Ehab Ayaub

3.1k total citations · 1 hit paper
19 papers, 1.7k citations indexed

About

Ehab Ayaub is a scholar working on Pulmonary and Respiratory Medicine, Cell Biology and Epidemiology. According to data from OpenAlex, Ehab Ayaub has authored 19 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Pulmonary and Respiratory Medicine, 6 papers in Cell Biology and 5 papers in Epidemiology. Recurrent topics in Ehab Ayaub's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (11 papers), Endoplasmic Reticulum Stress and Disease (6 papers) and Autophagy in Disease and Therapy (5 papers). Ehab Ayaub is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (11 papers), Endoplasmic Reticulum Stress and Disease (6 papers) and Autophagy in Disease and Therapy (5 papers). Ehab Ayaub collaborates with scholars based in Canada, United States and Australia. Ehab Ayaub's co-authors include Kjetil Ask, Iván O. Rosas, Jeffrey G. Dickhout, Sarah Chu, Philipp Kolb, Martin Kolb, Xiting Yan, Nir Neumark, Michael Januszyk and Jonas C. Schupp and has published in prestigious journals such as Journal of Biological Chemistry, Scientific Reports and CHEST Journal.

In The Last Decade

Ehab Ayaub

19 papers receiving 1.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis

2020 704 citations Taylor Adams, Jonas C. Schupp et al. Science Advances profile →

Peers

Countries citing papers authored by Ehab Ayaub

Since Specialization

Citations

This map shows the geographic impact of Ehab Ayaub's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ehab Ayaub with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ehab Ayaub more than expected).

Fields of papers citing papers by Ehab Ayaub

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ehab Ayaub. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ehab Ayaub. The network helps show where Ehab Ayaub may publish in the future.

Co-authorship network of co-authors of Ehab Ayaub

This figure shows the co-authorship network connecting the top 25 collaborators of Ehab Ayaub. A scholar is included among the top collaborators of Ehab Ayaub based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ehab Ayaub. Ehab Ayaub is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

#	Work	Indexed citations
1	Mesenchymal stromal cell‐derived syndecan‐2 regulates the immune response during sepsis to foster bacterial clearance and resolution of inflammation 2021 ·FEBS Journal ·(unknown), Yuanyuan Shi, Gareth R. Willis, (unknown), Min‐Young Kwon, Gu Li, Ehab Ayaub, Sailaja Ghanta, Julie Ng, (unknown), Konstantin Tsoyi, Souheil El‐Chemaly, Stella Kourembanas, S. Alex Mitsialis, Iván O. Rosas, Xiaoli Liu, Mark A. Perrella	13
2	Targeted inhibition of PI3 kinase/mTOR specifically in fibrotic lung fibroblasts suppresses pulmonary fibrosis in experimental models 2020 ·Science Translational Medicine ·Suraj U. Hettiarachchi, (unknown), Jyoti Roy, Fenghua Zhang, Estela Puchulu‐Campanella, (unknown), Madduri Srinivasarao, Konstantin Tsoyi, Xiaoliang Liang, Ehab Ayaub, Cheryl Nickerson‐Nutter, Iván O. Rosas, Philip S. Low	69
3	Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis breakdown → 2020 ·Science Advances ·Taylor Adams, Jonas C. Schupp, Sergio Poli, Ehab Ayaub, Nir Neumark, Farida Ahangari, Sarah Chu, Benjamin A. Raby, Giuseppe DeIuliis, Michael Januszyk, Qiaonan Duan, Heather A. Arnett, Asim Siddiqui, George R. Washko, Robert Homer, Xiting Yan, Iván O. Rosas, Naftali Kaminski	704
4	The importance of interventional timing in the bleomycin model of pulmonary fibrosis 2020 ·European Respiratory Journal ·(unknown), Chandak Upagupta, Megan Vierhout, Ehab Ayaub, (unknown), Jack Gauldie, Chiko Shimbori, Mark D. Inman, Kjetil Ask, Martin Kolb	140
5	IL-33 Mediates Lung Inflammation by the IL-6-Type Cytokine Oncostatin M 2020 ·Mediators of Inflammation ·Fernando Botelho, (unknown), Ehab Ayaub, (unknown), (unknown), (unknown), Roland Kolbeck, Carl D. Richards	16
6	Reprogramming of profibrotic macrophages for treatment of bleomycin‐induced pulmonary fibrosis 2020 ·EMBO Molecular Medicine ·Fenghua Zhang, Ehab Ayaub, Bingbing Wang, Estela Puchulu‐Campanella, (unknown), Suraj U. Hettiarachchi, (unknown), Qian Luo, (unknown), Madduri Srinivasarao, Abigail Cox, Konstantin Tsoyi, Cheryl Nickerson‐Nutter, Iván O. Rosas, Philip S. Low	68
7	Cell surface expression of 78-kDa glucose-regulated protein (GRP78) mediates diabetic nephropathy 2019 ·Journal of Biological Chemistry ·Richard Van Krieken, Neel Mehta, (unknown), (unknown), Renzhong Li, Bo Gao, Ehab Ayaub, Kjetil Ask, James C. Paton, Adrienne W. Paton, Richard C. Austin, Joan C. Krepinsky	38
8	Palmitic Acid–Rich High-Fat Diet Exacerbates Experimental Pulmonary Fibrosis by Modulating Endoplasmic Reticulum Stress 2019 ·American Journal of Respiratory Cell and Molecular Biology ·Sarah Chu, Julian A. Villalba, Xiaoliang Liang, (unknown), Konstantin Tsoyi, Bonna Ith, Ehab Ayaub, Raju V. V. Tatituri, Derek E. Byers, Fong‐Fu Hsu, Souheil El‐Chemaly, Edy Y. Kim, Yuanyuan Shi, Iván O. Rosas	89
9	Mechanical stress-induced mast cell degranulation activates TGF-β1 signalling pathway in pulmonary fibrosis 2019 ·Thorax ·Chiko Shimbori, Chandak Upagupta, Pierre‐Simon Bellaye, Ehab Ayaub, Seidai Sato, Toyoshi Yanagihara, Quan Zhou, (unknown), Kjetil Ask, Jack Gauldie, Paul Forsythe, Martin Kolb	67
10	Dectin-1 Is a Marker of Alternatively Activated Macrophages and a Therapeutic Target in Interstitial Fibrotic Lung Diseases 2019 ·(unknown), (unknown), (unknown), Megan Vierhout, (unknown), (unknown), Herry Patel, P.R. Parthasarathy, (unknown), Ehab Ayaub, Asghar Naqvi, Jeremy A. Hirota, Nathan Hambly, Martin Kolb, Kjetil Ask	2
11	IL‐6 mediates ER expansion during hyperpolarization of alternatively activated macrophages 2018 ·Immunology and Cell Biology ·Ehab Ayaub, (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Chandak Upagupta, (unknown), Anna Dvorkin‐Gheva, James S. Murphy, Philipp Kolb, Šárka Lhoták, Jeffrey G. Dickhout, (unknown), Martin Kolb, Carl D. Richards, Kjetil Ask	21
12	Overexpression of OSM and IL-6 impacts the polarization of pro-fibrotic macrophages and the development of bleomycin-induced lung fibrosis 2017 ·Scientific Reports ·Ehab Ayaub, (unknown), (unknown), Fernando Botelho, Martin Kolb, Carl D. Richards, Kjetil Ask	83
13	Nintedanib prevents IL-6-mediated secretion of CCL-18 in alternatively activated macrophages and transition of fibroblasts to myofibroblasts 2017 ·Kjetil Ask, (unknown), Ehab Ayaub, James S. Murphy, P.R. Parthasarathy, (unknown), (unknown), (unknown), Martin Kolb, (unknown), Franziska Herrmann, Maximilian Ackermann	1
14	Evaluation of novel LOXL2-selective inhibitors in models of pulmonary fibrosis 2017 ·(unknown), Victoria Tear, (unknown), (unknown), James S. Murphy, Chiko Shimbori, (unknown), (unknown), Ehab Ayaub, Mark G. Jones, Donna E. Davies, Wolfgang Jarolimek, Kjetil Ask, Jack Gauldie, Martin Kolb, Phillip Monk	2
15	Separate roles of IL‐6 and oncostatin M in mouse macrophage polarization in vitro and in vivo 2017 ·Immunology and Cell Biology ·(unknown), (unknown), Ehab Ayaub, (unknown), Kyle B. Stephenson, (unknown), (unknown), Richard C. Austin, Fernando Botelho, Kjetil Ask, Carl D. Richards	25
16	GRP78 and CHOP modulate macrophage apoptosis and the development of bleomycin‐induced pulmonary fibrosis 2016 ·The Journal of Pathology ·Ehab Ayaub, Philipp Kolb, Zahraa Mohammed‐Ali, (unknown), James S. Murphy, Pierre‐Simon Bellaye, Chiko Shimbori, Felix Boivin, Rocky Lai, Edward G. Lynn, Šárka Lhoták, Darren Bridgewater, Martin Kolb, Mark D. Inman, Jeffrey G. Dickhout, Richard C. Austin, Kjetil Ask	92
17	The therapeutic effects of 4-phenylbutyric acid in maintaining proteostasis 2015 ·The International Journal of Biochemistry & Cell Biology ·Philipp Kolb, Ehab Ayaub, Wenjing Zhou, (unknown), Jeffrey G. Dickhout, Kjetil Ask	224
18	Disruption of Calcium Signaling in Fibroblasts and Attenuation of Bleomycin-Induced Fibrosis by Nifedipine 2015 ·American Journal of Respiratory Cell and Molecular Biology ·Subhendu Mukherjee, Ehab Ayaub, James S. Murphy, Chao Lü, Martin Kolb, Kjetil Ask, Luke J. Janssen	42
19	Protein Misfolding and Endoplasmic Reticulum Stress in Chronic Lung Disease 2013 ·CHEST Journal ·James Cheng‐Chung Wei, (unknown), Ehab Ayaub, Jeffrey G. Dickhout, Kjetil Ask	53

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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