Robert Stawski
Impact in
- Genetics top 5%
- Glioma Diagnosis and Treatment
- Cancer Research top 10%
- MicroRNA in disease regulation
- Cancer Genomics and Diagnostics
- Cancer, Hypoxia, and Metabolism
- Cancer-related molecular mechanisms research
Papers in
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- Chromatin Remodeling and Cancer 3
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- Cancer Genomics and Diagnostics 6
- MicroRNA in disease regulation 3
- Co-authors
- Dariusz Nowak (15 shared papers)Ewelina Perdas (10 shared papers)Maria Zubrzycka (4 shared papers)Piotr Białasiewicz (4 shared papers)Agata Gabryelska (2 shared papers)Marcin Sochal (2 shared papers)Bartosz Szmyd (3 shared papers)Sumihito Nobusawa (2 shared papers)
- Journals
- International Journal of Molecular Sciences (4 papers)Biology (4 papers)Scientific Reports (3 papers)British Journal of Cancer (2 papers)Journal of Clinical Medicine (1 paper)
- Partner nations
- PolandUnited StatesFrance
In The Last Decade
Robert Stawski
30 papers receiving 746 citations
Peers
Comparison fields: 5 of 87
- Genetics 274
- Cancer Research 283
- Endocrine and Autonomic Systems 59
- Physiology 121
- Neurology 65
Countries citing papers authored by Robert Stawski
This map shows the geographic impact of Robert Stawski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Stawski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Stawski more than expected).
Fields of papers citing papers by Robert Stawski
This network shows the impact of papers produced by Robert Stawski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Stawski. The network helps show where Robert Stawski may publish in the future.
Co-authors
The 25 scholars most cited alongside Robert Stawski, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 32 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2010 | 175 | |
| 2 | 2020 | 72 | |
| 3 | 2011 | 71 | |
| 4 | 2017 | 55 | |
| 5 | 2016 | 44 | |
| 6 | 2011 | 37 | |
| 7 | 2020 | 33 | |
| 8 | 2010 | 32 | |
| 9 | 2020 | 29 | |
| 10 | 2018 | 20 | |
| 11 | 2019 | 18 | |
| 12 | 2021 | 18 | |
| 13 | 2008 | 17 | |
| 14 | 2019 | 17 | |
| 15 | 2022 | 13 | |
| 16 | 2012 | 13 | |
| 17 | Spontaneous in vitro senescence of glioma cells confirmed by an antibody against IDH1R132H. | 2014 | 13 |
| 18 | 2008 | 12 | |
| 19 | 2009 | 11 | |
| 20 | 2008 | 11 |
About Robert Stawski
Robert Stawski is a scholar working on Molecular Biology, Cancer Research, Genetics, Oncology and Physiology, having authored 32 papers that have together received 753 indexed citations. Recurring topics across this work include Cancer Genomics and Diagnostics (6 papers), Glioma Diagnosis and Treatment (6 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers), MicroRNA in disease regulation (3 papers), Chromatin Remodeling and Cancer (3 papers), Cancer-related Molecular Pathways (3 papers), High Altitude and Hypoxia (3 papers) and Exercise and Physiological Responses (3 papers). The work is most often cited by research in Genetics (274 citations), Cancer Research (283 citations), Endocrine and Autonomic Systems (59 citations), Physiology (121 citations) and Neurology (65 citations). Robert Stawski has collaborated with scholars based in Poland, United States and France. Frequent co-authors include Dariusz Nowak, Ewelina Perdas, Maria Zubrzycka, Piotr Białasiewicz, Agata Gabryelska, Marcin Sochal, Bartosz Szmyd, Sumihito Nobusawa, Young Ho Kim and Yoichi Nakazato. Their work appears in journals such as International Journal of Molecular Sciences, Biology, Scientific Reports, British Journal of Cancer and Journal of Clinical Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.