David Schlessinger

65.9k total citations · 3 hit papers
362 papers, 15.8k citations indexed

About

David Schlessinger is a scholar working on Molecular Biology, Genetics and Ecology. According to data from OpenAlex, David Schlessinger has authored 362 papers receiving a total of 15.8k indexed citations (citations by other indexed papers that have themselves been cited), including 263 papers in Molecular Biology, 100 papers in Genetics and 29 papers in Ecology. Recurrent topics in David Schlessinger's work include RNA and protein synthesis mechanisms (113 papers), RNA modifications and cancer (72 papers) and Bacterial Genetics and Biotechnology (40 papers). David Schlessinger is often cited by papers focused on RNA and protein synthesis mechanisms (113 papers), RNA modifications and cancer (72 papers) and Bacterial Genetics and Biotechnology (40 papers). David Schlessinger collaborates with scholars based in United States, Italy and Japan. David Schlessinger's co-authors include David Apirion, Anand Srivastava, Gerald Medoff, Chang‐Yi Cui, Manuela Uda, A. Tissières, Giuseppe Pilia, Antonino Forabosco, G S Kobayashi and James D. Watson and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

David Schlessinger

360 papers receiving 14.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Ribonucleoprotein particles from Escherichia coli

1959 597 citations David Schlessinger et al. profile →
Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndrome

1996 593 citations Giuseppe Pilia, Alex MacKenzie et al. Nature Genetics profile →
X–linked anhidrotic (hypohidrotic) ectodermal dysplasia is caused by mutation in a novel transmembrane protein

1996 557 citations Primo Baybayan, Ellson Y. Chen et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David Schlessinger United States	65	10.1k	3.9k	1.1k	990	962	362	15.8k
Lawrence I. Grossman United States	71	11.4k 1.1×	2.2k 0.6×	700 0.6×	770 0.8×	868 0.9×	302	15.9k
William B. Guggino United States	75	13.5k 1.3×	4.7k 1.2×	1.5k 1.3×	1.1k 1.1×	990 1.0×	248	22.4k
Pierre Gounon France	68	6.5k 0.6×	2.6k 0.7×	1.4k 1.3×	2.2k 2.3×	728 0.8×	158	14.4k
Stephen F. Kingsmore United States	54	9.2k 0.9×	3.3k 0.8×	765 0.7×	1.4k 1.4×	537 0.6×	193	14.9k
Lakshminarayan M. Iyer United States	56	10.7k 1.1×	2.3k 0.6×	528 0.5×	993 1.0×	1.8k 1.8×	115	14.8k
Leslie A. Leinwand United States	88	13.5k 1.3×	3.0k 0.8×	2.0k 1.9×	1.5k 1.5×	536 0.6×	330	24.9k
Donald D. Brown United States	91	14.0k 1.4×	4.4k 1.1×	1.1k 1.0×	795 0.8×	2.2k 2.3×	373	25.8k
Dale D. Dykes United States	17	6.7k 0.7×	4.9k 1.3×	632 0.6×	1.3k 1.3×	484 0.5×	47	18.2k
Carl R. Merril United States	44	7.3k 0.7×	2.3k 0.6×	989 0.9×	498 0.5×	1.3k 1.4×	153	12.7k
Douglas M. Ruden United States	35	5.7k 0.6×	3.6k 0.9×	610 0.6×	718 0.7×	2.2k 2.3×	104	12.3k

Countries citing papers authored by David Schlessinger

Since Specialization

Citations

This map shows the geographic impact of David Schlessinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Schlessinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Schlessinger more than expected).

Fields of papers citing papers by David Schlessinger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Schlessinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Schlessinger. The network helps show where David Schlessinger may publish in the future.

Co-authorship network of co-authors of David Schlessinger

This figure shows the co-authorship network connecting the top 25 collaborators of David Schlessinger. A scholar is included among the top collaborators of David Schlessinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Schlessinger. David Schlessinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kim, Jung-Hyun, Ramaiah Nagaraja, Vladimir N. Noskov, et al.. (2024). Comparative analysis and classification of highly divergent mouse rDNA units based on their intergenic spacer (IGS) variability. NAR Genomics and Bioinformatics. 6(2). lqae070–lqae070. 1 indexed citations

Serra, Rita, Antonio Pinna, Andrea Angius, et al.. (2024). Vascular density normative data of radial peripapillary capillary plexus in healthy Caucasian subjects. Graefe s Archive for Clinical and Experimental Ophthalmology. 263(1). 97–103. 1 indexed citations

Ganau, Antonello, Marco Orrù, Matteo Floris, et al.. (2023). Echocardiographic heart ageing patterns predict cardiovascular and non-cardiovascular events and reflect biological age: the SardiNIA study. European Journal of Preventive Cardiology. 31(6). 677–685. 8 indexed citations

Balliu, Brunilda, Matthew G. Durrant, Nathan S. Abell, et al.. (2019). Genetic regulation of gene expression and splicing during a 10-year period of human aging. Genome biology. 20(1). 230–230. 47 indexed citations

Cui, Chang‐Yi, Ji Heon Noh, Marc Michel, Myriam Gorospe, & David Schlessinger. (2017). STIM1, but not STIM2, Is the Calcium Sensor Critical for Sweat Secretion. Journal of Investigative Dermatology. 138(3). 704–707. 3 indexed citations

Correa-Cerro, Lina S., Yulan Piao, Alexei A. Sharov, et al.. (2011). Generation of mouse ES cell lines engineered for the forced induction of transcription factors. Scientific Reports. 1(1). 167–167. 38 indexed citations

Mumm, Steven, Luisa Herrera, Paul Waeltz, et al.. (2001). χ/Autosomal Translocations in the χq Critical Region Associated with Premature Ovarian Failure Fall within and outside Genes. Genomics. 76(1-3). 30–36. 33 indexed citations

Huber, Reid, Serafino Pantano, Ellson Y. Chen, et al.. (2000). PLAC1, an Xq26 Gene with Placenta-Specific Expression. Genomics. 68(3). 305–312. 97 indexed citations

Huber, Reid, R. Scott Hansen, Maria Strazzullo, et al.. (1999). DNA methylation in transcriptional repression of two differentially expressed X-linked genes,GPC3andSYBL1. Proceedings of the National Academy of Sciences. 96(2). 616–621. 42 indexed citations

10.

Huber, Reid, David Schlessinger, & Giuseppe Pilia. (1998). Multiple Sp1 sites efficiently drive transcription of the TATA-less promoter of the human glypican 3 (GPC3) gene. Gene. 214(1-2). 35–44. 36 indexed citations

11.

Esposito, Teresa, Alfredo Ciccodicola, Maria R. Matarazzo, et al.. (1997). Expressed STSs and transcription of human Xq28. Gene. 187(2). 185–191. 1 indexed citations

12.

Porta, Giovanni, Sandra MacMillan, Ramaiah Nagaraja, et al.. (1997). 4.5-Mb YAC STS contig at 50-kb resolution, spanning Xq25 deletions in two patients with lymphoproliferative syndrome.. Genome Research. 7(1). 27–36. 9 indexed citations

13.

Dixon, Peter, C Wooding, Dorothy Trump, et al.. (1996). Seven novel mutations in the PEX gene indicate molecular heterogeneity for X-linked hypophosphataemic rickets.. Journal of Bone and Mineral Research. 11. 165–165. 1 indexed citations

14.

Pilia, Giuseppe, Alex MacKenzie, Primo Baybayan, et al.. (1996). Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndrome. Nature Genetics. 12(3). 241–247. 593 indexed citations breakdown →

15.

Torigoe, K, Seiji Sato, Hitoshi Kusaba, et al.. (1995). A YAC-based contig of 1.5 Mb spanning the human multidrug resistance gene region and delineating the amplification unit in three human multidrug-resistant cell lines.. Genome Research. 5(3). 233–244. 19 indexed citations

16.

Zucchi, Ileana & David Schlessinger. (1992). Distribution of moderately repetitive sequences pTR5 and LF1 in Xq24–q28 human DNA and their use in assembling YAC contigs. Genomics. 12(2). 264–275. 20 indexed citations

17.

Bowman, Lewis, et al.. (1983). Location of the Initial Cleavage Sites in Mouse Pre-rRNA. Molecular and Cellular Biology. 3(8). 1501–1510. 18 indexed citations

18.

Schlessinger, David. (1974). Ribosome Formation in Escherichia coli. Cold Spring Harbor Monograph Archive. 4. 393–416. 9 indexed citations

19.

Medoff, Gerald, Fred Valeriote, Richard G. Lynch, David Schlessinger, & George S. Kobayashi. (1974). Jag orkar inte mer. Cancer Research. 34(5). 974–8. 49 indexed citations

20.

Silengo, Lorenzo, David Schlessinger, G Mangiarotti, & David Apirion. (1967). Induction of mutations to streptomycin and spectinomycin resistance in Escherichia coli by N-methyl-N′-nitroso-N-nitroguanidine and acridine half-mustard ICR-191. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 4(5). 701–703. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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