Ewa Golańska

917 total citations
30 papers, 535 citations indexed

About

Ewa Golańska is a scholar working on Molecular Biology, Physiology and Neurology. According to data from OpenAlex, Ewa Golańska has authored 30 papers receiving a total of 535 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 6 papers in Physiology and 5 papers in Neurology. Recurrent topics in Ewa Golańska's work include Prion Diseases and Protein Misfolding (13 papers), Alzheimer's disease research and treatments (6 papers) and Neurological diseases and metabolism (5 papers). Ewa Golańska is often cited by papers focused on Prion Diseases and Protein Misfolding (13 papers), Alzheimer's disease research and treatments (6 papers) and Neurological diseases and metabolism (5 papers). Ewa Golańska collaborates with scholars based in Poland, Germany and United States. Ewa Golańska's co-authors include Paweł P. Liberski, Piotr Rieske, Małgorzata Szybka, Inga Zerr, Franc Llorens, Beata Sikorska, Matthias Schmitz, Maria Barcikowska, Maria Styczyńska and Beata Pepłońska and has published in prestigious journals such as PLoS ONE, Neurology and Analytical Biochemistry.

In The Last Decade

Ewa Golańska

30 papers receiving 528 citations

Peers

Ewa Golańska

NEURO

PHYSI

NEURO

Ichiro Nozaki Japan

M. Miyazono Japan

Malene Ambjørn Denmark

Hanna Mierzewska Poland

Elena Panzeri Italy

Karin Seid Germany

Juraj Sepčić Croatia

Elisa Maggioli Italy

Yukihiko Kohda Japan

Yoshio Morimatsu Japan

Ichiro Nozaki Japan

Ewa Golańska

350 ×0.9

168 ×1.1

NEURO

88 ×0.8

PHYSI

100 ×1.1

NEURO

45 ×0.6

Citations per year, relative to Ewa Golańska Ewa Golańska (= 1×) peers Ichiro Nozaki

Countries citing papers authored by Ewa Golańska

Since Specialization

Citations

This map shows the geographic impact of Ewa Golańska's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ewa Golańska with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ewa Golańska more than expected).

Fields of papers citing papers by Ewa Golańska

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ewa Golańska. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ewa Golańska. The network helps show where Ewa Golańska may publish in the future.

Co-authorship network of co-authors of Ewa Golańska

This figure shows the co-authorship network connecting the top 25 collaborators of Ewa Golańska. A scholar is included among the top collaborators of Ewa Golańska based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ewa Golańska. Ewa Golańska is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kanata, Ειrini, Ewa Golańska, Anna Villar‐Piqué, et al.. (2018). Cerebrospinal fluid neurofilament light in suspected sporadic Creutzfeldt-Jakob disease. Journal of Clinical Neuroscience. 60. 124–127. 21 indexed citations

Llorens, Franc, André Karch, Ewa Golańska, et al.. (2017). Cerebrospinal Fluid Biomarker-Based Diagnosis of Sporadic Creutzfeldt-Jakob Disease: A Validation Study for Previously Established Cutoffs. Dementia and Geriatric Cognitive Disorders. 43(1-2). 71–80. 13 indexed citations

Llorens, Franc, Niels Kruse, André Karch, et al.. (2017). Validation of α-Synuclein as a CSF Biomarker for Sporadic Creutzfeldt-Jakob Disease. Molecular Neurobiology. 55(3). 2249–2257. 29 indexed citations

Golańska, Ewa, Małgorzata Szybka, Monika Rudzińska, et al.. (2015). Screening for THAP1 Mutations in Polish Patients with Dystonia Shows Known and Novel Substitutions. PLoS ONE. 10(6). e0129656–e0129656. 2 indexed citations

Schmitz, Matthias, Katharina Stoeck, André Karch, et al.. (2015). Validation of 14-3-3 Protein as a Marker in Sporadic Creutzfeldt-Jakob Disease Diagnostic. Molecular Neurobiology. 53(4). 2189–2199. 74 indexed citations

Golańska, Ewa, et al.. (2014). High variability of clinical symptoms in a Polish family with a novelTHAP1mutation. International Journal of Neuroscience. 125(10). 755–759. 6 indexed citations

Golańska, Ewa, Anna Pfeffer, Małgorzata Chodakowska-Żebrowska, et al.. (2013). APBB2 genetic polymorphisms are associated with severe cognitive impairment in centenarians. Experimental Gerontology. 48(4). 391–394. 12 indexed citations

Golańska, Ewa, Elizabeth H. Corder, Anna Pfeffer, et al.. (2013). The prion protein M129V polymorphism. Prion. 7(3). 244–247. 6 indexed citations

Golańska, Ewa, et al.. (2012). PRND3′UTR polymorphism may be associated with behavioral disturbances  in Alzheimer disease. Prion. 6(1). 73–80. 13 indexed citations

10.

Rieske, Piotr, Ewa Golańska, Magdalena Zakrzewska, et al.. (2009). Arrested neural and advanced mesenchymal differentiation of glioblastoma cells-comparative study with neural progenitors. BMC Cancer. 9(1). 54–54. 42 indexed citations

11.

Woźniak, Kinga, Sylwester Piaskowski, Ewa Golańska, et al.. (2008). BCR expression is decreased in meningiomas showing loss of heterozygosity of 22q within a new minimal deletion region. Cancer Genetics and Cytogenetics. 183(1). 14–20. 17 indexed citations

12.

Golańska, Ewa, Elizabeth H. Corder, Maria Styczyńska, et al.. (2008). Analysis of APBB2 gene polymorphisms in sporadic Alzheimer’s disease. Neuroscience Letters. 447(2-3). 164–166. 8 indexed citations

13.

Szybka, Małgorzata, Izabela Zawlik, Ewa Golańska, et al.. (2008). Elimination of wild-type P53 mRNA in glioblastomas showing heterozygous mutations of P53. British Journal of Cancer. 98(8). 1431–1433. 12 indexed citations

14.

Zakrzewska, Magdalena, Małgorzata Szybka, Krzysztof Zakrzewski, et al.. (2007). Diverse molecular pattern in a bihemispheric glioblastoma (butterfly glioma) in a 16-year-old boy. Cancer Genetics and Cytogenetics. 177(2). 125–130. 10 indexed citations

15.

Zawlik, Izabela, Magdalena Zakrzewska, Ewa Golańska, et al.. (2006). KCTD11 expression in medulloblastoma is lower than in adult cerebellum and higher than in neural stem cells. Cancer Genetics and Cytogenetics. 170(1). 24–28. 7 indexed citations

16.

Bratosiewicz‐Wąsik, Jolanta, et al.. (2006). Regulatory sequences of the PRNP gene influence susceptibility to sporadic Creutzfeldt–Jakob disease. Neuroscience Letters. 411(3). 163–167. 12 indexed citations

17.

Safranow, Krzysztof, Maria Styczyńska, Katarzyna Jakubowska, et al.. (2006). Prion protein gene M129 allele is a risk factor for Alzheimer’s disease. Journal of Neural Transmission. 113(11). 1747–1751. 23 indexed citations

18.

Golańska, Ewa, Piotr Rieske, Maria Styczyńska, et al.. (2005). CYP46: A risk factor for Alzheimer's disease or a coincidence?. Neuroscience Letters. 383(1-2). 105–108. 19 indexed citations

19.

Szybka, Małgorzata, Ewa Golańska, Piotr Rieske, et al.. (2005). Abnormalities of the P53, MDM2, BCL2 and BAX genes in acute leukemias.. PubMed. 52(4). 318–24. 44 indexed citations

20.

Golańska, Ewa, Ewa Rutkiewicz, Maria Styczyńska, et al.. (2004). Polymorphisms within the prion (PrP) and prion-like protein (Doppel) genes in AD. Neurology. 62(2). 313–315. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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