David Schlessinger
- Genetics top 2%
- Molecular Biology
- Public Health, Environmental and Occupational Health top 5%
- Reproductive Medicine top 2%
- Plant Science
- Co-authors
- Manuela UdaAntonino ForaboscoChris OttolenghiLaura CrisponiGiuseppe PiliaAntonio CaoManila DeianaJosé Elías García‐Ortíz
- Topics
- Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (6 papers)Bacterial Genetics and Biotechnology (3 papers)Reproductive Biology and Fertility (3 papers)
- Partner nations
- United StatesItalyMexico
In The Last Decade
David Schlessinger
11 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 75
- Genetics 729
- Molecular Biology 688
- Public Health, Environmental and Occupational Health 344
- Reproductive Medicine 291
- Plant Science 68
Countries citing papers authored by David Schlessinger
This map shows the geographic impact of David Schlessinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Schlessinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Schlessinger more than expected).
Fields of papers citing papers by David Schlessinger
This network shows the impact of papers produced by David Schlessinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Schlessinger. The network helps show where David Schlessinger may publish in the future.
Co-authorship network of co-authors of David Schlessinger
This figure shows the co-authorship network connecting the top 25 collaborators of David Schlessinger. A scholar is included among the top collaborators of David Schlessinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Schlessinger. David Schlessinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 23 | |
| 2 | 39 | |
| 3 | 115 | |
| 4 | 261 | |
| 5 | 415 | |
| 6 | 23 | |
| 7 | 25 | |
| 8 | 26 | |
| 9 | Potentiation of rifampicin, rifampicin analogs, and tetracycline against animal cells by amphotericin B and polymyxin B. | 34 |
| 10 | 46 | |
| 11 | 60 |
About David Schlessinger
David Schlessinger is a scholar working on Genetics, Reproductive Medicine and Molecular Medicine, having authored 11 papers that have together received 1.1k indexed citations. Recurring topics across this work include Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (6 papers), Bacterial Genetics and Biotechnology (3 papers) and Reproductive Biology and Fertility (3 papers). The work is most often cited by research in Reproductive Medicine (291 citations), Genetics (729 citations) and Physiology (68 citations). David Schlessinger has collaborated with scholars based in United States, Italy and Mexico. Frequent co-authors include Manuela Uda, Antonino Forabosco, Chris Ottolenghi, Laura Crisponi, Giuseppe Pilia, Antonio Cao, Manila Deiana, José Elías García‐Ortíz, José Elías García and Shakib Omari. Their work appears in journals such as Science, Nucleic Acids Research and Human Molecular Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.