Hadis Williams

406 total citations
8 papers, 148 citations indexed

About

Hadis Williams is a scholar working on Physiology, Pathology and Forensic Medicine and Molecular Biology. According to data from OpenAlex, Hadis Williams has authored 8 papers receiving a total of 148 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Physiology, 3 papers in Pathology and Forensic Medicine and 2 papers in Molecular Biology. Recurrent topics in Hadis Williams's work include Lysosomal Storage Disorders Research (7 papers), Biomedical Research and Pathophysiology (3 papers) and Cellular transport and secretion (2 papers). Hadis Williams is often cited by papers focused on Lysosomal Storage Disorders Research (7 papers), Biomedical Research and Pathophysiology (3 papers) and Cellular transport and secretion (2 papers). Hadis Williams collaborates with scholars based in United States, Japan and Australia. Hadis Williams's co-authors include David J. Lockhart, Sam Gandy, Eric R. Sjoberg, Brandon A. Wustman, Katsuhiko Yanagisawa, Joseph D. Buxbaum, Yi Lun, Richie Khanna, Kenneth J. Valenzano and Frédéric Checler and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Molecular Psychiatry.

In The Last Decade

Hadis Williams

8 papers receiving 145 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hadis Williams United States	4	124	49	35	35	22	8	148
Tamara Marín Chile	7	63 0.5×	48 1.0×	24 0.7×	19 0.5×	13 0.6×	7	119
Melis Köse Türkiye	8	59 0.5×	74 1.5×	22 0.6×	48 1.4×	26 1.2×	42	195
Nada Al Eisa United Kingdom	3	150 1.2×	37 0.8×	45 1.3×	19 0.5×	46 2.1×	3	175
Danijela Petković Ramadža Croatia	9	103 0.8×	134 2.7×	34 1.0×	34 1.0×	11 0.5×	26	280
Remy T. Schneider United States	4	139 1.1×	110 2.2×	24 0.7×	104 3.0×	9 0.4×	4	255
Hélio Pedro United States	8	50 0.4×	72 1.5×	13 0.4×	57 1.6×	33 1.5×	14	183
Tetsushi Kataura Japan	8	28 0.2×	111 2.3×	77 2.2×	22 0.6×	8 0.4×	19	224
Gema Tarrasón Spain	10	89 0.7×	179 3.7×	13 0.4×	32 0.9×	22 1.0×	21	324
Michele Sacchini Italy	6	70 0.6×	66 1.3×	20 0.6×	4 0.1×	15 0.7×	11	162
Rebecca Mardach United States	11	100 0.8×	193 3.9×	31 0.9×	82 2.3×	57 2.6×	17	351

Countries citing papers authored by Hadis Williams

Since Specialization

Citations

This map shows the geographic impact of Hadis Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hadis Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hadis Williams more than expected).

Fields of papers citing papers by Hadis Williams

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hadis Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hadis Williams. The network helps show where Hadis Williams may publish in the future.

Co-authorship network of co-authors of Hadis Williams

This figure shows the co-authorship network connecting the top 25 collaborators of Hadis Williams. A scholar is included among the top collaborators of Hadis Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hadis Williams. Hadis Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

Johnson, Franklin K., Shirley Wu, Hadis Williams, et al.. (2024). Pharmacokinetic evaluation of single-dose migalastat in non-Fabry disease subjects with ESRD receiving dialysis treatment, and use of modeling to select dose regimens in Fabry disease subjects with ESRD receiving dialysis treatment. PLoS ONE. 19(12). e0314030–e0314030. 3 indexed citations

Chien, Yin‐Hsiu, Benedikt Schoser, Priya S. Kishnani, et al.. (2022). Immunogenicity of cipaglucosidase alfa/miglustat versus alglucosidase alfa/placebo in late-onset Pompe disease (LOPD): A phase III, randomized study (PROPEL). Molecular Genetics and Metabolism. 135(2). S30–S30. 2 indexed citations

Nicholls, Kathy, et al.. (2019). The effects of long-term migalastat treatment in Fabry disease patients previously treated with enzyme replacement therapy who have migalastat-amenable variants with low alpha-galactosidase A response in the in vitro migalastat amenability assay. Molecular Genetics and Metabolism. 126(2). S108–S108. 1 indexed citations

Calligaro, Greg, et al.. (2018). The aetiology, clinical presentation and treatment of patients with pulmonary hypertension in Cape Town: A preliminary report from the Groote Schuur Hospital Pulmonary Hypertension Registry. PubMed. 24(4). 133–133. 3 indexed citations

Schiffmann, Raphael, Daniel G. Bichet, Dominique P. Germain, et al.. (2018). SP004EFFECTS OF LONG-TERM MIGALASTAT TREATMENT ON RENAL FUNCTION BY BASELINE PROTEINURIA IN PATIENTS (PTS) WITH FABRY DISEASE. Nephrology Dialysis Transplantation. 33(suppl_1). i347–i348. 4 indexed citations

Mauer, Michael, Jay Barth, Jeffrey P. Castelli, et al.. (2017). Reduction of podocyte globotriaosylceramide content in adult male patients with Fabry disease with amenable GLA mutations following 6 months of migalastat treatment. Journal of Medical Genetics. 54(11). 781–786. 35 indexed citations

Knight, Elysse M., Hadis Williams, A. Christopher Stevens, et al.. (2014). Evidence that small molecule enhancement of β-hexosaminidase activity corrects the behavioral phenotype in Dutch APPE693Q mice through reduction of ganglioside-bound Aβ. Molecular Psychiatry. 20(1). 109–117. 25 indexed citations

Lun, Yi, Anthony C. Stevens, Hadis Williams, et al.. (2012). Lysosomal Dysfunction in a Mouse Model of Sandhoff Disease Leads to Accumulation of Ganglioside-Bound Amyloid-β Peptide. Journal of Neuroscience. 32(15). 5223–5236. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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