Gregorio Valdez

4.0k total citations · 1 hit paper
52 papers, 3.0k citations indexed

About

Gregorio Valdez is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Gregorio Valdez has authored 52 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 24 papers in Cellular and Molecular Neuroscience and 12 papers in Neurology. Recurrent topics in Gregorio Valdez's work include Muscle Physiology and Disorders (17 papers), Nerve injury and regeneration (15 papers) and Amyotrophic Lateral Sclerosis Research (10 papers). Gregorio Valdez is often cited by papers focused on Muscle Physiology and Disorders (17 papers), Nerve injury and regeneration (15 papers) and Amyotrophic Lateral Sclerosis Research (10 papers). Gregorio Valdez collaborates with scholars based in United States, Canada and Italy. Gregorio Valdez's co-authors include Joshua R. Sanes, Jeff W. Lichtman, Juan Carlos Tapia, Thomas Taetzsch, Xiaoxia Qi, Jeffrey L. Elliott, John McAnally, Rhonda Bassel‐Duby, Simon Halegoua and Eric N. Olson and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Gregorio Valdez

48 papers receiving 3.0k citations

Hit Papers

MicroRNA-206 Delays ALS Progression and Promotes Regenera... 2009 2026 2014 2020 2009 100 200 300 400 500

Peers

Gregorio Valdez
Jérôme Mertens United States
Marı́a Laura Messi United States
Jingli Cai United States
Gihan Tennekoon United States
Gregorio Valdez
Citations per year, relative to Gregorio Valdez Gregorio Valdez (= 1×) peers Wado Akamatsu

Countries citing papers authored by Gregorio Valdez

Since Specialization
Citations

This map shows the geographic impact of Gregorio Valdez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gregorio Valdez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gregorio Valdez more than expected).

Fields of papers citing papers by Gregorio Valdez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gregorio Valdez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gregorio Valdez. The network helps show where Gregorio Valdez may publish in the future.

Co-authorship network of co-authors of Gregorio Valdez

This figure shows the co-authorship network connecting the top 25 collaborators of Gregorio Valdez. A scholar is included among the top collaborators of Gregorio Valdez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gregorio Valdez. Gregorio Valdez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mueller, Kaly A., et al.. (2025). Comprehensive characterization and validation of the Prp-hPFN1G118V mouse model: Guidelines for preclinical therapeutic testing for ALS. Neurobiology of Disease. 212. 106975–106975. 1 indexed citations
2.
Valdez, Gregorio, et al.. (2024). Origin, identity, and function of terminal Schwann cells. Trends in Neurosciences. 47(6). 432–446. 10 indexed citations
3.
Valdez, Gregorio. (2023). NEUROSCIENCE AND AGING. Innovation in Aging. 7(Supplement_1). 221–221.
4.
Biase, Lindsay M. De, et al.. (2023). Aging spinal cord microglia become phenotypically heterogeneous and preferentially target motor neurons and their synapses. Glia. 72(1). 206–221. 3 indexed citations
5.
Settlage, Robert E., et al.. (2023). Aging alters mechanisms underlying voluntary movements in spinal motor neurons of mice, primates, and humans. JCI Insight. 8(9). 12 indexed citations
6.
Taetzsch, Thomas, et al.. (2022). Argonaute 2 is lost from neuromuscular junctions affected with amyotrophic lateral sclerosis in SOD1G93A mice. Scientific Reports. 12(1). 4630–4630.
7.
Taetzsch, Thomas, et al.. (2021). Roles of the synaptic molecules Hevin and SPARC in mouse neuromuscular junction development and repair. Neuroscience Letters. 746. 135663–135663. 5 indexed citations
8.
Haldar, Saptarsi M., et al.. (2021). KLF15 overexpression in myocytes fails to ameliorate ALS-related pathology or extend the lifespan of SOD1G93A mice. Neurobiology of Disease. 162. 105583–105583. 2 indexed citations
9.
Taetzsch, Thomas, et al.. (2020). The microRNA miR‐133b functions to slow Duchenne muscular dystrophy pathogenesis. The Journal of Physiology. 599(1). 171–192. 15 indexed citations
10.
Doss, Sydney V., et al.. (2019). Attenuating Cholinergic Transmission Increases the Number of Satellite Cells and Preserves Muscle Mass in Old Age. Frontiers in Aging Neuroscience. 11. 262–262. 8 indexed citations
11.
Taetzsch, Thomas & Gregorio Valdez. (2018). NMJ maintenance and repair in aging. Current Opinion in Physiology. 4. 57–64. 58 indexed citations
12.
Motta‐Santos, Daisy, Sydney V. Doss, Patrícia Massara Martinelli, et al.. (2018). Fast and slow-twitching muscles are differentially affected by reduced cholinergic transmission in mice deficient for VAChT: A mouse model for congenital myasthenia. Neurochemistry International. 120. 1–12. 11 indexed citations
13.
Taetzsch, Thomas, et al.. (2016). Muscle Fibers Secrete FGFBP1 to Slow Degeneration of Neuromuscular Synapses during Aging and Progression of ALS. Journal of Neuroscience. 37(1). 70–82. 55 indexed citations
14.
Valdez, Gregorio, Juan Carlos Tapia, Hyuno Kang, et al.. (2010). Attenuation of age-related changes in mouse neuromuscular synapses by caloric restriction and exercise. Proceedings of the National Academy of Sciences. 107(33). 14863–14868. 406 indexed citations
15.
Philippidou, Polyxeni, Gregorio Valdez, Wendy Akmentin, et al.. (2010). Trk retrograde signaling requires persistent, Pincher-directed endosomes. Proceedings of the National Academy of Sciences. 108(2). 852–857. 50 indexed citations
16.
Carlson, Steven S., Gregorio Valdez, & Joshua R. Sanes. (2010). Presynaptic calcium channels and α3‐integrins are complexed with synaptic cleft laminins, cytoskeletal elements and active zone components. Journal of Neurochemistry. 115(3). 654–666. 48 indexed citations
17.
Valdez, Gregorio, Viviana Moresi, Xiaoxia Qi, et al.. (2009). MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice. Science. 326(5959). 1549–1554. 588 indexed citations breakdown →
18.
Nishimune, Hiroshi, Gregorio Valdez, George Jarad, et al.. (2008). Laminins promote postsynaptic maturation by an autocrine mechanism at the neuromuscular junction. The Journal of Cell Biology. 182(6). 1201–1215. 100 indexed citations
19.
Valdez, Gregorio, Wendy Akmentin, Polyxeni Philippidou, et al.. (2005). Pincher-Mediated Macroendocytosis Underlies Retrograde Signaling by Neurotrophin Receptors. Journal of Neuroscience. 25(21). 5236–5247. 108 indexed citations
20.
Kuruvilla, Rejji, Larry S. Zweifel, Natalia O. Glebova, et al.. (2004). A Neurotrophin Signaling Cascade Coordinates Sympathetic Neuron Development through Differential Control of TrkA Trafficking and Retrograde Signaling. Cell. 118(2). 243–255. 299 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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