Gennaro Schettini

198 papers receiving 7.9k citations

Peers

Gennaro Schettini
Comparison fields: 5 of 132
  • Neurology 1.0k
  • Biological Psychiatry 243
  • Cellular and Molecular Neuroscience 1.6k
  • Behavioral Neuroscience 259
  • Developmental Neuroscience 280
Replace Louis B. Hersh with:
Louis B. Hersh United States
Patrick Kitabgi France
Paul E. Gottschall United States
Christian Kaltschmidt Germany
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Citations per year

Countries citing papers authored by Gennaro Schettini

Since Specialization
Citations

This map shows the geographic impact of Gennaro Schettini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gennaro Schettini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gennaro Schettini more than expected).

Fields of papers citing papers by Gennaro Schettini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gennaro Schettini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gennaro Schettini. The network helps show where Gennaro Schettini may publish in the future.

Co-authors

The 25 scholars most cited alongside Gennaro Schettini, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gennaro Schettini Line = papers co-authored together Gennaro Schettini links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 200888
2 200619
3 2006130
4 200451
5 2000125
6 200060
7 199940
8 199814
9 199721
10 199747
11 199742
12 199422
13 19939
14 19903
15 19906
16 198910
17 19882
18 19881
19 198818
20
Role of tuberoinfundibular dopaminergic neurons on TRH-TSH secretion.
19773

About Gennaro Schettini

Gennaro Schettini is a scholar working on Behavioral Neuroscience, Cellular and Molecular Neuroscience, Endocrinology, Diabetes and Metabolism, Neurology and Molecular Biology, having authored 198 papers that have together received 8.2k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (31 papers), Neuroscience and Neuropharmacology Research (24 papers), Growth Hormone and Insulin-like Growth Factors (22 papers), Pituitary Gland Disorders and Treatments (19 papers), Receptor Mechanisms and Signaling (18 papers), Prion Diseases and Protein Misfolding (18 papers), Ion channel regulation and function (17 papers) and Chemokine receptors and signaling (16 papers). The work is most often cited by research in Neurology (1.0k citations), Biological Psychiatry (243 citations), Cellular and Molecular Neuroscience (1.6k citations), Behavioral Neuroscience (259 citations) and Developmental Neuroscience (280 citations). Gennaro Schettini has collaborated with scholars based in Italy, United States and Japan. Frequent co-authors include Tullio Florio, Adriana Bajetto, Simone Barbero, Rudy Bonavia, Claudio Russo, Stefano Thellung, Olimpia Meucci, Antonella Scorziello, Alessandro Corsaro and Carola Porcile. Their work appears in journals such as Annals of the New York Academy of Sciences, Journal of Neurochemistry, Endocrinology, Pharmacological Research and Biochemical and Biophysical Research Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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