Elisabetta Tronci
- Neurology top 2%
- Parkinson's Disease Mechanisms and Treatments 19
- Neurological disorders and treatments 6
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- Neuroscience and Neuropharmacology Research 18
- Neurotransmitter Receptor Influence on Behavior 18
- Nerve injury and regeneration 10
- Physiology top 2%
- Adenosine and Purinergic Signaling 5
- Developmental Neuroscience top 10%
- Neurology top 10%
- Parkinson's Disease Mechanisms and Treatments 19
- Neurological disorders and treatments 6
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- Receptor Mechanisms and Signaling 4
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- Autism Spectrum Disorder Research 3
In The Last Decade
Elisabetta Tronci
32 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 73
- Neurology 658
- Cellular and Molecular Neuroscience 728
- Physiology 130
- Developmental Neuroscience 40
- Neurology 65
Countries citing papers authored by Elisabetta Tronci
This map shows the geographic impact of Elisabetta Tronci's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elisabetta Tronci with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elisabetta Tronci more than expected).
Fields of papers citing papers by Elisabetta Tronci
This network shows the impact of papers produced by Elisabetta Tronci. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elisabetta Tronci. The network helps show where Elisabetta Tronci may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Elisabetta Tronci, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 19 | |
| 2 | 2018 | 31 | |
| 3 | 2017 | 23 | |
| 4 | 2017 | 20 | |
| 5 | 2015 | 54 | |
| 6 | 2015 | 18 | |
| 7 | 2015 | 43 | |
| 8 | 2014 | 33 | |
| 9 | 2014 | 50 | |
| 10 | 2013 | 38 | |
| 11 | 2013 | 15 | |
| 12 | 2013 | 45 | |
| 13 | 2012 | 24 | |
| 14 | 2009 | 36 | |
| 15 | 2009 | 79 | |
| 16 | 2007 | 64 | |
| 17 | 2006 | 46 | |
| 18 | 2006 | 11 | |
| 19 | 2005 | 53 | |
| 20 | 2003 | 31 |
About Elisabetta Tronci
Elisabetta Tronci is a scholar working on Cellular and Molecular Neuroscience, Neurology and Physiology, having authored 32 papers that have together received 1.0k indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (19 papers), Neuroscience and Neuropharmacology Research (18 papers), Neurotransmitter Receptor Influence on Behavior (18 papers), Nerve injury and regeneration (10 papers), Neurological disorders and treatments (6 papers), Adenosine and Purinergic Signaling (5 papers), Receptor Mechanisms and Signaling (4 papers) and Autism Spectrum Disorder Research (3 papers). The work is most often cited by research in Neurology (658 citations), Cellular and Molecular Neuroscience (728 citations) and Physiology (130 citations). Elisabetta Tronci has collaborated with scholars based in Italy, Sweden and Spain. Frequent co-authors include Manolo Carta, Micaela Morelli, Anders Björklund, Camino Fidalgo, Roberto Stancampiano, Anna R. Carta, Erwan Bézard, Annalisa Pinna, Ana Muñoz and Nicola Simola. Their work appears in journals such as Neurobiology of Disease, Experimental Neurology, Neuroscience, European Journal of Neuroscience and Neuroscience Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.