Edward G. Lynn

1.4k total citations
32 papers, 1.1k citations indexed

About

Edward G. Lynn is a scholar working on Molecular Biology, Surgery and Cell Biology. According to data from OpenAlex, Edward G. Lynn has authored 32 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 10 papers in Surgery and 7 papers in Cell Biology. Recurrent topics in Edward G. Lynn's work include Endoplasmic Reticulum Stress and Disease (7 papers), Lipoproteins and Cardiovascular Health (5 papers) and Adipose Tissue and Metabolism (5 papers). Edward G. Lynn is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (7 papers), Lipoproteins and Cardiovascular Health (5 papers) and Adipose Tissue and Metabolism (5 papers). Edward G. Lynn collaborates with scholars based in Canada, United States and Hong Kong. Edward G. Lynn's co-authors include O Karmin, Richard C. Austin, Yaw L. Siow, Michael N. Sack, Max Hamburgh, Šárka Lhoták, Ricky Y.K. Man, Christopher J. McLeod, Jianjun Bao and Peter J. Munson and has published in prestigious journals such as Journal of Biological Chemistry, Circulation Research and Diabetes.

In The Last Decade

Edward G. Lynn

32 papers receiving 1.0k citations

Peers

Edward G. Lynn

EPIDE

SURGE

PHYSI

Jun Hao China

Michelle T. Barati United States

Haruhisa Otani Japan

Chuan Qiu United States

Naotsuka Okayama Japan

Tamás Kardon Hungary

Cynthia Arbeeny United States

Jingzhou Chen China

M. Julia Brosnan United States

Seiji Umemoto Japan

Jun Hao China

Edward G. Lynn

629 ×1.4

159 ×0.9

EPIDE

146 ×0.9

SURGE

83 ×0.5

167 ×1.2

PHYSI

Citations per year, relative to Edward G. Lynn Edward G. Lynn (= 1×) peers Jun Hao

Countries citing papers authored by Edward G. Lynn

Since Specialization

Citations

This map shows the geographic impact of Edward G. Lynn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward G. Lynn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward G. Lynn more than expected).

Fields of papers citing papers by Edward G. Lynn

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edward G. Lynn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward G. Lynn. The network helps show where Edward G. Lynn may publish in the future.

Co-authorship network of co-authors of Edward G. Lynn

This figure shows the co-authorship network connecting the top 25 collaborators of Edward G. Lynn. A scholar is included among the top collaborators of Edward G. Lynn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edward G. Lynn. Edward G. Lynn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Lynn, Edward G., Jae Hyun Byun, Victor Kenyon, et al.. (2024). Small molecules targeting GRP78 mitigate anti-GRP78 autoantibody–mediated tissue factor procoagulant activity in cultured endothelial cells. Journal of Thrombosis and Haemostasis. 22(11). 3290–3304. 2 indexed citations

Platko, Khrystyna, Paul Lebeau, Melissa E. MacDonald, et al.. (2024). GDF10 is a negative regulator of vascular calcification. Journal of Biological Chemistry. 300(11). 107805–107805. 1 indexed citations

Lynn, Edward G., Šárka Lhoták, Hua Jiang, et al.. (2024). Restoration of the ER stress response protein TDAG51 in hepatocytes mitigates NAFLD in mice. Journal of Biological Chemistry. 300(2). 105655–105655. 2 indexed citations

Byun, Jae Hyun, Paul Lebeau, Khrystyna Platko, et al.. (2022). Inhibitory Antibodies against PCSK9 Reduce Surface CD36 and Mitigate Diet-Induced Renal Lipotoxicity. Kidney360. 3(8). 1394–1410. 23 indexed citations

Lynn, Edward G., et al.. (2022). Scratching the Surface—An Overview of the Roles of Cell Surface GRP78 in Cancer. Biomedicines. 10(5). 1098–1098. 14 indexed citations

Al‐Hashimi, Ali, Edward G. Lynn, Šárka Lhoták, et al.. (2018). Anti-GRP78 autoantibodies induce endothelial cell activation and accelerate the development of atherosclerotic lesions. JCI Insight. 3(24). 36 indexed citations

Ayaub, Ehab, Philipp Kolb, Zahraa Mohammed‐Ali, et al.. (2016). GRP78 and CHOP modulate macrophage apoptosis and the development of bleomycin‐induced pulmonary fibrosis. The Journal of Pathology. 239(4). 411–425. 92 indexed citations

Lynn, Edward G. & Richard C. Austin. (2010). Hydrogen sulfide in the pathogenesis of atherosclerosis and its therapeutic potential. Expert Review of Clinical Pharmacology. 4(1). 97–108. 49 indexed citations

Lynn, Edward G., et al.. (2010). Transient upregulation of PGC-1α diminishes cardiac ischemia tolerance via upregulation of ANT1. Journal of Molecular and Cellular Cardiology. 49(4). 693–698. 29 indexed citations

10.

Lacerda, Lydia, Joy McCarthy, Edward G. Lynn, et al.. (2010). TNFα protects cardiac mitochondria independently of its cell surface receptors. Basic Research in Cardiology. 105(6). 751–762. 38 indexed citations

11.

Lynn, Edward G., et al.. (2008). SIRT2 is a negative regulator of anoxia–reoxygenation tolerance via regulation of 14‐3‐3 ζ and BAD in H9c2 cells. FEBS Letters. 582(19). 2857–2862. 76 indexed citations

12.

Lynn, Edward G., et al.. (2007). The Regulation, Control, and Consequences of Mitochondrial Oxygen Utilization and Disposition in the Heart and Skeletal Muscle During Hypoxia. Antioxidants and Redox Signaling. 9(9). 1353–1362. 16 indexed citations

13.

Lynn, Edward G., et al.. (2003). Molecular characterization of the first avian LDL receptor. Journal of Lipid Research. 44(9). 1633–1642. 24 indexed citations

14.

Man, Ricky Y.K., Edward G. Lynn, Filly Cheung, Patricia Tsang, & O Karmin. (2002). Cholestin inhibits cholesterol synthesis and secretion in hepatic cells (HepG2). Molecular and Cellular Biochemistry. 233(1-2). 153–158. 30 indexed citations

15.

Lynn, Edward G., Yaw L. Siow, J. Fröhlich, Giselle Cheung, & O Karmin. (2001). Lipoprotein-X stimulates monocyte chemoattractant protein-1 expression in mesangial cells via nuclear factor-κB. Kidney International. 60(2). 520–532. 29 indexed citations

16.

Sung, Fion L., Yaw L. Siow, Guoping Wang, Edward G. Lynn, & O Karmin. (2001). Homocysteine stimulates the expression of monocyte chemoattractant protein-1 in endothelial cells leading to enhanced monocyte chemotaxis. Molecular and Cellular Biochemistry. 216(1-2). 121–128. 59 indexed citations

17.

Lynn, Edward G., Yaw L. Siow, & O Karmin. (2000). Very low-density lipoprotein stimulates the expression of monocyte chemoattractant protein-1 in mesangial cells. Kidney International. 57(4). 1472–1483. 33 indexed citations

18.

Karmin, O, et al.. (1998). Homocysteine stimulates the production and secretion of cholesterol in hepatic cells. Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism. 1393(2-3). 317–324. 63 indexed citations

19.

Lynn, Edward G., Patrick C. Choy, Alexander Magil, & O Karmin. (1997). Uptake and metabolism of lipoprotein-X in mesangial cells. Molecular and Cellular Biochemistry. 175(1-2). 187–194. 21 indexed citations

20.

Lynn, Edward G. & O Karmin. (1997). ROLE OF LIPOPROTEIN‐X IN FOAM CELL FORMATION AND RAT MESANGIAL CELL PROLIFERATION. Clinical and Experimental Pharmacology and Physiology. 24(12). 973–975. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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