Eunjin Bae

519 total citations
16 papers, 337 citations indexed

About

Eunjin Bae is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Oncology. According to data from OpenAlex, Eunjin Bae has authored 16 papers receiving a total of 337 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 3 papers in Pulmonary and Respiratory Medicine and 3 papers in Oncology. Recurrent topics in Eunjin Bae's work include TGF-β signaling in diseases (7 papers), Chronic Kidney Disease and Diabetes (2 papers) and Chronic Myeloid Leukemia Treatments (2 papers). Eunjin Bae is often cited by papers focused on TGF-β signaling in diseases (7 papers), Chronic Kidney Disease and Diabetes (2 papers) and Chronic Myeloid Leukemia Treatments (2 papers). Eunjin Bae collaborates with scholars based in South Korea, United States and Japan. Eunjin Bae's co-authors include Seong‐Jin Kim, Akira Ooshima, Suntaek Hong, Fang Liu, Yunshin Jung, Kazuhito Naka, Junil Kim, Joon Jeong, Jungsoo Gim and Kaori Ishihara and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and PLoS ONE.

In The Last Decade

Eunjin Bae

15 papers receiving 336 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Eunjin Bae South Korea	12	226	91	68	45	33	16	337
Liran Zhou United States	8	213 0.9×	100 1.1×	72 1.1×	64 1.4×	40 1.2×	10	324
Juan Lin China	9	235 1.0×	81 0.9×	111 1.6×	38 0.8×	41 1.2×	14	365
Tressa M. Allington United States	4	220 1.0×	163 1.8×	81 1.2×	49 1.1×	36 1.1×	4	345
Yann Duchartre United States	5	369 1.6×	94 1.0×	107 1.6×	48 1.1×	17 0.5×	7	467
Astrid Grottke Germany	9	281 1.2×	136 1.5×	87 1.3×	59 1.3×	19 0.6×	14	403
Mara Mazzoni Italy	10	196 0.9×	117 1.3×	58 0.9×	59 1.3×	55 1.7×	14	330
Neha Zutshi United States	9	214 0.9×	112 1.2×	35 0.5×	25 0.6×	50 1.5×	12	358
Giovanni Antico United States	9	147 0.7×	85 0.9×	72 1.1×	40 0.9×	25 0.8×	12	278
Sian Taylor United Kingdom	6	177 0.8×	125 1.4×	44 0.6×	39 0.9×	20 0.6×	8	302
Stephen Hillerman United States	4	241 1.1×	118 1.3×	105 1.5×	80 1.8×	15 0.5×	5	427

Countries citing papers authored by Eunjin Bae

Since Specialization

Citations

This map shows the geographic impact of Eunjin Bae's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eunjin Bae with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eunjin Bae more than expected).

Fields of papers citing papers by Eunjin Bae

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eunjin Bae. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eunjin Bae. The network helps show where Eunjin Bae may publish in the future.

Co-authorship network of co-authors of Eunjin Bae

This figure shows the co-authorship network connecting the top 25 collaborators of Eunjin Bae. A scholar is included among the top collaborators of Eunjin Bae based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eunjin Bae. Eunjin Bae is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Kwon, Soie, Kyu Hong Kim, Eunjin Bae, et al.. (2024). Unveiling the role of transgelin as a prognostic and therapeutic target in kidney fibrosis via a proteomic approach. Experimental & Molecular Medicine. 56(10). 2296–2308. 1 indexed citations

Bae, Eunjin, et al.. (2024). Amyloid-β-activated microglia can induce compound proteinopathies. Brain. 147(12). 4105–4120. 3 indexed citations

Bae, Eunjin, Mitsuyasu Kato, Keiji Miyazawa, et al.. (2017). Repression of Smad3 by Stat3 and c-Ski/SnoN induces gefitinib resistance in lung adenocarcinoma. Biochemical and Biophysical Research Communications. 484(2). 269–277. 21 indexed citations

Yang, Kyung‐Min, Eunjin Bae, Sung Gwe Ahn, et al.. (2017). Co-chaperone BAG2 Determines the Pro-oncogenic Role of Cathepsin B in Triple-Negative Breast Cancer Cells. Cell Reports. 21(10). 2952–2964. 45 indexed citations

Naka, Kazuhito, Kaori Ishihara, Junil Kim, et al.. (2015). Dipeptide species regulate p38MAPK–Smad3 signalling to maintain chronic myelogenous leukaemia stem cells. Nature Communications. 6(1). 8039–8039. 48 indexed citations

Naka, Kazuhito, Kaori Ishihara, Cheng Hua Jin, et al.. (2015). Novel oral transforming growth factor‐β signaling inhibitor EW‐7197 eradicates CML‐initiating cells. Cancer Science. 107(2). 140–148. 28 indexed citations

Liu, Fang, Wen Xie, Eunjin Bae, et al.. (2015). Abstract 4940: Role of Smad3 linker phosphorylation in breast cancer progression. Cancer Research. 75(15_Supplement). 4940–4940. 1 indexed citations

Bae, Eunjin, Misako Sato, Ran‐Ju Kim, et al.. (2014). Definition of Smad3 Phosphorylation Events That Affect Malignant and Metastatic Behaviors in Breast Cancer Cells. Cancer Research. 74(21). 6139–6149. 29 indexed citations

Kim, Ran‐Ju, Eunjin Bae, Young Kwon Hong, et al.. (2014). PTEN Loss-Mediated Akt Activation Increases the Properties of Cancer Stem-Like Cell Populations in Prostate Cancer. Oncology. 87(5). 270–279. 18 indexed citations

10.

Bae, Eunjin, Akira Ooshima, & Seong‐Jin Kim. (2014). Abstract 1043: Distinctive roles of Smad3 C-tail and linker phosphorylation between cancer progression and suppression in TGF-beta1/Smad3 signal. Cancer Research. 74(19_Supplement). 1043–1043. 1 indexed citations

11.

Hong, Suntaek, Hye-Youn Kim, Jooyoung Kim, et al.. (2012). Smad7 Protein Induces Interferon Regulatory Factor 1-dependent Transcriptional Activation of Caspase 8 to Restore Tumor Necrosis Factor-related Apoptosis-inducing Ligand (TRAIL)-mediated Apoptosis. Journal of Biological Chemistry. 288(5). 3560–3570. 21 indexed citations

12.

Lim, Seunghwan, Eunjin Bae, Kyunghee Byun, et al.. (2012). TRAF6 Mediates IL-1β/LPS-Induced Suppression of TGF-β Signaling through Its Interaction with the Type III TGF-β Receptor. PLoS ONE. 7(3). e32705–e32705. 18 indexed citations

13.

Bae, Eunjin, Seong‐Jin Kim, Suntaek Hong, Fang Liu, & Akira Ooshima. (2012). Smad3 linker phosphorylation attenuates Smad3 transcriptional activity and TGF-β1/Smad3-induced epithelial–mesenchymal transition in renal epithelial cells. Biochemical and Biophysical Research Communications. 427(3). 593–599. 25 indexed citations

14.

Kim, Wonjoo, Eunjin Bae, Jungsoo Gim, et al.. (2012). DRAK2 Participates in a Negative Feedback Loop to Control TGF-β/Smads Signaling by Binding to Type I TGF-β Receptor. Cell Reports. 2(5). 1286–1299. 33 indexed citations

15.

Tsai, Chun-Ming, John Wen-Cheng Chang, Heung Tae Kim, et al.. (2011). Cost‐effectiveness of bevacizumab‐based therapy versus cisplatin plus pemetrexed for the first‐line treatment of advanced non‐squamous NSCLC in Korea and Taiwan. Asia-Pacific Journal of Clinical Oncology. 7(s2). 22–33. 20 indexed citations

16.

Jeon, Woo-Kwang, et al.. (2010). 14-3-3 Sigma and 14-3-3 Zeta Plays an Opposite Role in Cell Growth Inhibition Mediated by Transforming Growth Factor-Beta 1. Molecules and Cells. 29(3). 305–310. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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