Hideaki Ito

6.1k citations
169 papers · 5.0k indexed · 1 hit paper · h-index 34

Hideaki Ito

154 papers receiving 4.9k citations

Hit Papers

Role of autophagy in temozolomide-induced cytotoxicity fo...8262004202620112018250500750

Peers

Hideaki Ito
Comparison fields: 5 of 135
  • Endocrinology 418
  • Physiology 205
  • Urology 275
  • Cancer Research 614
  • Molecular Medicine 200
Replace Bernard Rossi with:
Bernard Rossi France
Geertrui Denecker Belgium
Fengyi Wan United States
Baharia Mograbi France
Jin Zhang China
Eugenio Scanziani Italy
Sandra Barth Germany
Wenqing Gao China
Hang Thi Thu Nguyen United States
Jennifer Martinez United States
Hideaki Ito relative to Bernard Rossi France Bernard Rossi's profile →
Citations per field
00.5×4.7×
Bernard Rossi · 1×
Citations per year

Countries citing papers authored by Hideaki Ito

Since Specialization
Citations

This map shows the geographic impact of Hideaki Ito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideaki Ito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideaki Ito more than expected).

Fields of papers citing papers by Hideaki Ito

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hideaki Ito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideaki Ito. The network helps show where Hideaki Ito may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Hideaki Ito, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Hideaki Ito Line = papers co-authored together Hideaki Ito links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20240
2 20241
3 20221
4 202010
5 20196
6 20130
7 20120
8 20125
9 201112
10
Relationship between nocturia and metabolic syndrome
20092
11 200820
12 200616
13 199813
14 19976
15 199640
16 199611
17 19916
18 19914
19 19904
20 198916

About Hideaki Ito

Hideaki Ito is a scholar working on Urology, Endocrinology and Molecular Medicine, having authored 169 papers that have together received 5.0k indexed citations. Recurring topics across this work include Urinary Bladder and Prostate Research (25 papers), Pelvic floor disorders treatments (15 papers), Bacterial Genetics and Biotechnology (13 papers), Antibiotic Resistance in Bacteria (11 papers), Escherichia coli research studies (10 papers), Renal cell carcinoma treatment (8 papers), Urinary Tract Infections Management (8 papers) and Cancer Immunotherapy and Biomarkers (7 papers). The work is most often cited by research in Endocrinology (418 citations), Physiology (205 citations) and Urology (275 citations). Hideaki Ito has collaborated with scholars based in Japan, United States and Bangladesh. Frequent co-authors include Yasuko Kondo, Takao Kanzawa, Isabelle M. Germano, Seiji Kondo, Tadashi Komata, Sei Kondo, Yoshihiro Oyamada, Jun‐ichi Yamagishi, Osamu Yokoyama and Mikio Namiki. Their work appears in journals such as The Journal of Urology, Neurourology and Urodynamics, Journal of Medical Microbiology, Bulletin of the Chemical Society of Japan and International Journal of Urology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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