Thomas Taetzsch

1.1k total citations
18 papers, 824 citations indexed

About

Thomas Taetzsch is a scholar working on Molecular Biology, Neurology and Neurology. According to data from OpenAlex, Thomas Taetzsch has authored 18 papers receiving a total of 824 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Neurology and 4 papers in Neurology. Recurrent topics in Thomas Taetzsch's work include Muscle Physiology and Disorders (8 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Amyotrophic Lateral Sclerosis Research (4 papers). Thomas Taetzsch is often cited by papers focused on Muscle Physiology and Disorders (8 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Amyotrophic Lateral Sclerosis Research (4 papers). Thomas Taetzsch collaborates with scholars based in United States and Netherlands. Thomas Taetzsch's co-authors include Gregorio Valdez, Michelle L. Block, Shannon Levesque, Jo Anne Johnson, Melinda E. Lull, Krisztián Stadler, Michael Surace, Prasada Rao S. Kodavanti, Constance McGraw and Alison F. Wagner and has published in prestigious journals such as Journal of Neuroscience, The Journal of Physiology and Scientific Reports.

In The Last Decade

Thomas Taetzsch

17 papers receiving 813 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Taetzsch United States 13 301 268 174 117 106 18 824
G. Li United States 5 183 0.6× 244 0.9× 197 1.1× 70 0.6× 186 1.8× 6 715
Shannon Levesque United States 9 638 2.1× 138 0.5× 282 1.6× 247 2.1× 66 0.6× 9 1.1k
Michael Surace United States 11 486 1.6× 235 0.9× 201 1.2× 196 1.7× 49 0.5× 20 990
Paula Korhonen Finland 18 75 0.2× 327 1.2× 306 1.8× 17 0.1× 156 1.5× 33 950
Zhou Ou China 16 70 0.2× 202 0.8× 119 0.7× 13 0.1× 70 0.7× 30 644
Hengsheng Chen China 15 182 0.6× 187 0.7× 85 0.5× 14 0.1× 205 1.9× 48 672
Natàlia Vilor‐Tejedor Spain 15 216 0.7× 131 0.5× 80 0.5× 71 0.6× 56 0.5× 45 732
Majid Sheykhzade Denmark 23 267 0.9× 380 1.4× 71 0.4× 14 0.1× 334 3.2× 84 1.5k
Toyoko Arimoto Japan 7 217 0.7× 151 0.6× 168 1.0× 12 0.1× 118 1.1× 9 603
Mireya Alcaraz-Zubeldia Mexico 16 214 0.7× 134 0.5× 79 0.5× 17 0.1× 130 1.2× 26 777

Countries citing papers authored by Thomas Taetzsch

Since Specialization
Citations

This map shows the geographic impact of Thomas Taetzsch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Taetzsch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Taetzsch more than expected).

Fields of papers citing papers by Thomas Taetzsch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Taetzsch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Taetzsch. The network helps show where Thomas Taetzsch may publish in the future.

Co-authorship network of co-authors of Thomas Taetzsch

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Taetzsch. A scholar is included among the top collaborators of Thomas Taetzsch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Taetzsch. Thomas Taetzsch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Taetzsch, Thomas, et al.. (2022). Argonaute 2 is lost from neuromuscular junctions affected with amyotrophic lateral sclerosis in SOD1G93A mice. Scientific Reports. 12(1). 4630–4630.
2.
Taetzsch, Thomas, et al.. (2021). Roles of the synaptic molecules Hevin and SPARC in mouse neuromuscular junction development and repair. Neuroscience Letters. 746. 135663–135663. 5 indexed citations
3.
Haldar, Saptarsi M., et al.. (2021). KLF15 overexpression in myocytes fails to ameliorate ALS-related pathology or extend the lifespan of SOD1G93A mice. Neurobiology of Disease. 162. 105583–105583. 2 indexed citations
4.
Taetzsch, Thomas, et al.. (2020). The microRNA miR‐133b functions to slow Duchenne muscular dystrophy pathogenesis. The Journal of Physiology. 599(1). 171–192. 15 indexed citations
5.
Taetzsch, Thomas, et al.. (2020). Specific labeling of synaptic schwann cells reveals unique cellular and molecular features. eLife. 9. 52 indexed citations
6.
Taetzsch, Thomas, et al.. (2019). Loss of NF-κB p50 function synergistically augments microglial priming in the middle-aged brain. Journal of Neuroinflammation. 16(1). 60–60. 14 indexed citations
7.
Zeitz, Μ., Thomas Taetzsch, Kijana K. George, et al.. (2019). Dynamic UTR Usage Regulates Alternative Translation to Modulate Gap Junction Formation during Stress and Aging. Cell Reports. 27(9). 2737–2747.e5. 22 indexed citations
8.
Hyer, Molly M., et al.. (2019). Sex modifies the consequences of extended fructose consumption on liver health, motor function, and physiological damage in rats. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 317(6). R903–R911. 17 indexed citations
9.
Taetzsch, Thomas & Gregorio Valdez. (2018). NMJ maintenance and repair in aging. Current Opinion in Physiology. 4. 57–64. 58 indexed citations
10.
Taetzsch, Thomas, et al.. (2018). FGF binding proteins (FGFBPs): Modulators of FGF signaling in the developing, adult, and stressed nervous system. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864(9). 2983–2991. 25 indexed citations
11.
Taetzsch, Thomas, et al.. (2017). Muscle Fibers Secrete FGFBP1 to Slow Degeneration of Neuromuscular Synapses during Aging and Progression of ALS. Journal of Neuroscience. 37(1). 70–82. 2 indexed citations
12.
Taetzsch, Thomas, et al.. (2016). Muscle Fibers Secrete FGFBP1 to Slow Degeneration of Neuromuscular Synapses during Aging and Progression of ALS. Journal of Neuroscience. 37(1). 70–82. 55 indexed citations
13.
Taetzsch, Thomas, et al.. (2016). The Fibular Nerve Injury Method: A Reliable Assay to Identify and Test Factors That Repair Neuromuscular Junctions. Journal of Visualized Experiments. 13 indexed citations
14.
Taetzsch, Thomas, et al.. (2016). The Fibular Nerve Injury Method: A Reliable Assay to Identify and Test Factors That Repair Neuromuscular Junctions. Journal of Visualized Experiments. 6 indexed citations
15.
Taetzsch, Thomas, Shannon Levesque, Constance McGraw, et al.. (2014). Redox regulation of NF‐κB p50 and M1 polarization in microglia. Glia. 63(3). 423–440. 116 indexed citations
16.
Taetzsch, Thomas & Michelle L. Block. (2013). Pesticides, Microglial NOX2, and Parkinson's Disease. Journal of Biochemical and Molecular Toxicology. 27(2). 137–149. 47 indexed citations
17.
Levesque, Shannon, Thomas Taetzsch, Melinda E. Lull, et al.. (2013). The role of MAC1 in diesel exhaust particle‐induced microglial activation and loss of dopaminergic neuron function. Journal of Neurochemistry. 125(5). 756–765. 82 indexed citations
18.
Levesque, Shannon, Thomas Taetzsch, Melinda E. Lull, et al.. (2011). Diesel Exhaust Activates and Primes Microglia: Air Pollution, Neuroinflammation, and Regulation of Dopaminergic Neurotoxicity. Environmental Health Perspectives. 119(8). 1149–1155. 293 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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