Giuseppe Pilia
- Molecular Biology top 5%
- Genetics top 1%
- Public Health, Environmental and Occupational Health top 5%
- Reproductive Medicine top 2%
- Cell Biology top 5%
- Co-authors
- David SchlessingerAntonino ForaboscoAntonio CaoLaura CrisponiManuela UdaChris OttolenghiReid HuberAlex MacKenzie
- Topics
- Genetic Syndromes and Imprinting (11 papers)Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (8 papers)RNA modifications and cancer (8 papers)
- Partner nations
- United StatesItalyFrance
In The Last Decade
Giuseppe Pilia
40 papers receiving 2.9k citations
Hit Papers
Peers
Comparison fields: 5 of 102
- Molecular Biology 2.0k
- Genetics 1.5k
- Public Health, Environmental and Occupational Health 449
- Reproductive Medicine 329
- Cell Biology 321
Countries citing papers authored by Giuseppe Pilia
This map shows the geographic impact of Giuseppe Pilia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giuseppe Pilia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giuseppe Pilia more than expected).
Fields of papers citing papers by Giuseppe Pilia
This network shows the impact of papers produced by Giuseppe Pilia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giuseppe Pilia. The network helps show where Giuseppe Pilia may publish in the future.
Co-authorship network of co-authors of Giuseppe Pilia
This figure shows the co-authorship network connecting the top 25 collaborators of Giuseppe Pilia. A scholar is included among the top collaborators of Giuseppe Pilia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Giuseppe Pilia. Giuseppe Pilia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 26 | |
| 2 | 67 | |
| 3 | 42 | |
| 4 | 109 | |
| 5 | 217 | |
| 6 | 42 | |
| 7 | 36 | |
| 8 | 36 | |
| 9 | 1 | |
| 10 | 2 | |
| 11 | 94 | |
| 12 | 9 | |
| 13 | 58 | |
| 14 | Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndromebreakdown → | 593 |
| 15 | 16 | |
| 16 | 13 | |
| 17 | 8 | |
| 18 | 6 | |
| 19 | 16 | |
| 20 | 1 |
About Giuseppe Pilia
Giuseppe Pilia is a scholar working on Genetics, Molecular Biology and Developmental Biology, having authored 41 papers that have together received 2.9k indexed citations. Recurring topics across this work include Genetic Syndromes and Imprinting (11 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (8 papers) and RNA modifications and cancer (8 papers). The work is most often cited by research in Genetics (1.5k citations), Reproductive Medicine (329 citations) and Molecular Biology (2.0k citations). Giuseppe Pilia has collaborated with scholars based in United States, Italy and France. Frequent co-authors include David Schlessinger, Antonino Forabosco, Antonio Cao, Laura Crisponi, Manuela Uda, Chris Ottolenghi, Reid Huber, David Schlessinger, Alex MacKenzie and Ellson Y. Chen. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Genetics and Genome Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.