citations) and Cancer Research (112 citations). Holger Richly has collaborated with scholars based in Germany, Spain and France. Frequent co-authors include Stefan Jentsch, Michael Rapé, Sebastian Rumpf, Luciano Di Croce, Carsten Hoege, Sigurd Braun, Luigi Aloia, Thorsten Hoppe, Ingo H. Gorr and Christian Schuberth. Their work appears in journals such as Cell Cycle, The Journal of Cell Biology, Cell, BioEssays and Oncotarget.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.