Aaron Burberry

3.0k total citations · 2 hit papers
16 papers, 1.9k citations indexed

About

Aaron Burberry is a scholar working on Neurology, Molecular Biology and Physiology. According to data from OpenAlex, Aaron Burberry has authored 16 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Neurology, 6 papers in Molecular Biology and 4 papers in Physiology. Recurrent topics in Aaron Burberry's work include Amyotrophic Lateral Sclerosis Research (7 papers), Alzheimer's disease research and treatments (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Aaron Burberry is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (7 papers), Alzheimer's disease research and treatments (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Aaron Burberry collaborates with scholars based in United States, Netherlands and Belgium. Aaron Burberry's co-authors include Gabriel Núñez, Yuumi Nakamura, Kevin Eggan, Yun‐Gi Kim, Luigi Franchi, Raúl Muñoz-Planillo, Yuan He, Saranyaraajan Varadarajan, Daniel A. Mordes and Peter Kuffa and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Neuron.

In The Last Decade

Aaron Burberry

16 papers receiving 1.9k citations

Hit Papers

ALS-implicated protein TDP-43 sustains levels of STMN2, a... 2019 2026 2021 2023 2019 2023 100 200 300

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Aaron Burberry United States 14 1.1k 595 426 367 191 16 1.9k
Laxminarayana R. Devireddy United States 19 735 0.7× 133 0.2× 370 0.9× 297 0.8× 200 1.0× 26 2.3k
Vincenzo Mattei Italy 28 1.1k 1.0× 71 0.1× 252 0.6× 260 0.7× 214 1.1× 87 2.0k
Nicole R. Bianco United States 27 1.5k 1.4× 163 0.3× 470 1.1× 100 0.3× 64 0.3× 54 2.5k
Chung‐May Yang Taiwan 37 794 0.7× 750 1.3× 164 0.4× 50 0.1× 81 0.4× 252 4.7k
Yara Zayed Canada 7 2.7k 2.5× 62 0.1× 364 0.9× 100 0.3× 181 0.9× 7 3.9k
Yoshihiro Yoshida Japan 24 497 0.5× 104 0.2× 212 0.5× 54 0.1× 104 0.5× 104 1.8k
Jarrod A. Dudakov United States 24 586 0.5× 96 0.2× 1.4k 3.3× 96 0.3× 164 0.9× 65 2.6k
Axel Weber Germany 14 707 0.7× 59 0.1× 560 1.3× 63 0.2× 86 0.5× 25 1.6k
Michael H. Askenase United States 11 294 0.3× 276 0.5× 598 1.4× 25 0.1× 76 0.4× 12 1.3k
Rafael Valdés‐Mas Spain 15 1.1k 1.0× 43 0.1× 175 0.4× 273 0.7× 241 1.3× 23 1.8k

Countries citing papers authored by Aaron Burberry

Since Specialization
Citations

This map shows the geographic impact of Aaron Burberry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aaron Burberry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aaron Burberry more than expected).

Fields of papers citing papers by Aaron Burberry

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aaron Burberry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aaron Burberry. The network helps show where Aaron Burberry may publish in the future.

Co-authorship network of co-authors of Aaron Burberry

This figure shows the co-authorship network connecting the top 25 collaborators of Aaron Burberry. A scholar is included among the top collaborators of Aaron Burberry based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aaron Burberry. Aaron Burberry is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Limone, Francesco, Alexander Benavides Couto, Jinyuan Wang, et al.. (2024). Myeloid and lymphoid expression of C9orf72 regulates IL-17A signaling in mice. Science Translational Medicine. 16(732). eadg7895–eadg7895. 7 indexed citations
2.
Limone, Francesco, Daniel A. Mordes, Alexander Benavides Couto, et al.. (2024). Single-nucleus sequencing reveals enriched expression of genetic risk factors in extratelencephalic neurons sensitive to degeneration in ALS. Nature Aging. 4(7). 984–997. 13 indexed citations
4.
Cao, Xuan, et al.. (2023). ggpicrust2: an R package for PICRUSt2 predicted functional profile analysis and visualization. Bioinformatics. 39(8). 124 indexed citations breakdown →
5.
Juan, Irune Guerra San, Leslie A. Nash, Kevin S. Smith, et al.. (2022). Loss of mouse Stmn2 function causes motor neuropathy. Neuron. 110(10). 1671–1688.e6. 40 indexed citations
6.
Burberry, Aaron, Michael F. Wells, Francesco Limone, et al.. (2020). C9orf72 suppresses systemic and neural inflammation induced by gut bacteria. Nature. 582(7810). 89–94. 189 indexed citations
7.
Klim, Joseph R., Luis A. Williams, Francesco Limone, et al.. (2019). ALS-implicated protein TDP-43 sustains levels of STMN2, a mediator of motor neuron growth and repair. Nature Neuroscience. 22(2). 167–179. 345 indexed citations breakdown →
8.
Zhang, Yingying, Aaron Burberry, Jackson Sandoe, et al.. (2018). The C9orf72-interacting protein Smcr8 is a negative regulator of autoimmunity and lysosomal exocytosis. Genes & Development. 32(13-14). 929–943. 50 indexed citations
9.
Burberry, Aaron, Naoki Suzuki, Rob Moccia, et al.. (2016). Loss-of-function mutations in the C9ORF72 mouse ortholog cause fatal autoimmune disease. Science Translational Medicine. 8(347). 347ra93–347ra93. 188 indexed citations
10.
Burberry, Aaron, Melody Yue Zeng, Lei Ding, et al.. (2014). Infection Mobilizes Hematopoietic Stem Cells through Cooperative NOD-like Receptor and Toll-like Receptor Signaling. Cell Host & Microbe. 15(6). 779–791. 139 indexed citations
11.
Warner, Neil, et al.. (2014). A Genome-wide Small Interfering RNA (siRNA) Screen Reveals Nuclear Factor-κB (NF-κB)-independent Regulators of NOD2-induced Interleukin-8 (IL-8) Secretion. Journal of Biological Chemistry. 289(41). 28213–28224. 35 indexed citations
12.
He, Yuan, Saranyaraajan Varadarajan, Raúl Muñoz-Planillo, et al.. (2013). 3,4-Methylenedioxy-β-nitrostyrene Inhibits NLRP3 Inflammasome Activation by Blocking Assembly of the Inflammasome. Journal of Biological Chemistry. 289(2). 1142–1150. 233 indexed citations
13.
Marchesan, Julie T., Andrei D. Taut, Seung‐Yun Shin, et al.. (2013). Porphyromonas gingivalis oral infection exacerbates the development and severity of collagen-induced arthritis. Arthritis Research & Therapy. 15(6). R186–R186. 92 indexed citations
14.
Warner, Neil, Aaron Burberry, Luigi Franchi, et al.. (2013). A Genome-Wide siRNA Screen Reveals Positive and Negative Regulators of the NOD2 and NF-κB Signaling Pathways. Science Signaling. 6(258). rs3–rs3. 52 indexed citations
15.
Franchi, Luigi, Nobuhiko Kamada, Yuumi Nakamura, et al.. (2012). NLRC4-driven production of IL-1β discriminates between pathogenic and commensal bacteria and promotes host intestinal defense. Nature Immunology. 13(5). 449–456. 323 indexed citations
16.
Feng, Ying, Guido T. Bommer, Jenny Zhao, et al.. (2011). Mutant Kras Promotes Hyperplasia and Alters Differentiation in the Colon Epithelium but Does Not Expand the Presumptive Stem Cell Pool. Gastroenterology. 141(3). 1003–1013.e10. 99 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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