Greta Pintacuda

3.0k total citations · 1 hit paper
20 papers, 1.9k citations indexed

About

Greta Pintacuda is a scholar working on Molecular Biology, Genetics and Neurology. According to data from OpenAlex, Greta Pintacuda has authored 20 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 6 papers in Genetics and 3 papers in Neurology. Recurrent topics in Greta Pintacuda's work include CRISPR and Genetic Engineering (6 papers), RNA Research and Splicing (6 papers) and RNA modifications and cancer (5 papers). Greta Pintacuda is often cited by papers focused on CRISPR and Genetic Engineering (6 papers), RNA Research and Splicing (6 papers) and RNA modifications and cancer (5 papers). Greta Pintacuda collaborates with scholars based in United Kingdom, United States and Italy. Greta Pintacuda's co-authors include Andrea Cerase, Neil Brockdorff, Tatyana B. Nesterova, Benoît Moindrot, Heather Coker, Osamu Masui, Lothar Schermelleh, Alexandra Sapetschnig, Leonard D. Goldstein and Eva‐Maria Weick and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Greta Pintacuda

18 papers receiving 1.9k citations

Hit Papers

piRNAs Can Trigger a Multigenerational Epigenetic Memory ... 2012 2026 2016 2021 2012 100 200 300 400 500

Peers

Greta Pintacuda
Frank M. J. Jacobs Netherlands
Györgyi Csankovszki United States
Xinxian Deng United States
Peri Tate United Kingdom
Justin Cotney United States
Irene Hernando-Herraez United Kingdom
Ian King United States
Greta Pintacuda
Citations per year, relative to Greta Pintacuda Greta Pintacuda (= 1×) peers Corinna Menzel

Countries citing papers authored by Greta Pintacuda

Since Specialization
Citations

This map shows the geographic impact of Greta Pintacuda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Greta Pintacuda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Greta Pintacuda more than expected).

Fields of papers citing papers by Greta Pintacuda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Greta Pintacuda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Greta Pintacuda. The network helps show where Greta Pintacuda may publish in the future.

Co-authorship network of co-authors of Greta Pintacuda

This figure shows the co-authorship network connecting the top 25 collaborators of Greta Pintacuda. A scholar is included among the top collaborators of Greta Pintacuda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Greta Pintacuda. Greta Pintacuda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pintacuda, Greta, et al.. (2026). Developmental convergence and divergence in human stem cell models of autism. Nature. 651(8106). 707–719.
2.
Pintacuda, Greta, Yu-Han H. Hsu, Kalliopi Tsafou, et al.. (2023). Protein interaction studies in human induced neurons indicate convergent biology underlying autism spectrum disorders. Cell Genomics. 3(3). 100250–100250. 22 indexed citations
3.
Juan, Irune Guerra San, Leslie A. Nash, Kevin S. Smith, et al.. (2022). Loss of mouse Stmn2 function causes motor neuropathy. Neuron. 110(10). 1671–1688.e6. 40 indexed citations
4.
Lage, Kasper, Greta Pintacuda, Yu-Han H. Hsu, et al.. (2022). INTERACTION STUDIES OF RISK PROTEINS IN HUMAN INDUCED NEURONS REVEAL CONVERGENT BIOLOGY AND NOVEL MECHANISMS UNDERLYING AUTISM SPECTRUM DISORDERS. European Neuropsychopharmacology. 63. e21–e21.
5.
Pintacuda, Greta, Yu-Han H. Hsu, Edyta Małolepsza, et al.. (2021). Genoppi is an open-source software for robust and standardized integration of proteomic and genetic data. Nature Communications. 12(1). 2580–2580. 15 indexed citations
6.
Wei, Guifeng, Mafalda Almeida, Greta Pintacuda, et al.. (2021). Acute depletion of METTL3 implicates N 6 -methyladenosine in alternative intron/exon inclusion in the nascent transcriptome. Genome Research. 31(8). 1395–1408. 47 indexed citations
7.
Klim, Joseph R., Greta Pintacuda, Leslie A. Nash, Irune Guerra San Juan, & Kevin Eggan. (2021). Connecting TDP-43 Pathology with Neuropathy. Trends in Neurosciences. 44(6). 424–440. 44 indexed citations
8.
Pintacuda, Greta, et al.. (2021). Mind the translational gap: using iPS cell models to bridge from genetic discoveries to perturbed pathways and therapeutic targets. Molecular Autism. 12(1). 10–10. 16 indexed citations
9.
Hartl, Christopher, Gokul Ramaswami, William G. Pembroke, et al.. (2021). Coexpression network architecture reveals the brain-wide and multiregional basis of disease susceptibility. Nature Neuroscience. 24(9). 1313–1323. 41 indexed citations
10.
Nesterova, Tatyana B., Guifeng Wei, Heather Coker, et al.. (2019). Systematic allelic analysis defines the interplay of key pathways in X chromosome inactivation. Nature Communications. 10(1). 3129–3129. 100 indexed citations
11.
Gdula, Michał R., Tatyana B. Nesterova, Greta Pintacuda, et al.. (2018). The non-canonical SMC protein SmcHD1 antagonises TAD formation and compartmentalisation on the inactive X chromosome. Nature Communications. 10(1). 30–30. 80 indexed citations
12.
Jansz, Natasha, Tatyana B. Nesterova, Andrew Keniry, et al.. (2018). Smchd1 Targeting to the Inactive X Is Dependent on the Xist-HnrnpK-PRC1 Pathway. Cell Reports. 25(7). 1912–1923.e9. 51 indexed citations
13.
Almeida, Mafalda, Greta Pintacuda, Osamu Masui, et al.. (2017). PCGF3/5–PRC1 initiates Polycomb recruitment in X chromosome inactivation. Science. 356(6342). 1081–1084. 198 indexed citations
14.
Stewart, Emma, Tatyana B. Nesterova, Heather Coker, et al.. (2017). The nuclear matrix protein CIZ1 facilitates localization of Xist RNA to the inactive X-chromosome territory. Genes & Development. 31(9). 876–888. 90 indexed citations
15.
Pintacuda, Greta, et al.. (2017). Function by Structure: Spotlights on Xist Long Non-coding RNA. Frontiers in Molecular Biosciences. 4. 90–90. 73 indexed citations
16.
Pintacuda, Greta, Guifeng Wei, Chloë Roustan, et al.. (2017). hnRNPK Recruits PCGF3/5-PRC1 to the Xist RNA B-Repeat to Establish Polycomb-Mediated Chromosomal Silencing. Molecular Cell. 68(5). 955–969.e10. 227 indexed citations
17.
Cerase, Andrea, Greta Pintacuda, Anna Tattermusch, & Philip Avner. (2015). Xist localization and function: new insights from multiple levels. Genome biology. 16(1). 166–166. 130 indexed citations
18.
Pintacuda, Greta & Andrea Cerase. (2015). X Inactivation Lessons from Differentiating Mouse Embryonic Stem Cells. Stem Cell Reviews and Reports. 11(5). 699–705. 14 indexed citations
19.
Moindrot, Benoît, Andrea Cerase, Heather Coker, et al.. (2015). A Pooled shRNA Screen Identifies Rbm15, Spen, and Wtap as Factors Required for Xist RNA-Mediated Silencing. Cell Reports. 12(4). 562–572. 206 indexed citations
20.
Ashe, Alyson, Alexandra Sapetschnig, Eva‐Maria Weick, et al.. (2012). piRNAs Can Trigger a Multigenerational Epigenetic Memory in the Germline of C. elegans. Cell. 150(1). 88–99. 521 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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