Jun‐ichi Niwa

1.4k total citations
40 papers, 1.1k citations indexed

About

Jun‐ichi Niwa is a scholar working on Neurology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Jun‐ichi Niwa has authored 40 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Neurology, 10 papers in Molecular Biology and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in Jun‐ichi Niwa's work include Amyotrophic Lateral Sclerosis Research (13 papers), Parkinson's Disease Mechanisms and Treatments (8 papers) and Neurogenetic and Muscular Disorders Research (7 papers). Jun‐ichi Niwa is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (13 papers), Parkinson's Disease Mechanisms and Treatments (8 papers) and Neurogenetic and Muscular Disorders Research (7 papers). Jun‐ichi Niwa collaborates with scholars based in Japan and United States. Jun‐ichi Niwa's co-authors include Gen Sobue, Manabu Doyu, Shinsuke Ishigaki, Fumiaki Tanaka, Masahisa Katsuno, Hideyuki Takeuchi, Tsuyoshi Yoshihara, Yasushi Kobayashi, Tetsuo Sakai and Masahiko Yamamoto and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and The Journal of Physical Chemistry B.

In The Last Decade

Jun‐ichi Niwa

38 papers receiving 1.1k citations

Peers

Jun‐ichi Niwa

NEURO

GENET

CMN

NEURO

Takahiro Takeda Japan

Erdong Liu China

Masashi Nakajima Japan

Yuanjing Liu China

Li Huang China

Maria Demestre Germany

Dejun Yang China

Akira Taniguchi Japan

Yuan Cheng China

A. Nascimento Spain

Takahiro Takeda Japan

Jun‐ichi Niwa

411 ×0.8

NEURO

134 ×0.3

133 ×0.5

GENET

70 ×0.4

CMN

106 ×0.7

NEURO

Citations per year, relative to Jun‐ichi Niwa Jun‐ichi Niwa (= 1×) peers Takahiro Takeda

Countries citing papers authored by Jun‐ichi Niwa

Since Specialization

Citations

This map shows the geographic impact of Jun‐ichi Niwa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun‐ichi Niwa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun‐ichi Niwa more than expected).

Fields of papers citing papers by Jun‐ichi Niwa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jun‐ichi Niwa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun‐ichi Niwa. The network helps show where Jun‐ichi Niwa may publish in the future.

Co-authorship network of co-authors of Jun‐ichi Niwa

This figure shows the co-authorship network connecting the top 25 collaborators of Jun‐ichi Niwa. A scholar is included among the top collaborators of Jun‐ichi Niwa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jun‐ichi Niwa. Jun‐ichi Niwa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ando, Hiroaki, Yuichi Kawagashira, Jun‐ichi Niwa, Akio Akagi, & Manabu Doyu. (2025). An Autopsy Case of Varicella-zoster Virus Uveitis Progressing to Vasculopathy with Multiple Cerebral Infarctions. Internal Medicine. 64(18). 2774–2778.

Niwa, Jun‐ichi, et al.. (2022). Recurring Weakness in Rhabdomyolysis Following Pfizer–BioNTech Coronavirus Disease 2019 mRNA Vaccination. Vaccines. 10(6). 935–935. 4 indexed citations

Niwa, Jun‐ichi, et al.. (2022). Multiple cerebral infarctions due to calcified amorphous tumor growing rapidly from an antecedent infection and decreasing rapidly by detachment of fibrin and antithrombotic drugs: a case report. BMC Neurology. 22(1). 391–391. 1 indexed citations

Azuma, Yoshiteru, et al.. (2022). Subcortical infarction in a young adult with Hunter syndrome. Brain and Development. 44(5). 343–346. 2 indexed citations

Ito, Chihiro, Hiroaki Ando, Keisuke Tokui, et al.. (2021). Antiparkinsonian drugs as potent contributors to nocturnal sleep in patients with Parkinson’s disease. PLoS ONE. 16(7). e0255274–e0255274. 4 indexed citations

Tanaka, Hirotaka, Masamitsu Shimazawa, Masafumi Takata, et al.. (2012). The potential of GPNMB as novel neuroprotective factor in amyotrophic lateral sclerosis. Scientific Reports. 2(1). 573–573. 108 indexed citations

Niwa, Jun‐ichi, et al.. (2012). Potent primary leptomeningeal lymphoma masquerading tuberculous meningitis -A case report. Rinsho Shinkeigaku. 52(6). 416–420. 4 indexed citations

Iguchi, Yohei, Masahisa Katsuno, Shinnosuke Takagi, et al.. (2011). Oxidative stress induced by glutathione depletion reproduces pathological modifications of TDP-43 linked to TDP-43 proteinopathies. Neurobiology of Disease. 45(3). 862–870. 79 indexed citations

Sone, Jun, Jun‐ichi Niwa, Kaori Kawai, et al.. (2009). Dorfin ameliorates phenotypes in a transgenic mouse model of amyotrophic lateral sclerosis. Journal of Neuroscience Research. 88(1). 123–135. 19 indexed citations

10.

Niwa, Jun‐ichi, Shin‐ichi Yamada, Shinsuke Ishigaki, et al.. (2007). Disulfide Bond Mediates Aggregation, Toxicity, and Ubiquitylation of Familial Amyotrophic Lateral Sclerosis-linked Mutant SOD1. Journal of Biological Chemistry. 282(38). 28087–28095. 136 indexed citations

11.

Yamada, Shin‐ichi, Jun‐ichi Niwa, Shinsuke Ishigaki, et al.. (2006). Archaeal Proteasomes Effectively Degrade Aggregation-prone Proteins and Reduce Cellular Toxicities in Mammalian Cells. Journal of Biological Chemistry. 281(33). 23842–23851. 10 indexed citations

12.

Huang, Ying, Jun‐ichi Niwa, Gen Sobue, & Gerda E. Breitwieser. (2006). Calcium-sensing Receptor Ubiquitination and Degradation Mediated by the E3 Ubiquitin Ligase Dorfin. Journal of Biological Chemistry. 281(17). 11610–11617. 69 indexed citations

13.

Ishigaki, Shinsuke, Jun‐ichi Niwa, Shin‐ichi Yamada, et al.. (2006). Dorfin-CHIP chimeric proteins potently ubiquitylate and degrade familial ALS-related mutant SOD1 proteins and reduce their cellular toxicity. Neurobiology of Disease. 25(2). 331–341. 31 indexed citations

14.

Takeuchi, Hideyuki, Jun‐ichi Niwa, Nozomi Hishikawa, et al.. (2004). Dorfin prevents cell death by reducing mitochondrial localizing mutant superoxide dismutase 1 in a neuronal cell model of familial amyotrophic lateral sclerosis. Journal of Neurochemistry. 89(1). 64–72. 18 indexed citations

15.

Takeuchi, Hideyuki, Yasushi Kobayashi, Tsuyoshi Yoshihara, et al.. (2002). Hsp70 and Hsp40 improve neurite outgrowth and suppress intracytoplasmic aggregate formation in cultured neuronal cells expressing mutant SOD1. Brain Research. 949(1-2). 11–22. 85 indexed citations

16.

Ishigaki, Shinsuke, Yideng Liang, Masahiko Yamamoto, et al.. (2002). X‐Linked inhibitor of apoptosis protein is involved in mutant SOD1‐mediated neuronal degeneration. Journal of Neurochemistry. 82(3). 576–584. 42 indexed citations

17.

Doyu, Manabu, Koichi Shinchi, Norimasa Mitsuma, et al.. (2001). Gene expression profile in Alzheimer’s brain screened by molecular indexing. Molecular Brain Research. 87(1). 1–11. 12 indexed citations

18.

Ito, Tomotaka, Mayumi Jijiwa, Yukio Ando, et al.. (2000). [A patient with choreoathetosis of the left upper extremity due to acute cerebral infarction].. PubMed. 40(2). 184–6. 2 indexed citations

19.

Ishigaki, Shinsuke, Jun‐ichi Niwa, Tsuyoshi Yoshihara, et al.. (2000). Two Novel Genes, Human neugrin and Mouse m-neugrin, Are Upregulated with Neuronal Differentiation in Neuroblastoma Cells. Biochemical and Biophysical Research Communications. 279(2). 526–533. 12 indexed citations

20.

Kobayashi, Kunihiko, Ryu‐Ichiro Hata, Shinya Nagai, Jun‐ichi Niwa, & Takeshi Hoshino. (1990). Direct visualization of affected collagen molecules synthesized by cultured fibroblasts from an osteogenesis imperfecta patient. Biochemical and Biophysical Research Communications. 172(1). 217–222. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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