Géraldine Liot
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 1%
- Physiology top 5%
- Neurology top 2%
- Cell Biology top 5%
- Co-authors
- Frédéric SaudouDiana ZalaElla Bossy‐WetzelBlaise BossyMark H. EllismanGuy PerkinsYulia KushnarevaDenis Vivien
- Topics
- Mitochondrial Function and Pathology (10 papers)Genetic Neurodegenerative Diseases (8 papers)Protease and Inhibitor Mechanisms (5 papers)
- Partner nations
- FranceUnited StatesSwitzerland
In The Last Decade
Géraldine Liot
27 papers receiving 3.1k citations
Hit Papers
Peers
Comparison fields: 5 of 100
- Molecular Biology 2.2k
- Cellular and Molecular Neuroscience 1.2k
- Physiology 627
- Neurology 466
- Cell Biology 443
Countries citing papers authored by Géraldine Liot
This map shows the geographic impact of Géraldine Liot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Géraldine Liot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Géraldine Liot more than expected).
Fields of papers citing papers by Géraldine Liot
This network shows the impact of papers produced by Géraldine Liot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Géraldine Liot. The network helps show where Géraldine Liot may publish in the future.
Co-authorship network of co-authors of Géraldine Liot
This figure shows the co-authorship network connecting the top 25 collaborators of Géraldine Liot. A scholar is included among the top collaborators of Géraldine Liot based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Géraldine Liot. Géraldine Liot is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 28 | |
| 2 | 50 | |
| 3 | 17 | |
| 4 | 131 | |
| 5 | 352 | |
| 6 | 72 | |
| 7 | 446 | |
| 8 | 123 | |
| 9 | 205 | |
| 10 | 126 | |
| 11 | 268 | |
| 12 | 61 | |
| 13 | Nitric oxide‐induced mitochondrial fission is regulated by dynamin‐related GTPases in neuronsbreakdown → | 561 |
| 14 | 111 | |
| 15 | 60 | |
| 16 | 52 | |
| 17 | 16 | |
| 18 | 14 | |
| 19 | 125 | |
| 20 | 91 |
About Géraldine Liot
Géraldine Liot is a scholar working on Cellular and Molecular Neuroscience, Cell Biology and Neurology, having authored 27 papers that have together received 3.2k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (10 papers), Genetic Neurodegenerative Diseases (8 papers) and Protease and Inhibitor Mechanisms (5 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.2k citations), Developmental Neuroscience (160 citations) and Clinical Biochemistry (256 citations). Géraldine Liot has collaborated with scholars based in France, United States and Switzerland. Frequent co-authors include Frédéric Saudou, Diana Zala, Ella Bossy‐Wetzel, Blaise Bossy, Mark H. Ellisman, Guy Perkins, Yulia Kushnareva, Denis Vivien, Hélène Rangone and Sandrine Humbert. Their work appears in journals such as Cell, Journal of Biological Chemistry and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.