Mitsutaka Kadota

2.5k citations
47 papers · 1.1k indexed · h-index 20
Topics
Genomics and Chromatin Dynamics (12 papers)Epigenetics and DNA Methylation (11 papers)Genomics and Phylogenetic Studies (9 papers)

In The Last Decade

Mitsutaka Kadota

43 papers receiving 1.1k citations

Peers

Mitsutaka Kadota
Comparison fields: 5 of 95
  • Molecular Biology 805
  • Genetics 284
  • Cancer Research 173
  • Oncology 146
  • Plant Science 84
Replace Arnaud Krebs with:
Arnaud Krebs Switzerland
Xiongfeng Chen China
Michael J. Clarkson Australia
Mary E. Donohoe United States
Monika Bialecka Netherlands
Kathryn J. Brayer United States
Alexey Ruzov United Kingdom
Roman Alpatov United States
Hannah K. Long United Kingdom
Hatice S Kaya-Okur United States
Mitsutaka Kadota relative to Arnaud Krebs Switzerland Arnaud Krebs's profile →
Citations per field
00.5×1.6×
Arnaud Krebs · 1×
Citations per year

Countries citing papers authored by Mitsutaka Kadota

Since Specialization
Citations

This map shows the geographic impact of Mitsutaka Kadota's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mitsutaka Kadota with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mitsutaka Kadota more than expected).

Fields of papers citing papers by Mitsutaka Kadota

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mitsutaka Kadota. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mitsutaka Kadota. The network helps show where Mitsutaka Kadota may publish in the future.

Co-authorship network of co-authors of Mitsutaka Kadota

This figure shows the co-authorship network connecting the top 25 collaborators of Mitsutaka Kadota. A scholar is included among the top collaborators of Mitsutaka Kadota based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mitsutaka Kadota. Mitsutaka Kadota is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 1
3 1
4 2
5 30
6 12
7 31
8 32
9 5
10 4
11 1
12 8
13 3
14 27
15 29
16 119
17 97
18 45
19 9
20 54

About Mitsutaka Kadota

Mitsutaka Kadota is a scholar working on Physiology, Genetics and Aging, having authored 47 papers that have together received 1.1k indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (12 papers), Epigenetics and DNA Methylation (11 papers) and Genomics and Phylogenetic Studies (9 papers). The work is most often cited by research in Molecular Biology (805 citations), Cancer Research (173 citations) and Genetics (284 citations). Mitsutaka Kadota has collaborated with scholars based in Japan, United States and United Kingdom. Frequent co-authors include Mitsuo Oshimura, Osamu Nishimura, Maxwell P. Lee, Shigehiro Kuraku, Howard H. Yang, Misako Sato, Lalage M. Wakefield, Barbara K. Dunn, Jiuhong Yuan and Jean‐Pierre J. Issa. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Nature Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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